According VBG O
two CD O
different JJ O
studies NNS O
it PRP O
seems VBZ O
plausible JJ O
that IN O
the DT O
Pohang NNP B-C
earthquake NN I-C
was VBD O
induced VBN O
by IN O
EGS NNP B-C
operations NNS I-C
. . O

There EX O
are VBP O
three CD O
known VBN O
mutations NNS B-C
in IN I-C
CLCN2 NNP I-C
associated VBN O
with IN O
JME NNP B-C
, , O
M200fs NNP B-C
X231 CD I-C
, , O
74_117 CD B-C
del NNP I-C
, , O
and CC O
G715E NNP B-C
. . O

The DT O
age NN B-C
- HYPH O
associated VBN O
deterioration NN B-C
of IN I-C
force NN I-C
- HYPH I-C
generating VBG I-C
properties NNS I-C
of IN I-C
skeletal JJ I-C
muscles NNS I-C
can MD O
be VB O
directly RB O
associated VBN O
with IN O
increased VBN B-C
risk NN I-C
of IN I-C
physical JJ I-C
disability NN I-C
, , O
functional JJ B-C
impairments NNS I-C
, , O
and CC O
increased VBN B-C
mortality NN I-C
. . O

In IN O
turn NN O
, , O
these DT O
stimulate VBP O
fibroblasts NNS B-C
to TO O
proliferate VB O
and CC O
produce VB O
collagen NN B-C
around IN I-C
the DT I-C
silica NN I-C
particle NN I-C
, , O
thus RB O
resulting VBG O
in IN O
fibrosis NN B-C
and CC O
the DT O
formation NN B-C
of IN I-C
the DT I-C
nodular JJ I-C
lesions NNS I-C
. . O

Although IN O
silicosis NN O
has VBZ O
been VBN O
known VBN O
for IN O
centuries NNS O
, , O
the DT O
industrialization NN B-C
of IN I-C
mining NN I-C
has VBZ O
led VBN O
to IN O
an DT O
increase NN B-C
in IN I-C
silicosis NN I-C
cases NNS O
. . O

Activated VBN O
mast NN O
cells NNS O
and CC O
basophils NNS O
undergo VBP O
a DT O
process NN O
called VBN O
degranulation NN O
, , O
during IN O
which WDT O
they PRP O
release VBP B-C
histamine NN I-C
and CC I-C
other JJ I-C
inflammatory JJ I-C
chemical JJ I-C
mediators NNS I-C
called VBN O
( -LRB- O
cytokines NNS O
, , O
interleukins NNS O
, , O
leukotrienes NNS O
, , O
and CC O
prostaglandins NNS O
) -RRB- O
into IN O
the DT O
surrounding VBG O
tissue NN O
causing VBG O
several JJ B-C
systemic JJ I-C
effects NNS I-C
, , O
such JJ O
as IN O
vasodilation NN B-C
, , O
mucous NN B-C
secretion NN I-C
, , O
nerve NN B-C
stimulation NN I-C
, , O
and CC O
smooth JJ B-C
- HYPH I-C
muscle NN I-C
contraction NN I-C
. . O

These DT O
voids NNS B-C
may MD I-C
collapse VB I-C
producing VBG O
seismic JJ B-C
waves NNS I-C
and CC O
in IN O
some DT O
cases NNS O
reactivate VB B-C
existing VBG I-C
faults NNS I-C
causing VBG O
minor JJ B-C
earthquakes NNS I-C
. . O

The DT O
main JJ O
symptom NN O
of IN O
benign JJ B-C
fasciculation NN I-C
syndrome NN I-C
is VBZ O
focal JJ O
or CC O
widespread JJ O
involuntary JJ B-C
muscle NN I-C
activity NN I-C
( -LRB- O
twitching NN O
) -RRB- O
, , O
which WDT O
can MD O
occur VB O
at IN O
random JJ O
or CC O
specific JJ O
times NNS O
( -LRB- O
or CC O
places NNS O
) -RRB- O
. . O

The DT O
pneumatic JJ B-C
hammer NN I-C
drill NN I-C
was VBD O
introduced VBN O
in IN O
1897 CD O
and CC O
sandblasting NN B-C
was VBD O
introduced VBN O
in IN O
about RB O
1904 CD O
, , O
both CC O
significantly RB O
contributing VBG O
to IN O
the DT O
increased VBN O
prevalence NN O
of IN O
silicosis NN B-C
. . O

There EX O
are VBP O
lots NNS O
of IN O
different JJ O
causes NNS O
of IN O
dry JJ B-C
mouth NN I-C
including VBG O
side JJ B-C
effect NN I-C
of IN I-C
various JJ I-C
drugs NNS I-C
. . O

People NNS O
sometimes RB O
have VBP O
recent JJ O
onset NN O
of IN O
atypical JJ O
type NN O
2 CD O
diabetes NNS O
that WDT O
is VBZ O
difficult JJ O
to TO O
control VB O
, , O
a DT O
history NN O
of IN O
recent JJ O
but CC O
unexplained JJ O
blood NN B-C
vessel NN I-C
inflammation NN I-C
caused VBN O
by IN O
blood NN B-C
clots NNS I-C
( -LRB- O
thrombophlebitis NN O
) -RRB- O
known VBN O
as IN O
Trousseau NNP O
sign NN O
, , O
or CC O
a DT O
previous JJ O
attack NN O
of IN O
pancreatitis NN O
. . O

Studies NNS O
have VBP O
indicated VBN O
that DT O
soft JJ B-C
, , I-C
consistent JJ I-C
bit NN I-C
contact NN I-C
between IN I-C
the DT I-C
rider NN I-C
and CC I-C
horse NN I-C
causes VBZ O
the DT O
animal NN O
less JJR B-C
stress NN I-C
than IN O
intermittent JJ B-C
or CC I-C
unpredictable JJ I-C
contact NN I-C
. . O

A DT O
temperature NN B-C
inversion NN I-C
had VBD O
developed VBN O
, , O
trapping VBG O
moisture NN B-C
low JJ I-C
in IN I-C
the DT I-C
atmosphere NN I-C
and CC O
causing VBG O
a DT O
dense JJ B-C
fog NN I-C
to TO I-C
develop VB I-C
. . O

This DT O
phase NN O
can MD O
either RB O
subside VB O
or CC O
progress VB O
into IN O
a DT O
" `` O
late JJ B-C
- HYPH I-C
phase NN I-C
reaction NN I-C
" '' O
which WDT O
can MD O
substantially RB O
prolong VB O
the DT O
symptoms NNS O
of IN O
a DT O
response NN O
, , O
and CC O
result VB O
in IN O
more JJR O
tissue NN B-C
damage NN I-C
. . O

Chronic NN B-C
nosebleeds NNS I-C
and CC O
digestive JJ B-C
tract NN I-C
bleeding NN I-C
can MD O
both RB O
lead VB O
to IN O
anemia NN B-C
; , O
if IN O
the DT O
bleeding NN O
itself PRP O
can MD O
not RB O
be VB O
completely RB O
stopped VBN O
, , O
the DT O
anemia NN O
requires VBZ O
treatment NN O
with IN O
iron NN O
supplements NNS O
. . O

Induced VBN B-C
seismicity NN I-C
can MD O
be VB O
also RB O
related JJ O
to IN O
underground JJ B-C
gas NN I-C
storage NN I-C
operations NNS I-C
. . O

Signs NNPS O
of IN O
JME NNP B-C
are VBP O
brief JJ O
episodes NNS O
of IN O
involuntary JJ B-C
muscle NN I-C
twitching NN I-C
occurring VBG O
early RB O
in IN O
the DT O
morning NN O
or CC O
shortly RB O
before IN O
falling VBG O
asleep RB O
. . O

These DT O
attacks NNS B-C
induce VBP O
a DT O
nightmare NN B-C
that WDT O
leads VBZ O
to TO O
sleep VB B-C
paralysis NN I-C
when WRB O
the DT O
victim NN O
is VBZ O
conscious JJ O
and CC O
experiencing VBG O
pressure NN O
on IN O
the DT O
chest NN O
. . O

The DT O
events NNS B-C
caused VBD O
hundreds NNS O
of IN O
thousands NNS O
of IN O
dollars NNS O
worth JJ O
of IN O
property NN B-C
damage NN I-C
to IN O
City NNP O
Hall NNP O
and CC O
the DT O
surrounding VBG O
area NN O
, , O
as RB O
well RB O
as IN O
injuries NNS B-C
to IN I-C
police NNS I-C
officers NNS I-C
and CC I-C
rioters NNS I-C
. . O

Electron NN B-C
beam NN I-C
sources NNS I-C
and CC O
particle NN B-C
accelerators NNS I-C
can MD O
be VB O
also RB O
sources NNS O
of IN O
beta NN B-C
burns NNS I-C
. . O

Unexplained VBN B-C
weight NN I-C
loss NN I-C
, , O
either CC O
from IN O
loss NN B-C
of IN I-C
appetite NN I-C
, , O
or CC O
loss NN B-C
of IN I-C
exocrine JJ I-C
function NN I-C
resulting VBG O
in IN O
poor JJ B-C
digestion NN I-C
. . O

This DT O
results VBZ O
in IN O
weight NN B-C
gain NN I-C
because IN O
students NNS B-C
do VBP I-C
not RB I-C
workout VB I-C
as RB O
much JJ O
and CC O
become VB B-C
lazier JJR I-C
in IN I-C
everyday NN I-C
tasks NNS I-C
. . O

In IN O
humans NNS O
, , O
a DT O
HSAN JJ B-C
ID NN I-C
- HYPH O
associated VBN O
mutation NN B-C
caused VBD O
significant JJ B-C
loss NN I-C
of IN I-C
the DT I-C
GTPase NN I-C
activity NN I-C
of IN I-C
the DT I-C
enzyme NN I-C
, , O
leading VBG O
to IN O
prominent JJ B-C
disruption NN I-C
of IN I-C
ER NN I-C
network NN I-C
. . O

Medical JJ B-C
problems NNS I-C
can MD O
result VB O
in IN O
interventions NNS B-C
that WDT O
can MD O
be VB O
frightening JJ O
. . O

Additionally RB O
, , O
testing NN O
for IN O
common JJ O
risk NN O
factors NNS O
such JJ O
as IN O
Hepatitis NN B-C
C NN I-C
and CC O
hemochromatosis NN B-C
is VBZ O
strongly RB O
suggested VBN O
, , O
as IN O
their PRP$ O
high JJ O
prevalence NN O
in IN O
patients NNS O
with IN O
PCT NNP B-C
may MD O
require VB O
additional JJ O
treatment NN O
. . O

Ulcerated VBN B-C
hemangiomas NNS I-C
can MD O
present VB O
as IN O
black JJ B-C
crusted VBN I-C
papules NNS I-C
or CC I-C
plaques NNS I-C
, , O
or CC O
painful JJ B-C
erosions NNS I-C
or CC O
ulcers NNS B-C
. . O

Infection NN B-C
with IN I-C
the DT I-C
bacteria NN I-C
Helicobacter NN I-C
bilis NN I-C
and CC O
Helicobacter NN B-C
hepaticus JJ I-C
species NNS I-C
can MD O
cause VB O
biliary JJ B-C
cancer NN I-C
. . O

Large JJ B-C
AVMs NNS I-C
may MD O
lead VB O
to IN O
platypnea NN B-C
, , O
difficulty NN O
in IN O
breathing NN O
that WDT O
is VBZ O
more RBR O
marked VBN O
when WRB O
sitting VBG O
up RP O
compared VBN O
to IN O
lying VBG O
down RP O
; , O
this DT O
probably RB O
reflects VBZ O
changes NNS O
in IN O
blood NN O
flow NN O
associated VBN O
with IN O
positioning NN O
. . O

To TO O
limit VB O
the DT O
anxiety NN B-C
due IN O
to IN O
these DT O
symptoms NNS B-C
you PRP O
can MD O
consult VB O
a DT O
health NN O
professional JJ O
or CC O
a DT O
psychologist NN O
. . O

However RB O
, , O
bites NNS O
from IN O
male JJ O
redback NN O
spiders NNS O
have VBP O
been VBN O
reported VBN O
suggesting VBG O
male JJ B-C
Latrodectus NNP I-C
spiders NNS I-C
can MD O
cause VB O
envenoming VBG B-C
in IN I-C
humans NNS I-C
. . O

The DT O
U.S. NNP O
Environmental NNP O
Protection NNP O
Agency NNP O
has VBZ O
asserted VBN O
the DT O
same JJ O
sentiment NN O
and CC O
testified VBN O
before IN O
the DT O
U.S. NNP O
Congress NNP O
that IN O
approximately RB O
34 CD O
million CD O
Americans NNPS O
are VBP O
exposed VBN O
to IN O
noise NN B-C
pollution NN I-C
levels NNS O
( -LRB- O
mostly RB O
from IN O
roadway NN O
and CC O
aircraft NN O
noise NN O
) -RRB- O
that WDT O
expose VBP O
humans NNS O
to TO O
noise VB O
health NN O
effects NNS O
including VBG O
the DT O
risk NN O
of IN O
hearing NN B-C
loss NN I-C
( -LRB- O
EPA NNP O
, , O
1972 CD O
) -RRB- O
. . O

The DT O
functioning NN B-C
types NNS I-C
secrete VBP O
hormones NNS O
such JJ O
as IN O
insulin NN O
, , O
gastrin NN O
, , O
and CC O
glucagon NN O
into IN O
the DT O
bloodstream NN O
, , O
often RB O
in IN O
large JJ O
quantities NNS O
, , O
giving VBG O
rise NN O
to IN O
serious JJ O
symptoms NNS O
such JJ O
as IN O
low JJ B-C
blood NN I-C
sugar NN I-C
, , O
but CC O
also RB O
favoring VBG O
relatively RB O
early JJ O
detection NN O
. . O

Although IN O
there EX O
is VBZ O
no DT O
known VBN O
cause NN O
for IN O
piblokto NN B-C
, , O
Western JJ O
scientists NNS O
have VBP O
attributed VBN O
the DT O
disorder NN O
to IN O
the DT O
lack NN B-C
of IN I-C
sun NN I-C
, , O
the DT O
extreme JJ B-C
cold JJ I-C
, , O
and CC O
the DT O
desolate JJ B-C
state NN I-C
of IN I-C
most JJS I-C
villages NNS I-C
in IN I-C
the DT I-C
region NN I-C
. . O

It PRP O
is VBZ O
also RB O
felt VBN O
to TO O
increase VB O
the DT O
uptake NN B-C
of IN I-C
iron NN I-C
in IN I-C
liver NN I-C
cells NNS I-C
, , O
leading VBG O
to IN O
further JJ O
oxidation NN B-C
of IN I-C
uroporphyrinogen NN I-C
by IN O
the DT O
release NN O
of IN O
activated VBN O
oxygen NN O
species NNS O
. . O

The DT O
diagnosis NN O
of IN O
AGEP NNP O
may MD O
be VB O
forthright JJ O
in IN O
typical JJ O
cases NNS O
in IN O
which WDT O
an DT O
individual NN O
: : O
has VBZ O
taken VBN O
a DT O
drug NN B-C
known VBN O
to TO O
cause VB O
the DT O
disorder NN B-C
; , O
develops VBZ O
multiple JJ O
sterile JJ O
pustules NNS O
overlying VBG O
large JJ O
areas NNS O
of IN O
red JJ O
swollen JJ O
skin NN O
starting VBG O
a DT O
few JJ O
days NNS O
after IN O
initial JJ O
drug NN O
intake NN O
; , O
and CC O
has VBZ O
a DT O
histology NN O
of IN O
biopsied VBN O
lesions NNS O
that WDT O
shows VBZ O
pustules NNS O
just RB O
below IN O
the DT O
skin NN O
's POS O
Stratum NN O
corneum NN O
( -LRB- O
outermost JJ O
layer NN O
) -RRB- O
, , O
apoptotic JJ O
( -LRB- O
i.e. FW O
necrotic JJ O
) -RRB- O
keratinocytes NNS O
, , O
spongiosis NN O
of IN O
the DT O
stratum NN O
spinosum NN O
, , O
and CC O
infiltration NN O
of IN O
these DT O
tissues NNS O
by IN O
neutrophils NNS O
plus CC O
, , O
in IN O
many JJ O
but CC O
not RB O
all DT O
cases NNS O
, , O
eosinophils NNS O
. . O

Known VBN O
risk NN O
factors NNS O
for IN O
cholangiocarcinoma NN B-C
include VBP O
primary JJ B-C
sclerosing NN I-C
cholangitis NN I-C
( -LRB- O
an DT O
inflammatory JJ O
disease NN O
of IN O
the DT O
bile NN O
ducts NNS O
) -RRB- O
, , O
infection NN B-C
with IN I-C
the DT I-C
parasitic JJ I-C
liver NN I-C
flukes NNS I-C
Opisthorchis NN I-C
viverrini NN I-C
or CC I-C
Clonorchis NN I-C
sinensis NN I-C
, , O
some DT O
congenital JJ B-C
liver NN I-C
malformations NNS I-C
, , O
and CC O
exposure NN B-C
to IN I-C
Thorotrast NNP I-C
( -LRB- O
thorium NN O
dioxide NN O
) -RRB- O
, , O
a DT O
chemical JJ O
formerly RB O
used VBN O
in IN O
medical JJ O
imaging NN O
. . O

Doroshenko NNP B-C
's POS I-C
pro-Turkish JJ I-C
policy NN I-C
caused VBD O
open JJ B-C
resentment NN I-C
among IN I-C
many JJ I-C
colonels NNS I-C
and CC I-C
a DT I-C
large JJ I-C
part NN I-C
of IN I-C
the DT I-C
Right NNP I-C
Bank NNP I-C
Cossacks NNPS I-C
. . O

The DT O
relative JJ B-C
quiet JJ I-C
of IN I-C
the DT I-C
Tatar JJ I-C
attacks NNS I-C
on IN I-C
Russia NNP I-C
in IN I-C
the DT I-C
late JJ I-C
30s NNS I-C
and CC I-C
early JJ I-C
40s NNS I-C
of IN I-C
the DT I-C
17th JJ I-C
century NN I-C
is VBZ O
undoubtedly RB O
due IN O
to IN O
the DT O
occupation NN B-C
of IN I-C
Azov NNP I-C
by IN I-C
the DT I-C
Don NNP I-C
Cossacks NNPS I-C
. . O

A DT O
2011 CD O
study NN O
found VBD O
that IN O
pollution NN B-C
is VBZ O
making VBG O
Arabian NNP B-C
Sea NNP I-C
cyclones NNS I-C
more RBR I-C
intense JJ I-C
as IN O
the DT O
atmospheric JJ B-C
brown JJ I-C
clouds NNS I-C
has VBZ O
been VBN O
producing VBG O
weakening VBG B-C
wind NN I-C
patterns NNS I-C
which WDT O
prevent VBP O
wind NN O
shear NN O
patterns NNS O
that WDT O
historically RB O
have VBP O
prohibited VBN O
cyclones NNS O
in IN O
the DT O
Arabian NNP O
Sea NNP O
from IN O
becoming VBG O
major JJ O
storms NNS O
. . O

The DT O
most RBS O
common JJ O
cause NN O
of IN O
chronic NN B-C
venous JJ I-C
insufficiency NN I-C
is VBZ O
reflux NN B-C
of IN I-C
the DT I-C
venous JJ I-C
valves NNS I-C
of IN I-C
superficial JJ I-C
veins NNS I-C
. . O

The DT O
Siege NNP B-C
of IN I-C
Warsaw NNP I-C
( -LRB- O
1939 CD O
) -RRB- O
caused VBD O
a DT O
huge JJ B-C
toll NN I-C
of IN I-C
civilian JJ I-C
casualties NNS I-C
. . O

It PRP O
should MD O
be VB O
noted VBN O
that IN O
chloroquine NN B-C
treatment NN I-C
can MD O
induce VB O
porphyria JJ B-C
attacks NNS I-C
within IN O
the DT O
first JJ O
couple NN O
of IN O
months NNS O
of IN O
treatment NN O
due IN O
to IN O
the DT O
mass JJ B-C
mobilization NN I-C
of IN I-C
porphyrins NNS I-C
from IN I-C
the DT I-C
liver NN I-C
into IN I-C
the DT I-C
blood NN I-C
stream NN I-C
. . O

Third RB B-C
and CC I-C
fourth JJ I-C
degree NN I-C
beta NN I-C
burns NNS I-C
result NN O
in IN O
deeper JJR O
, , O
wet JJ O
ulcerated VBN B-C
lesions NNS I-C
, , O
which WDT O
heal VBP O
with IN O
routine NN O
medical JJ O
care NN O
after IN O
covering VBG O
themselves PRP O
with IN O
dry JJ O
scab NN O
. . O

Mining NN B-C
leaves VBZ I-C
voids NNS O
that WDT O
generally RB O
alter VBP B-C
the DT I-C
balance NN I-C
of IN I-C
forces NNS I-C
in IN I-C
the DT I-C
rock NN I-C
, , O
many JJ O
times NNS O
causing VBG O
rock NN B-C
bursts NNS I-C
. . O

After IN O
a DT O
landslide NN B-C
almost RB I-C
filled VBD I-C
the DT I-C
reservoir NN I-C
in IN I-C
1963 CD I-C
, , O
causing VBG O
a DT O
massive JJ B-C
flooding NN I-C
and CC O
around RB O
2,000 CD B-C
deaths NNS I-C
, , O
it PRP O
was VBD O
drained VBN O
and CC O
consequently RB O
seismic JJ O
activity NN O
was VBD O
almost RB O
non-existent JJ O
. . O

At IN O
this DT O
point NN O
, , O
barely RB O
emerging VBG O
from IN O
the DT O
seasonal JJ B-C
low JJ I-C
demand NN I-C
caused VBN O
by IN O
summer NN B-C
, , O
a DT O
large JJ O
number NN O
of IN O
industrial JJ O
facilities NNS O
and CC O
power NN O
plants NNS O
started VBD O
suffering VBG O
intermittent JJ O
cuts NNS O
in IN O
their PRP$ O
supply NN O
of IN O
natural JJ O
gas NN O
. . O

In IN O
advanced JJ O
cases NNS O
, , O
the DT O
following VBG O
may MD O
also RB O
occur VB O
: : O
Cyanosis NN O
, , O
pallor NN O
along IN O
upper JJ O
parts NNS O
of IN O
body NN O
( -LRB- O
blue JJ O
skin NN O
) -RRB- O
Cor NN O
pulmonale NN O
( -LRB- O
right JJ O
ventricle NN O
heart NN O
disease NN O
) -RRB- O
Respiratory JJ O
insufficiency NN O
Patients NNS O
with IN O
silicosis NN B-C
are VBP O
particularly RB O
susceptible JJ O
to IN O
tuberculosis NN B-C
( -LRB- O
TB NN O
) -RRB- O
infection NN O
— , O
known VBN O
as IN O
silicotuberculosis NN O
. . O

In IN O
certain JJ O
patients NNS O
with IN O
temporal JJ O
lobe NN O
epilepsy NN O
it PRP O
has VBZ O
been VBN O
found VBN O
that IN O
the DT O
subiculum NN B-C
could MD O
generate VB O
epileptic JJ B-C
activity NN I-C
. . O

Other JJ O
problems NNS B-C
become VBP O
apparent JJ O
due IN O
to IN O
aging NN B-C
of IN I-C
bodily NN I-C
and CC I-C
cellular JJ I-C
maintenance NN I-C
processes NNS I-C
. . O

While IN O
the DT O
report NN O
stated VBD O
that IN O
pilot NN B-C
error NN I-C
was VBD O
the DT O
main JJ O
cause NN O
of IN O
the DT O
accident NN B-C
, , O
with IN O
the DT O
crew NN O
lacking VBG O
adequate JJ O
training NN O
in IN O
operating VBG O
in IN O
adverse JJ O
weather NN O
conditions NNS O
, , O
it PRP O
differed VBD O
from IN O
the DT O
Russian JJ O
report NN O
in IN O
several JJ O
aspects NNS O
. . O

With IN O
regard NN O
to IN O
the DT O
relation NN O
of IN O
higher JJR B-C
levels NNS I-C
of IN I-C
muscular JJ I-C
strength NN I-C
to IN O
a DT O
lower JJR B-C
risk NN I-C
of IN I-C
premature JJ I-C
death NN I-C
, , O
studies NNS O
by IN O
Newman NNP O
et FW O
al NNP O
. . O
have VBP O
shown VBN O
that IN O
grip NN O
and CC O
knee NN O
extensor NN O
muscle NN O
strength NN O
are VBP O
strongly RB O
correlated VBN O
with IN O
mortality NN O
. . O

Schemas NNS B-C
cause VBP O
the DT O
activation NN B-C
of IN I-C
behaviors NNS I-C
; , O
the DT O
greater JJR O
the DT O
excitation NN O
of IN O
the DT O
activity NN O
the DT O
more RBR O
easily RB O
it PRP O
is VBZ O
to TO O
achieve VB O
the DT O
subgoals NNS O
and CC O
complete VB O
the DT O
schema NN O
. . O

Large JJ B-C
segmental JJ I-C
hemangiomas NNS I-C
of IN I-C
the DT I-C
head NN I-C
and CC I-C
neck NN I-C
can MD O
be VB O
associated VBN O
with IN O
a DT O
disorder NN O
called VBN O
PHACES NNS B-C
syndrome NN I-C
. . O

Viral NN B-C
infections NNS I-C
have VBP O
also RB O
been VBN O
observed VBN O
to TO O
be VB O
associated VBN O
with IN O
the DT O
development NN O
of IN O
SJS NN B-C
, , O
SJS NN B-C
/ , I-C
TEN CD I-C
, , O
and CC O
TEN CD B-C
in IN O
the DT O
absence NN O
of IN O
a DT O
causative JJ O
drug NN O
. . O

A DT O
2009 CD O
study NN O
by IN O
the DT O
University NNP O
of IN O
Bristol NNP O
’s POS O
Department NNP O
of IN O
Clinical NNP O
Veterinary NNP O
Sciences NNPS O
showed VBD O
that IN O
methods NNS B-C
of IN I-C
handling NN I-C
that WDT I-C
relied VBD I-C
on IN I-C
dominance NN I-C
theory NN I-C
actually RB O
provoked VBD O
aggressive JJ B-C
behavior NN I-C
in IN I-C
dogs NNS I-C
with IN O
no DT O
previous JJ O
known VBN O
history NN O
of IN O
aggression NN O
. . O

Ramachandran NNP O
was VBD O
able JJ O
to TO O
show VB O
that IN O
patients NNS O
with IN O
TLE NNP B-C
showed VBD O
enhanced VBN B-C
emotional JJ I-C
responses NNS I-C
to IN I-C
the DT I-C
religious JJ I-C
words NNS I-C
, , O
diminished VBN B-C
responses NNS I-C
to IN I-C
the DT I-C
sexually RB I-C
charged VBN I-C
words NNS I-C
, , O
and CC O
normal JJ O
responses NNS O
to IN O
the DT O
neutral JJ O
words NNS O
. . O

The DT O
other JJ O
factor NN O
being VBG O
a DT O
direct JJ O
relationship NN O
between IN O
increased VBN B-C
resistance NN I-C
training NN I-C
volume NN I-C
, , O
which WDT O
is VBZ O
the DT O
total JJ O
number NN O
of IN O
exercise NN O
sets NNS O
performed VBN O
in IN O
a DT O
session NN O
, , O
and CC O
improvements NNS B-C
in IN I-C
lean JJ I-C
body NN I-C
mass NN I-C
. . O

Though IN O
safe JJ O
practices NNS O
and CC O
existing VBG O
technologies NNS O
can MD O
be VB O
utilized VBN O
to TO O
reduce VB O
the DT O
risk NN O
of IN O
induced VBN B-C
seismicity NN I-C
due IN O
to IN O
injection NN B-C
of IN I-C
carbon NN I-C
dioxide NN I-C
, , O
the DT O
risk NN O
is VBZ O
still RB O
significant JJ O
if IN O
the DT O
storage NN O
is VBZ O
large JJ O
in IN O
scale NN O
. . O

Cigarette NN B-C
smoking NN I-C
is VBZ O
the DT O
best JJS O
- HYPH O
established VBN O
avoidable JJ O
risk NN O
factor NN O
for IN O
pancreatic JJ B-C
cancer NN I-C
, , O
approximately RB O
doubling VBG O
risk NN O
among IN O
long JJ O
- HYPH O
term NN O
smokers NNS O
, , O
the DT O
risk NN O
increasing VBG O
with IN O
the DT O
number NN O
of IN O
cigarettes NNS O
smoked VBN O
and CC O
the DT O
years NNS O
of IN O
smoking NN O
. . O

While IN O
structural JJ O
violence NN O
impacts NNS O
Haiti NNP O
as IN O
a DT O
whole JJ O
, , O
due IN O
to IN O
the DT O
presence NN O
of IN O
gender NN B-C
inequality NN I-C
, , O
women NNS B-C
within IN I-C
Haiti NNP I-C
tend VBP I-C
to TO I-C
be VB I-C
more RBR I-C
heavily RB I-C
impacted VBN I-C
by IN I-C
structural JJ I-C
violence NN I-C
than IN I-C
men NNS I-C
. . O

There EX O
is VBZ O
growing VBG O
recognition NN O
that IN O
the DT O
infection NN O
of IN O
Baylisascaris NNP B-C
shroederi VBD I-C
is VBZ O
one CD O
of IN O
the DT O
major JJ O
causes NNS O
of IN O
death NN B-C
in IN O
the DT O
species NNS O
. . O

Successful JJ B-C
transmission NN I-C
and CC I-C
infection NN I-C
by IN I-C
either CC I-C
SPONV NN I-C
strain NN I-C
can MD O
result VB O
in IN O
the DT O
infectious JJ B-C
disease NN I-C
known VBN O
as IN O
Spondweni NNP B-C
fever NN I-C
. . O

Malnutrition NN B-C
can MD O
be VB O
caused VBN O
by IN O
a DT O
number NN O
of IN O
things NNS O
including VBG O
inadequate JJ B-C
or CC I-C
unbalanced JJ I-C
diet NN I-C
, , O
problems NNS B-C
with IN I-C
digestion NN I-C
or CC I-C
absorption NN I-C
, , O
or CC O
certain JJ O
medical JJ B-C
conditions NNS I-C
. . O

For IN O
uncomplicated JJ O
silicosis NN B-C
, , O
chest NN O
x NN O
- HYPH O
ray NN O
will MD O
confirm VB O
the DT O
presence NN O
of IN O
small JJ O
( -LRB- O
< -LRB- O
10 CD O
mm NN O
) -RRB- O
nodules NNS B-C
in IN I-C
the DT I-C
lungs NNS I-C
, , O
especially RB O
in IN O
the DT O
upper JJ O
lung NN O
zones NNS O
. . O

Chronic NN B-C
radiodermatitis NN I-C
occurs VBZ O
with IN O
chronic JJ O
exposure NN O
to IN O
" `` O
sub NN O
- HYPH O
erythema NN O
" '' O
doses NNS O
of IN O
ionizing NN B-C
radiation NN I-C
over IN O
a DT O
prolonged VBN O
period NN O
, , O
producing VBG O
varying VBG O
degrees NNS O
of IN O
damage NN B-C
to IN I-C
the DT I-C
skin NN I-C
and CC O
its PRP$ O
underlying JJ O
parts NNS O
after IN O
a DT O
variable JJ O
latent JJ O
period NN O
of IN O
several JJ O
months NNS O
to IN O
several JJ O
decades NNS O
. . O

When WRB O
the DT O
loss NN B-C
is VBZ O
due IN O
to IN O
heredity NN B-C
, , O
total JJ B-C
deafness NN I-C
is VBZ O
often RB O
the DT O
end NN O
result NN O
. . O

Piblokto NNP B-C
is VBZ O
an DT O
abrupt JJ O
dissociative JJ O
episode NN O
with IN O
four CD O
phases NNS O
: : O
social JJ B-C
withdrawal NN I-C
, , O
excitement NN B-C
, , O
convulsions NNS B-C
and CC O
stupor NN B-C
, , O
and CC O
recovery NN B-C
. . O

Risk NN O
factors NNS O
for IN O
pancreatic JJ B-C
cancer NN I-C
include VBP O
tobacco NN B-C
smoking NN I-C
, , O
obesity NN B-C
, , O
diabetes NNS B-C
, , O
and CC O
certain JJ O
rare JJ B-C
genetic JJ I-C
conditions NNS I-C
. . O

Possible JJ O
biological JJ O
contributors NNS O
to TO O
dynapenia VB B-C
include VBP O
the DT O
nervous JJ B-C
system NN I-C
’s POS I-C
deteriorating VBG I-C
control NN I-C
of IN I-C
voluntary JJ I-C
skeletal JJ I-C
muscle NN I-C
activation NN I-C
and CC O
a DT O
decreased VBN B-C
number NN I-C
of IN I-C
functioning VBG I-C
motor NN I-C
units NNS I-C
. . O

Following VBG O
a DT O
strain NN O
or CC O
partial JJ O
rupture NN O
of IN O
patellar NN O
ligament NN O
the DT O
patient NN O
develops VBZ O
a DT O
traction NN B-C
‘ `` I-C
tendinitis NN I-C
’ '' I-C
characterized VBN O
by IN O
pain NN B-C
and CC O
point NN B-C
tenderness NN I-C
at IN I-C
the DT I-C
inferior JJ I-C
( -LRB- I-C
lower JJR I-C
) -RRB- I-C
pole NN I-C
of IN I-C
the DT I-C
patella NN I-C
associated VBN O
with IN O
focal JJ O
swelling NN O
. . O

Many JJ O
well RB O
- HYPH O
known VBN O
sources NNS O
of IN O
fluoride NN O
may MD O
contribute VB O
to IN O
overexposure NN B-C
including VBG O
dentifrice JJ B-C
/ , I-C
fluoridated VBN I-C
mouthrinse NN I-C
( -LRB- O
which WDT O
young JJ O
children NNS O
may MD O
swallow VB O
) -RRB- O
, , O
excessive JJ B-C
ingestion NN I-C
of IN I-C
fluoride NN I-C
toothpaste NN I-C
, , O
bottled VBN B-C
waters NNS I-C
which WDT O
are VBP O
not RB O
tested VBN O
for IN O
their PRP$ O
fluoride NN O
content NN O
, , O
inappropriate JJ B-C
use NN I-C
of IN I-C
fluoride NN I-C
supplements NNS I-C
, , O
ingestion NN B-C
of IN I-C
foods NNS I-C
especially RB O
imported VBN O
from IN O
other JJ O
countries NNS O
, , O
and CC O
public JJ B-C
water NN I-C
fluoridation NN I-C
. . O

The DT O
pits NNS B-C
, , O
bands NNS B-C
, , O
and CC O
loss NN B-C
of IN I-C
areas NNS I-C
of IN I-C
enamel NN I-C
seen VBN O
in IN O
severe JJ B-C
fluorosis NN I-C
are VBP O
the DT O
result NN O
of IN O
damage NN B-C
to IN I-C
the DT I-C
severely RB I-C
hypomineralized VBN I-C
, , I-C
brittle JJ I-C
and CC I-C
fragile JJ I-C
enamel NN I-C
which WDT O
occurs VBZ O
after IN O
they PRP O
erupt VBP O
into IN O
the DT O
mouth NN O
. . O

There EX O
are VBP O
at RB O
least RBS O
two CD O
mutations NNS B-C
in IN O
the DT O
β NN O
4 CD O
subunit NN O
associated VBN O
with IN O
JME NNP B-C
, , O
C104 NNP O
F NNP O
and CC O
R482X CD O
. . O

The DT O
clinical JJ O
features NNS O
of IN O
latrodectism NN B-C
are VBP O
similar JJ O
for IN O
all DT O
species NNS O
of IN O
Latrodectus NNP O
spiders NNS O
and CC O
is VBZ O
generally RB O
characterised VBN O
by IN O
extreme JJ B-C
pain NN I-C
. . O

If IN O
the DT O
AVM NNP B-C
creates VBZ I-C
a DT I-C
connection NN I-C
between IN I-C
the DT I-C
portal JJ I-C
vein NN I-C
and CC I-C
the DT I-C
blood NN I-C
vessels NNS I-C
of IN O
the DT O
liver NN O
, , O
the DT O
result NN O
may MD O
be VB O
portal JJ B-C
hypertension NN I-C
( -LRB- O
increased VBN O
portal JJ O
vein NN O
pressure NN O
) -RRB- O
, , O
in IN O
which WDT O
collateral NN O
blood NN O
vessels NNS O
form NN O
in IN O
the DT O
esophagus NN O
( -LRB- O
esophageal JJ O
varices NNS O
) -RRB- O
, , O
which WDT O
may MD O
bleed VB O
violently RB O
; , O
furthermore RB O
, , O
the DT O
increased VBN B-C
pressure NN I-C
may MD O
give VB O
rise NN O
to IN O
fluid NN B-C
accumulation NN I-C
in IN I-C
the DT I-C
abdominal JJ I-C
cavity NN I-C
( -LRB- O
ascites NNS O
) -RRB- O
. . O

This DT O
situation NN O
has VBZ O
led VBN O
to IN O
severe JJ B-C
underinvestment NN I-C
and CC O
unavailability NN B-C
to TO I-C
keep VB I-C
up RP I-C
with IN I-C
an DT I-C
increasing VBG I-C
demand NN I-C
, , O
factors NNS O
that WDT O
contributed VBD O
to IN O
the DT O
2003 CD B-C
- HYPH I-C
2004 CD I-C
energy NN I-C
crisis NN I-C
. . O

The DT O
take NN O
- HYPH O
off NN O
warning NN O
system NN O
that WDT O
would MD O
have VB O
sounded VBN O
an DT O
alarm NN B-C
due IN O
to IN O
the DT O
flaps NNS B-C
not RB I-C
being VBG I-C
lowered VBN I-C
did VBD O
not RB O
have VB O
a DT O
separate JJ O
warning NN O
that IN O
the DT O
slats NNS O
' POS O
pneumatic JJ O
valve NN O
had VBD O
not RB O
been VBN O
opened VBN O
by IN O
the DT O
flight NN O
engineer NN O
. . O

If IN O
the DT O
SFF NNP B-C
perforates VBZ I-C
the DT I-C
armor NN I-C
, , O
extensive JJ B-C
behind IN I-C
- HYPH I-C
armor NN I-C
damage NN I-C
( -LRB- O
BAD JJ O
, , O
also RB O
called VBN O
behind IN O
- HYPH O
armor NN O
effect NN O
( -LRB- O
BAE NNP O
) -RRB- O
) -RRB- O
occurs VBZ O
. . O

Officers NNS B-C
also RB I-C
notified VBD I-C
the DT I-C
employer NN I-C
and CC I-C
family NN I-C
of IN I-C
the DT I-C
accused VBN I-C
, , O
causing VBG O
serious JJ B-C
damage NN I-C
to IN I-C
their PRP$ I-C
reputations NNS I-C
. . O

The DT O
2013 CD B-C
September NNP I-C
– HYPH I-C
October NNP I-C
seismic JJ I-C
sequence NN I-C
occurred VBD O
21 CD O
km NNS O
off IN O
the DT O
coast NN O
of IN O
the DT O
Valencia NNP O
Gulf NNP O
( -LRB- O
Spain NNP O
) -RRB- O
is VBZ O
probably RB O
the DT O
most RBS O
known VBN O
case NN O
of IN O
induced VBN B-C
seismicity NN I-C
related VBN O
to IN O
Underground NNP B-C
Gas NNP I-C
Storage NNP I-C
operations NNS I-C
( -LRB- O
the DT O
Castor NNP O
Project NNP O
) -RRB- O
. . O

It PRP O
has VBZ O
been VBN O
proposed VBN O
that IN O
low JJ B-C
levels NNS I-C
of IN I-C
Vitamin NN I-C
E NN I-C
, , I-C
carotenoids NNS I-C
, , I-C
and CC I-C
selenium NN I-C
are VBP O
associated VBN O
with IN O
lower JJR B-C
levels NNS I-C
of IN I-C
muscle NN I-C
strength NN I-C
. . O

Although IN O
the DT O
psychosocial JJ O
impacts NNS O
of IN O
epilepsy NN O
may MD O
be VB O
causative JJ O
, , O
there EX O
are VBP O
also RB O
links NNS O
in IN O
the DT O
phenomenology NN B-C
and CC I-C
neurobiology NN I-C
of IN I-C
TLE NN I-C
and CC O
depression NN B-C
. . O

Other JJ O
triggers NNS O
believed VBN O
to TO O
be VB O
important JJ O
in IN O
acute JJ B-C
episodes NNS I-C
of IN I-C
arthritis NN I-C
include VBP O
cool JJ B-C
temperatures NNS I-C
, , O
rapid JJ B-C
changes NNS I-C
in IN I-C
uric NN I-C
acid NN I-C
levels NNS I-C
, , O
acidosis NN B-C
, , O
articular JJ B-C
hydration NN I-C
and CC O
extracellular JJ B-C
matrix NN I-C
proteins NNS I-C
, , O
such JJ O
as IN O
proteoglycans NNS B-C
, , O
collagens NNS B-C
, , O
and CC O
chondroitin NN B-C
sulfate NN I-C
. . O

In IN O
advanced JJ O
cases NNS O
of IN O
silicosis NN B-C
, , O
large JJ B-C
opacity NN I-C
( -LRB- O
> -RRB- O
1 CD O
cm NN O
) -RRB- O
occurs VBZ O
from IN O
coalescence NN B-C
of IN I-C
small JJ I-C
opacities NNS I-C
, , O
particularly RB O
in IN O
the DT O
upper JJ O
lung NN O
zones NNS O
. . O

In IN O
the DT O
United NNP O
States NNP O
, , O
the DT O
Food NNP B-C
Allergen NNP I-C
Labeling NNP I-C
and CC I-C
Consumer NNP I-C
Protection NNP I-C
Act NNP I-C
of IN I-C
2004 CD O
causes VBZ O
people NNS B-C
to TO I-C
be VB I-C
reminded VBN I-C
of IN I-C
allergy NN I-C
problems NNS I-C
every DT I-C
time NN I-C
they PRP I-C
handle VBP I-C
a DT I-C
food NN I-C
package NN I-C
, , O
and CC O
restaurants NNS B-C
have VBP I-C
added VBN I-C
allergen JJ I-C
warnings NNS I-C
to IN I-C
menus NNS I-C
. . O

The DT O
purported VBN O
causes NNS O
of IN O
this DT O
weight NN B-C
gain NN I-C
are VBP O
increased VBN B-C
alcohol NN I-C
intake NN I-C
and CC O
the DT O
consumption NN B-C
of IN I-C
fat JJ I-C
and CC O
carbohydrate NN B-C
- HYPH I-C
rich JJ I-C
cafeteria NN I-C
- HYPH I-C
style NN I-C
food NN I-C
in IN O
university NN O
dormitories NNS O
and CC O
fast JJ B-C
food NN I-C
in IN O
nearby RB O
restaurants NNS O
. . O

While IN O
long RB B-C
- HYPH I-C
standing NN I-C
diabetes NNS I-C
is VBZ O
a DT O
known VBN O
risk NN O
factor NN O
for IN O
pancreatic JJ B-C
cancer NN I-C
( -LRB- O
see VB O
Risk NN O
factors NNS O
) -RRB- O
, , O
the DT O
cancer NN B-C
can MD O
itself PRP O
cause VB O
diabetes NNS O
, , O
in IN O
which WDT O
case NN O
recent JJ O
onset NN O
of IN O
diabetes NNS B-C
could MD O
be VB O
considered VBN O
an DT O
early JJ O
sign NN O
of IN O
the DT O
disease NN O
. . O

The DT O
abundance NN B-C
of IN I-C
flammable JJ I-C
fuel NN I-C
can MD O
cause VB O
an DT O
inferno NN B-C
with IN O
a DT O
single JJ O
spark NN O
. . O

Diabatic JJ B-C
processes NNS I-C
lead VBP O
to IN O
the DT O
stabilization NN B-C
of IN I-C
an DT I-C
air NN I-C
mass NN I-C
approaching VBG O
the DT O
Appalachians NNPS O
. . O

The DT O
effects NNS O
of IN O
the DT O
earthquake NN B-C
were VBD O
felt VBN O
away RB O
in IN O
Bombay NNP O
with IN O
tremors NNS B-C
and CC O
power NN B-C
outages NNS I-C
. . O

According VBG O
to IN O
a DT O
Harvard NNP O
Mental NNP O
Health NNP O
Letter NNP O
, , O
" `` O
Anxiety NNP B-C
usually RB O
has VBZ O
physical JJ O
symptoms NNS O
that WDT O
may MD O
include VB O
a DT O
racing NN B-C
heart NN I-C
, , O
a DT O
dry JJ B-C
mouth NN I-C
, , O
a DT O
shaky JJ B-C
voice NN I-C
, , O
blushing NN B-C
, , O
trembling NN B-C
, , O
sweating NN B-C
, , O
lightheadedness NN B-C
, , O
and CC O
nausea NN B-C
" '' O
. . O

Areas NNS O
of IN O
prior JJ O
ulceration NN B-C
may MD O
leave VB O
behind IN O
permanent JJ B-C
scarring NN I-C
. . O

On IN O
November NNP O
2017 CD O
a DT O
Mw NNP B-C
5.5 CD I-C
struck VBD O
the DT O
city NN O
of IN O
Pohang NNP O
( -LRB- O
South NNP O
Korea NNP O
) -RRB- O
injuring VBG O
several JJ O
people NNS O
and CC O
causing VBG O
extensive JJ B-C
damage NN I-C
, , O
the DT O
proximity NN O
of IN O
the DT O
seismic JJ O
sequence NN O
with IN O
an DT O
EGS NNP O
site NN O
, , O
where WRB O
stimulation NN O
operations NNS O
has VBZ O
taken VBN O
place NN O
few JJ O
months NNS O
prior JJ O
the DT O
earthquake NN O
raised VBD O
the DT O
possibility NN O
that WDT O
raises VBZ O
the DT O
possibility NN O
that IN O
this DT O
earthquake NN O
was VBD O
anthropogenic NN O
. . O

Acquired VBN B-C
thrombocytopenia NN I-C
may MD O
be VB O
associated VBN O
with IN O
a DT O
history NN B-C
of IN I-C
drug NN I-C
use NN I-C
. . O

The DT O
limited JJ B-C
success NN I-C
of IN I-C
outcomes NNS I-C
after IN I-C
surgery NN I-C
has VBZ O
led VBN O
to IN O
a DT O
number NN O
of IN O
trials NNS B-C
that WDT O
were VBD O
running VBG O
in IN O
2014 CD O
to TO O
test VB O
outcomes NNS O
using VBG O
chemotherapy NN O
or CC O
radiochemotherapy NN O
before IN O
surgery NN O
. . O

Individuals NNS O
with IN O
temporal JJ B-C
lobe NN I-C
epilepsy NN I-C
have VBP O
a DT O
higher JJR O
prevalence NN O
of IN O
depression NN B-C
than IN O
the DT O
general JJ O
population NN O
. . O

Alcoholic JJ O
hepatitis NN O
is VBZ O
hepatitis NN B-C
( -LRB- O
inflammation NN O
of IN O
the DT O
liver NN O
) -RRB- O
due IN O
to IN O
excessive JJ B-C
intake NN I-C
of IN I-C
alcohol NN I-C
. . O

These DT O
can MD O
oxidize VB B-C
the DT I-C
UROD NN I-C
substrate NN I-C
uroporphyrinogen NN I-C
, , O
which WDT O
can MD O
result VB O
in IN O
the DT O
inhibition NN B-C
of IN I-C
UROD NN I-C
and CC O
lead VB O
to IN O
deficient JJ B-C
activity NN I-C
of IN I-C
this DT I-C
key JJ I-C
enzyme NN I-C
. . O

Low JJ B-C
- HYPH I-C
energy NN I-C
beta NN I-C
( -LRB- O
sulfur NN O
- , O
35 CD O
, , O
170 CD O
keV NN O
) -RRB- O
produces VBZ O
shallow JJ B-C
ulcers NNS I-C
with IN O
little JJ O
damage NN O
to IN O
dermis NN O
, , O
while IN O
cobalt NN B-C
- HYPH I-C
60 CD I-C
( -LRB- O
310 CD O
keV NN O
) -RRB- O
, , O
caesium NN B-C
- HYPH I-C
137 CD I-C
( -LRB- O
550 CD O
keV NN O
) -RRB- O
, , O
phosphorus- NN B-C
32 CD I-C
( -LRB- O
1.71 CD O
MeV NN O
) -RRB- O
, , O
strontium NN B-C
- HYPH I-C
90 CD I-C
( -LRB- O
650 CD O
keV NN O
) -RRB- O
and CC O
its PRP$ O
daughter NN O
product NN O
yttrium NN B-C
- HYPH I-C
90 CD I-C
( -LRB- O
2.3 CD O
MeV NN O
) -RRB- O
damage NN B-C
deeper JJR I-C
levels NNS I-C
of IN I-C
the DT I-C
dermis NN I-C
and CC O
can MD O
result VB O
in IN O
chronic JJ B-C
radiation NN I-C
dermatitis NN I-C
. . O

Silicosis NN B-C
resulted VBD O
in IN O
46,000 CD O
deaths NNS B-C
globally RB O
in IN O
2013 CD O
down RB O
from IN O
55,000 CD O
deaths NNS O
in IN O
1990 CD O
. . O

Interleukin NNP B-C
- HYPH I-C
1 CD I-C
inhibitors NNS I-C
, , O
such JJ O
as IN O
canakinumab JJ B-C
, , O
showed VBD O
moderate JJ O
effectiveness NN O
for IN O
pain NN B-C
relief NN I-C
and CC O
reduction NN B-C
of IN I-C
joint JJ I-C
swelling NN I-C
, , O
but CC O
have VBP O
increased VBN O
risk NN O
of IN O
adverse JJ B-C
events NNS I-C
, , O
such JJ O
as IN O
back NN B-C
pain NN I-C
, , O
headache NN B-C
, , O
and CC O
increased VBN B-C
blood NN I-C
pressure NN I-C
. . O

Medium JJ B-C
- HYPH I-C
chain NN I-C
and CC I-C
short JJ I-C
- HYPH I-C
chain NN I-C
fatty NN I-C
acids NNS I-C
or CC O
partially RB B-C
metabolized VBN I-C
fatty NN I-C
acids NNS I-C
may MD O
build VB O
up RP O
in IN O
tissues NNS O
and CC O
damage VB O
the DT O
liver NN O
, , O
heart NN O
, , O
and CC O
muscles NNS O
, , O
causing VBG O
more RBR O
serious JJ B-C
complications NNS I-C
. . O

Adie NNP B-C
syndrome NN I-C
presents VBZ O
with IN O
three CD O
hallmark JJ B-C
symptoms NNS I-C
, , O
namely RB O
at RB O
least RBS O
one CD O
abnormally RB B-C
dilated VBN I-C
pupil NN I-C
( -LRB- O
mydriasis NN O
) -RRB- O
which WDT O
does VBZ O
not RB O
constrict VB O
in IN O
response NN O
to IN O
light NN O
, , O
loss NN O
of IN O
deep JJ O
tendon NN O
reflexes NNS O
, , O
and CC O
abnormalities NNS O
of IN O
sweating NN O
. . O

A DT O
vitamin NN B-C
D NNP I-C
deficiency NN I-C
may MD O
also RB O
cause VB O
fasciculations NNS B-C
, , O
stemming VBG O
from IN O
reduced VBN O
ionized VBN O
calcium NN O
in IN O
the DT O
blood NN O
( -LRB- O
hypocalcemia NN O
) -RRB- O
. . O

Twin JJ B-C
chimerism NN I-C
results NNS O
from IN O
mixing NN B-C
of IN I-C
blood NN I-C
between IN I-C
two CD I-C
twin JJ I-C
fetuses NNS I-C
through IN I-C
placental JJ I-C
blood NN I-C
vessel NN I-C
anastomoses NNS I-C
, , O
leading VBG O
to IN O
engraftment NN B-C
of IN I-C
hematopoietic JJ I-C
stem NN I-C
cells NNS I-C
from IN I-C
one CD I-C
twin NN I-C
within IN I-C
the DT I-C
marrow NN I-C
of IN I-C
the DT I-C
other JJ I-C
. . O

Phytophthora NN B-C
a DT O
genus NN O
of IN O
plant NN O
- HYPH O
damaging VBG O
oomycetes NNS O
( -LRB- O
water NN O
molds NNS O
) -RRB- O
, , O
whose WP$ O
member NN O
species NNS O
are VBP O
capable JJ O
of IN O
causing VBG O
enormous JJ B-C
economic JJ I-C
losses NNS I-C
on IN I-C
crops NNS I-C
worldwide RB O
, , O
as RB O
well RB O
as IN O
environmental JJ B-C
damage NN I-C
in IN O
natural JJ O
ecosystems NNS O
. . O

Excess NN B-C
alcohol NN I-C
abuse NN I-C
is VBZ O
frequently RB O
associated VBN O
with IN O
both CC O
inducing VBG O
PCT NN B-C
and CC O
aggravating VBG B-C
a DT I-C
preexisting JJ I-C
diagnosis NN I-C
of IN O
the DT O
disorder NN O
. . O

Additionally RB O
, , O
patients NNS O
will MD O
often RB O
void VB O
a DT O
wine NN B-C
- HYPH I-C
red JJ I-C
color NN I-C
urine NN I-C
with IN O
an DT O
increased VBN B-C
concentration NN I-C
of IN I-C
uroporphyrin NN I-C
I PRP I-C
due IN O
to IN O
their PRP$ O
enzymatic JJ B-C
deficiency NN I-C
. . O

The DT O
theatrical JJ B-C
trailer NN I-C
caused VBD O
even RB O
more JJR O
media NN B-C
attention NN I-C
because IN O
it PRP O
was VBD O
premiered VBN O
in IN O
theaters NNS O
and CC O
screened VBN O
at IN O
the DT O
ShoWest NNP O
Convention NNP O
in IN O
Las NNP O
Vegas NNPS O
, , O
and CC O
was VBD O
aired VBN O
on IN O
Entertainment NNP O
Tonight NN O
and CC O
Access NNP O
Hollywood NNP O
. . O

Desert NNP O
lung NN O
disease NN O
may MD O
be VB O
related VBN O
to IN O
Al NNP B-C
Eskan NNP I-C
disease NN I-C
, , O
a DT O
lung NN O
disorder NN O
thought VBN O
to TO O
be VB O
caused VBN O
by IN O
exposure NN B-C
to IN I-C
sand NN I-C
dust NN I-C
containing VBG O
organic JJ O
antigens NNS O
, , O
which WDT O
was VBD O
first RB O
diagnosed VBN O
after IN O
the DT O
1990 CD O
Gulf NNP O
war NN O
. . O

Those DT O
with IN O
gout NN B-C
are VBP O
at IN O
increased VBN O
risk NN O
of IN O
hypertension NN B-C
, , O
diabetes NNS B-C
mellitus NN I-C
, , O
metabolic NN B-C
syndrome NN I-C
, , O
and CC O
kidney NN B-C
and CC I-C
cardiovascular JJ I-C
disease NN I-C
and CC O
thus RB O
are VBP O
at IN O
increased VBN O
risk NN O
of IN O
death NN B-C
. . O

Pain NN B-C
associated VBN O
with IN O
facet NN B-C
syndrome NN I-C
is VBZ O
often RB O
called VBN O
" `` O
referred VBN O
pain NN O
" '' O
because IN O
symptoms NNS O
do VBP O
not RB O
follow VB O
a DT O
specific JJ O
nerve NN O
root NN O
pattern NN O
and CC O
the DT O
brain NN O
can MD O
have VB O
difficulty NN O
localizing VBG O
the DT O
specific JJ O
area NN O
of IN O
the DT O
spine NN O
that WDT O
is VBZ O
affected VBN O
. . O

The DT O
earliest JJS O
sign NN O
of IN O
a DT O
contracture NN B-C
is VBZ O
a DT O
triangular JJ B-C
“ `` I-C
puckering NN I-C
” '' I-C
of IN I-C
the DT I-C
skin NN I-C
of IN I-C
the DT I-C
palm NN I-C
as IN O
it PRP O
passes VBZ O
over IN O
the DT O
flexor NN O
tendon NN O
just RB O
before IN O
the DT O
flexor NN O
crease NN O
of IN O
the DT O
finger NN O
, , O
at IN O
the DT O
metacarpophalangeal JJ O
( -LRB- O
MCP NN O
) -RRB- O
joint NN O
. . O

Unable JJ B-C
to TO I-C
buy VB I-C
the DT I-C
required VBN I-C
hyperdrive NN I-C
parts NNS I-C
due IN O
to IN O
the DT O
Republic NNP B-C
's POS I-C
currency NN I-C
being VBG I-C
worthless JJ I-C
on IN O
Tatooine NNP O
, , O
Qui NNP O
- HYPH O
Gon NNP O
wagers NNS O
both CC O
the DT O
parts NNS O
and CC O
Anakin NNP O
's POS O
freedom NN O
with IN O
Watto NNP O
in IN O
a DT O
Podrace NNP O
. . O

Treatment NN O
consists NNS O
of IN O
two CD O
phases NNS O
: : O
stopping VBG O
the DT O
urushiol JJ B-C
contact NN I-C
that WDT O
is VBZ O
causing VBG O
the DT O
reaction NN B-C
( -LRB- O
this DT O
must MD O
be VB O
done VBN O
within IN O
minutes NNS O
) -RRB- O
and CC O
, , O
later RB O
, , O
reducing VBG O
the DT O
pain NN O
and CC O
/ , O
or CC O
itching NN O
. . O

A DT O
borderline JJ B-C
iron NN I-C
deficiency NN I-C
has VBZ O
been VBN O
found VBN O
to TO O
have VB O
a DT O
protective JJ O
affect NN O
by IN O
limiting VBG B-C
heme NN I-C
synthesis NN I-C
. . O

A DT O
study NN O
by IN O
Houston NNP O
and CC O
colleagues NNS O
showed VBD O
how WRB O
decreased VBN B-C
protein NN I-C
intake NN I-C
may MD O
increase VB O
the DT O
risk NN O
of IN O
developing VBG O
mobility NN B-C
limitations NNS I-C
later RB O
on RB O
. . O

Additionally RB O
, , O
alcohol NN B-C
has VBZ O
been VBN O
shown VBN O
to TO O
increase VB O
the DT O
activity NN B-C
of IN I-C
the DT I-C
delta-aminolevulinic JJ I-C
acid NN I-C
synthetase NN I-C
( -LRB- O
ALA NN O
synthetase NN O
) -RRB- O
, , O
the DT O
rate NN O
- HYPH O
limiting VBG O
enzymatic JJ O
step NN O
in IN O
heme NN O
synthesis NN O
in IN O
the DT O
mitochondria NN O
, , O
in IN O
rats NNS O
. . O

Acute JJ B-C
radiodermatitis NN I-C
occurs VBZ O
when WRB O
an DT O
" `` O
erythema JJ O
dose NN O
" '' O
of IN O
ionizing NN B-C
radiation NN I-C
is VBZ O
given VBN O
to IN O
the DT O
skin NN O
, , O
after IN O
which WDT O
visible JJ O
erythema NN O
appears VBZ O
up IN O
to IN O
24 CD O
hours NNS O
after RB O
. . O

If IN O
the DT O
exposure NN O
to IN O
beta NNP O
radiation NN O
is VBZ O
intense JJ O
, , O
the DT O
beta NN B-C
burns NNS I-C
may MD O
first RB O
manifest VB O
in IN O
24 CD O
– SYM O
48 CD O
hours NNS O
by IN O
itching NN B-C
and CC O
/ , O
or CC O
burning NN B-C
sensation NN I-C
that WDT O
last JJ O
for IN O
one CD O
or CC O
two CD O
days NNS O
, , O
sometimes RB O
accompanied VBN O
by IN O
hyperaemia NN B-C
. . O

Arteriovenous NN B-C
fistula NN I-C
( -LRB- O
an DT O
abnormal JJ O
connection NN O
or CC O
passageway NN O
between IN O
an DT O
artery NN O
and CC O
a DT O
vein NN O
) -RRB- O
may MD O
cause VB O
chronic JJ B-C
venous JJ I-C
insufficiency NN I-C
even RB O
with IN O
working VBG O
vein NN O
valves NNS O
. . O

Gout NN B-C
is VBZ O
due IN O
to IN O
persistently RB B-C
elevated VBN I-C
levels NNS I-C
of IN I-C
uric NN I-C
acid NN I-C
in IN I-C
the DT I-C
blood NN I-C
. . O

High JJ B-C
infection NN I-C
rates NNS I-C
for IN I-C
diseases NNS I-C
such JJ O
as IN O
respiratory JJ B-C
infections NNS I-C
, , O
malaria NN B-C
, , O
tuberculosis NN B-C
, , O
as RB O
well RB O
as IN O
sexually RB B-C
transmitted VBN I-C
diseases NNS I-C
like IN O
HIV NNP B-C
/ , I-C
AIDS NNP I-C
are VBP O
the DT O
leading VBG O
causes NNS O
of IN O
death NN B-C
for IN O
Haitians NNPS O
. . O

That DT O
binder NN O
contained VBN O
polyurethane NN B-C
that WDT O
soaks VBZ O
up RP O
water NN O
and CC O
causes VBZ O
the DT O
urethane NN B-C
to TO I-C
rise VB I-C
to IN I-C
the DT I-C
tape NN I-C
's POS I-C
surface NN I-C
. . O

Magnesium NN B-C
deficiency NN I-C
can MD O
cause VB O
both DT O
fasciculations NNS B-C
and CC O
anxiety NN B-C
. . O

Additionally RB O
, , O
it PRP O
showed VBD O
that IN O
college NN O
students NNS O
did VBD O
not RB O
gain VB O
any DT O
more JJR O
weight NN O
than IN O
non-college JJ O
students NNS O
of IN O
the DT O
same JJ O
age NN O
, , O
and CC O
that IN O
the DT O
only JJ O
factor NN O
that WDT O
increased VBD B-C
weight NN I-C
gain NN I-C
was VBD O
heavy JJ B-C
drinking VBG I-C
. . O

Bleeding VBG B-C
from IN I-C
lung NN I-C
AVMs NNS I-C
is VBZ O
relatively RB O
unusual JJ O
, , O
but CC O
may MD O
cause VB O
hemoptysis NN B-C
( -LRB- O
coughing VBG O
up RP O
blood NN O
) -RRB- O
or CC O
hemothorax NN B-C
( -LRB- O
blood NN O
accumulating VBG O
in IN O
the DT O
chest NN O
cavity NN O
) -RRB- O
. . O

Stereotyped JJ B-C
individuals NNS I-C
who WP O
receive VBP O
negative JJ O
feedback NN O
can MD O
attribute VB O
it PRP O
either CC O
to IN O
personal JJ B-C
shortcomings NNS I-C
, , O
such JJ O
as IN O
lack NN B-C
of IN I-C
ability NN I-C
or CC O
poor JJ B-C
effort NN I-C
, , O
or CC O
the DT O
evaluator NN B-C
's POS I-C
stereotypes NNS I-C
and CC O
prejudice NN B-C
toward IN I-C
their PRP$ I-C
social JJ I-C
group NN I-C
. . O

Chronic NN B-C
venous JJ I-C
insufficiency NN I-C
caused VBN O
by IN O
DVT NN B-C
may MD O
be VB O
described VBN O
as IN O
postthrombotic JJ O
syndrome NN O
. . O

The DT O
main JJ O
complication NN O
resulting VBG O
from IN O
pseudocholinesterase JJ B-C
deficiency NN I-C
is VBZ O
the DT O
possibility NN O
of IN O
respiratory JJ B-C
failure NN I-C
secondary JJ O
to IN O
succinylcholine JJ B-C
or CC O
mivacurium JJ B-C
- HYPH O
induced VBN O
neuromuscular JJ B-C
paralysis NN I-C
. . O

The DT O
extra JJ B-C
water NN I-C
pressure NN I-C
created VBN O
by IN O
large JJ B-C
reservoirs NNS I-C
is VBZ O
the DT O
most RBS O
accepted VBN O
explanation NN O
for IN O
the DT O
seismic JJ B-C
activity NN I-C
. . O

In IN O
addition NN O
, , O
carbon NN B-C
capture NN I-C
and CC I-C
storage—a JJ I-C
technology NN O
for IN O
storing VBG O
excess JJ O
carbon NN O
dioxide NN O
underground JJ O
—may NN O
have VBP O
the DT O
potential NN O
for IN O
inducing VBG O
seismic JJ B-C
events NNS I-C
, , O
because IN O
significant JJ O
volumes NNS O
of IN O
fluids NNS O
are VBP O
injected VBN O
underground JJ O
over IN O
long JJ O
periods NNS O
of IN O
time NN O
. . O

Self PRP O
- HYPH O
harm NN O
may MD O
be VB O
used VBN O
as IN O
an DT O
alternative NN O
to IN O
compensating VBG O
the DT O
object NN O
of IN O
one PRP O
's POS O
transgression NN O
– , O
perhaps RB O
in IN O
the DT O
form NN O
of IN O
not RB B-C
allowing VBG I-C
oneself PRP I-C
to TO I-C
enjoy VB I-C
opportunities NNS I-C
open JJ I-C
to IN I-C
one CD I-C
, , O
or CC O
benefits NNS O
due IN O
, , O
as IN O
a DT O
result NN O
of IN O
uncompensated JJ B-C
guilt NN I-C
feelings NNS I-C
. . O

Fluoroscopy NNP B-C
may MD O
cause VB O
burns NNS B-C
if IN O
performed VBN O
repeatedly RB O
or CC O
for IN O
too RB O
long RB O
. . O

MG NNP B-C
may MD O
be VB O
limited VBN O
to IN O
the DT O
muscles NNS O
of IN O
the DT O
eye NN O
( -LRB- O
ocular JJ O
MG NN O
) -RRB- O
, , O
leading VBG O
to IN O
abrupt JJ B-C
onset NN I-C
of IN I-C
weakness NN I-C
/ , O
fatigability NN B-C
of IN I-C
the DT I-C
eyelids NNS I-C
or CC I-C
eye NN I-C
movement NN I-C
. . O

PanNETs NNS B-C
have VBP O
been VBN O
associated VBN O
with IN O
multiple JJ B-C
endocrine JJ I-C
neoplasia NN I-C
type NN I-C
1 CD I-C
( -LRB- O
MEN1 NN O
) -RRB- O
and CC O
von NNP B-C
Hippel NNP I-C
Lindau NNP I-C
syndromes NNS I-C
. . O

Large JJ B-C
segmental JJ I-C
hemangiomas NNS I-C
over IN I-C
the DT I-C
lumbar NN I-C
spine NN I-C
can MD O
be VB O
associated VBN O
with IN O
dysraphism NN B-C
, , O
renal JJ B-C
, , I-C
and CC I-C
urogenital JJ I-C
problems NNS I-C
in IN O
association NN O
with IN O
a DT O
disorder NN O
called VBN O
LUMBAR NNP B-C
syndrome NN I-C
. . O

There EX O
are VBP O
5 CD O
mutations NNS B-C
in IN I-C
EFHC1 NNP I-C
associated VBN O
with IN O
JME NNP B-C
; , O
D210N NNP B-C
, , O
F229L NNP B-C
, , O
D253Y CD B-C
, , O
P77T NN B-C
and CC O
R221H NN B-C
. . O

Two CD O
legal JJ O
scholars NNS O
have VBP O
also RB O
questioned VBN O
whether IN O
Congress NNP B-C
has VBZ O
contributed VBN O
to IN O
the DT O
proliferation NN B-C
of IN I-C
czars NNS I-C
. . O

This DT O
oxidative NN B-C
stress NN I-C
then RB O
leads VBZ O
to IN O
inhibition NN B-C
of IN I-C
uroporphyrinogen NN I-C
decarboxylase NN I-C
, , O
creating VBG O
an DT O
excess NN B-C
of IN I-C
uroporphyrinogen NN I-C
III CD I-C
which WDT O
is VBZ O
oxidized VBN O
from IN O
the DT O
relatively RB O
harmless JJ O
porphyrinogens NNS O
into IN O
their PRP$ O
reduced VBN O
porphyrins NNS O
form NN O
. . O

Firefighters NNS O
often RB O
get VBP O
rashes NNS B-C
and CC O
eye NN B-C
inflammation NN I-C
from IN O
smoke NN B-C
- HYPH I-C
related JJ I-C
contact NN I-C
. . O

Either CC O
top JJ O
- HYPH O
down NN O
fashion NN O
activates VBZ O
schemas NNS O
, , O
where WRB O
intentions NNS O
are VBP O
governed VBN O
by IN O
some DT O
type NN O
of IN O
cognitive JJ O
system NN O
, , O
or CC O
by IN O
bottom NN O
- HYPH O
up NN O
fashion NN O
where WRB O
features NNS B-C
or CC I-C
an DT I-C
object NN I-C
in IN I-C
the DT I-C
environment NN I-C
trigger VBP O
a DT O
schema NN B-C
to TO I-C
begin VB I-C
. . O

The DT O
swelling NN B-C
is VBZ O
capable JJ O
of IN O
blocking VBG B-C
nearby RB I-C
biliary JJ I-C
ducts NNS I-C
, , O
leading VBG O
to IN O
diffuse JJ B-C
cholestasis NN I-C
. . O

The DT O
colonels NNS B-C
had VBD I-C
to TO I-C
withdraw VB I-C
due IN O
to IN O
the DT O
unauthorized JJ B-C
flight NN I-C
of IN I-C
many JJ I-C
of IN I-C
the DT I-C
Cossacks NNPS I-C
. . O

Multiple JJ B-C
gynecological JJ I-C
or CC I-C
rectal JJ I-C
surgeries NNS I-C
can MD O
also RB O
lead VB O
to IN O
weakening NN B-C
of IN I-C
the DT I-C
pelvic JJ I-C
floor NN I-C
. . O

First RB O
, , O
the DT O
patient JJ O
history NN O
should MD O
reveal VB O
exposure NN B-C
to IN I-C
sufficient JJ I-C
silica NN I-C
dust NN I-C
to TO O
cause VB O
this DT O
illness NN B-C
. . O

The DT O
incidence NN O
of IN O
deaths NNS B-C
due IN O
to IN O
silicosis NN B-C
declined VBD O
by IN O
84 CD O
% NN O
between IN O
1968 CD O
and CC O
1999 CD O
, , O
and CC O
only RB O
187 CD O
deaths NNS B-C
in IN O
1999 CD O
had VBD O
silicosis NN B-C
as IN O
the DT O
underlying JJ O
or CC O
contributing NN O
cause NN O
. . O

However RB O
, , O
unlike IN O
the DT O
permanent JJ B-C
hardness NN I-C
caused VBN O
by IN O
sulphate NN B-C
and CC I-C
chloride NN I-C
compounds NNS I-C
, , O
this DT O
" `` O
temporary JJ O
" '' O
hardness NN O
can MD O
be VB O
reduced VBN O
either CC O
by IN O
boiling VBG O
the DT O
water NN O
, , O
or CC O
by IN O
the DT O
addition NN O
of IN O
lime NN O
( -LRB- O
calcium NN O
hydroxide NN O
) -RRB- O
through IN O
the DT O
process NN O
of IN O
lime NN O
softening NN O
. . O

AGEP NNP B-C
is VBZ O
an DT O
acute JJ O
drug NN O
eruption NN O
characterized VBN O
by IN O
numerous JJ O
small JJ O
, , O
primarily RB O
non-follicular JJ O
, , O
sterile JJ B-C
skin NN I-C
pustules NNS I-C
arising VBG O
within IN O
large JJ O
areas NNS O
of IN O
red JJ O
swollen JJ O
skin NN O
usually RB O
within IN O
days NNS O
of IN O
taking VBG O
an DT O
inciting VBG O
drug NN O
. . O

If IN O
the DT O
impaired JJ B-C
vein NN I-C
function NN I-C
causes VBZ O
significant JJ B-C
symptoms NNS I-C
, , O
such JJ O
as IN O
swelling NN B-C
and CC O
ulcer NN B-C
formation NN I-C
, , O
it PRP O
is VBZ O
referred VBN O
to IN O
as IN O
chronic JJ O
venous JJ O
disease NN O
. . O

High JJ B-C
exposure NN I-C
to IN I-C
X NN I-C
- HYPH I-C
rays NNS I-C
during IN O
diagnostic JJ O
medical JJ O
imaging NN O
or CC O
radiotherapy NN O
can MD O
also RB O
result VB O
in IN O
radiation NN B-C
burns NNS I-C
. . O

Underexcretion NN B-C
of IN I-C
uric NN I-C
acid NN I-C
by IN I-C
the DT I-C
kidney NN I-C
is VBZ O
the DT O
primary JJ O
cause NN O
of IN O
hyperuricemia NN B-C
in IN O
about RB O
90 CD O
% NN O
of IN O
cases NNS O
, , O
while IN O
overproduction NN B-C
is VBZ O
the DT O
cause NN O
in IN O
less JJR O
than IN O
10% CD O
. . O

One CD O
cause NN O
that WDT O
has VBZ O
resulted VBN O
in IN O
significant JJ O
levels NNS O
of IN O
structural JJ B-C
violence NN I-C
within IN O
Haiti NNP O
is VBZ O
political JJ B-C
instability NN I-C
and CC O
corruption NN B-C
. . O

Further JJ O
funneling NN B-C
of IN I-C
cool JJ I-C
air NN I-C
occurs VBZ I-C
within IN I-C
the DT I-C
Isthmus NNP I-C
, , O
which WDT O
can MD O
lead VB O
to IN O
winds NNS B-C
of IN I-C
gale NNP I-C
and CC I-C
hurricane JJ I-C
- HYPH I-C
force NN I-C
, , O
referred VBN O
to IN O
as IN O
a DT O
Tehuantepecer NNP O
. . O

Complicated JJ O
silicosis NN O
is VBZ O
more RBR O
common JJ O
with IN O
accelerated VBN O
silicosis NN O
than IN O
with IN O
the DT O
chronic JJ O
variety NN O
. . O
Acute JJ B-C
silicosis NN I-C
: : O
Silicosis NN O
that WDT O
develops VBZ O
a DT O
few JJ O
weeks NNS O
to IN O
5 CD O
years NNS O
after IN O
exposure NN B-C
to IN I-C
high JJ I-C
concentrations NNS I-C
of IN I-C
respirable JJ I-C
silica NN I-C
dust NN I-C
. . O

If IN O
the DT O
flow NN B-C
in IN I-C
the DT I-C
AVM NN I-C
is VBZ I-C
in IN I-C
the DT I-C
other JJ I-C
direction NN I-C
, , O
portal JJ B-C
venous JJ I-C
blood NN I-C
flows VBZ I-C
directly RB I-C
into IN I-C
the DT I-C
veins NNS I-C
rather RB O
than IN O
running VBG O
through IN O
the DT O
liver NN O
; , O
this DT O
may MD O
lead VB O
to IN O
hepatic JJ B-C
encephalopathy NN I-C
( -LRB- O
confusion NN O
due IN O
to IN O
portal JJ O
waste NN O
products NNS O
irritating VBG O
the DT O
brain NN O
) -RRB- O
. . O

About RB O
90 CD O
% NN O
of IN O
AGEP NN B-C
reactions NNS I-C
are VBP O
associated VBN O
with IN O
medications NNS B-C
. . O

Some DT O
degree NN O
of IN O
non-cyclical JJ B-C
breast NN I-C
tenderness NN I-C
can MD O
normally RB O
be VB O
present JJ O
due IN O
to IN O
hormonal JJ B-C
changes NNS I-C
in IN O
puberty NN O
( -LRB- O
both CC O
in IN O
girls NNS O
and CC O
boys NNS O
) -RRB- O
, , O
in IN O
menopause NN O
and CC O
during IN O
pregnancy NN O
. . O

Additional JJ O
" `` O
contributing VBG O
factors NNS O
" '' O
were VBD O
the DT O
Navigator NNP O
calling VBG O
out RP O
radar NN O
altitudes NNS O
without IN O
considering VBG O
the DT O
uneven JJ O
terrain NN O
in IN O
the DT O
area NN O
, , O
utilization NN O
of IN O
the DT O
autopilot NN O
and CC O
autothrottles NNS O
much RB O
lower JJR O
than IN O
minimum JJ O
descent NN O
altitude NN O
which WDT O
did VBD O
not RB O
comply VB O
with IN O
the DT O
Flight NNP O
Crew NNP O
Operations NNPS O
Manual NNP O
for IN O
the DT O
Tu NNP O
- HYPH O
154 CD O
, , O
and CC O
the DT O
late JJ B-C
start NN I-C
of IN I-C
the DT I-C
final JJ I-C
descent NN I-C
which WDT O
caused VBD O
the DT O
crew NN B-C
to TO I-C
maintain VB I-C
a DT I-C
higher JJR I-C
than IN I-C
normal JJ I-C
vertical JJ I-C
speed NN I-C
. . O

The DT O
central JJ O
plot NN O
is VBZ O
Palpatine NNP B-C
's POS I-C
intent NN I-C
to TO I-C
become VB I-C
Chancellor NNP I-C
, , O
which WDT O
leads VBZ O
to IN O
the DT O
Trade NNP B-C
Federation NNP I-C
's POS I-C
attack NN I-C
on IN I-C
Naboo NNP I-C
, , O
the DT O
Jedi NNP B-C
being VBG I-C
sent VBN I-C
there RB I-C
, , O
Anakin NNP B-C
being VBG I-C
met VBN I-C
along IN I-C
the DT I-C
way NN I-C
, , O
and CC O
the DT O
rise NN B-C
of IN I-C
the DT I-C
Sith NNP I-C
Lords NNPS I-C
. . O

People NNS O
who WP O
drink VBP O
large JJ B-C
amounts NNS I-C
of IN I-C
alcohol NN I-C
have VBP O
a DT O
good JJ O
chance NN O
of IN O
becoming VBG B-C
malnourished VBN I-C
or CC O
losing VBG B-C
an DT I-C
unhealthy JJ I-C
amount NN I-C
of IN I-C
weight NN I-C
because IN O
of IN O
the DT O
absorption NN B-C
blocking VBG I-C
qualities NNS I-C
of IN I-C
alcohol NN I-C
. . O

If IN O
located VBN O
near IN O
the DT O
eye NN O
, , O
a DT O
growing VBG B-C
hemangioma NN I-C
may MD O
cause VB O
an DT O
occlusion NN B-C
or CC O
deviation NN B-C
of IN I-C
the DT I-C
eye NN I-C
that WDT O
can MD O
lead VB O
to IN O
amblyopia NN B-C
. . O

Embolization NN B-C
treatment NN I-C
has VBZ O
been VBN O
attempted VBN O
, , O
but CC O
leads VBZ O
to IN O
severe JJ B-C
complications NNS I-C
in IN O
a DT O
proportion NN O
of IN O
patients NNS O
and CC O
is VBZ O
discouraged VBN O
. . O

Serum NN B-C
sickness NN I-C
can MD O
be VB O
developed VBN O
as IN O
a DT O
result NN O
of IN O
exposure NN B-C
to IN I-C
antibodies NNS I-C
derived VBN I-C
from IN I-C
animals NNS I-C
. . O

Dynapenia NNP B-C
( -LRB- O
pronounced VBN O
dahy NN O
- HYPH O
nuh NN O
- HYPH O
pē NN O
- HYPH O
nē- NN O
a DT O
, , O
Greek JJ O
translation NN O
for IN O
poverty NN O
of IN O
strength NN O
, , O
power NN O
, , O
or CC O
force NN O
) -RRB- O
is VBZ O
the DT O
loss NN O
of IN O
muscular JJ O
strength NN O
not RB O
caused VBN O
by IN O
neurological JJ B-C
or CC I-C
muscular JJ I-C
disease NN I-C
that WDT O
typically RB O
is VBZ O
associated VBN O
with IN O
older JJR O
adults NNS O
. . O

Signs NNS O
and CC O
symptoms NNS O
include VBP O
: : O
Dyspnea NN O
( -LRB- O
shortness NN O
of IN O
breath NN O
) -RRB- O
exacerbated VBN O
by IN O
exertion NN O
Cough NN O
, , O
often RB O
persistent JJ O
and CC O
sometimes RB O
severe JJ O
Fatigue NN O
Tachypnea NN O
( -LRB- O
rapid JJ O
breathing NN O
) -RRB- O
which WDT O
is VBZ O
often RB O
labored VBN O
, , O
Loss NN O
of IN O
appetite NN O
and CC O
weight NN O
loss NN O
Chest NN O
pain NN O
Fever NN O
Gradual JJ O
darkening NN O
of IN O
skin NN O
( -LRB- O
blue JJ O
skin NN O
) -RRB- O
Gradual JJ B-C
dark JJ I-C
shallow JJ I-C
rifts NNS I-C
in IN I-C
nails NNS I-C
eventually RB O
leading VBG O
to IN O
cracks NNS B-C
as IN O
protein NN O
fibers NNS O
within IN O
nail NN O
beds NNS O
are VBP O
destroyed VBN O
. . O

The DT O
R482X NN B-C
mutation NN I-C
also RB O
increases VBZ O
the DT O
rate NN O
of IN O
fast JJ B-C
inactivation NN I-C
of IN I-C
the DT I-C
channel NN I-C
. . O

Depending VBG O
on IN O
the DT O
photon NN O
energy NN O
, , O
gamma JJ B-C
radiation NN I-C
can MD O
cause VB O
very RB O
deep JJ B-C
gamma JJ I-C
burns NNS I-C
, , O
with IN O
60 CD O
Co NN O
internal JJ O
burns NNS O
are VBP O
common JJ O
. . O

Mutations NNS B-C
in IN I-C
the DT I-C
MADH4 NN I-C
gene NN I-C
is VBZ O
usually RB O
associated VBN O
with IN O
juvenile JJ B-C
polyposis NN I-C
, , O
and CC O
detection NN O
of IN O
such PDT O
a DT O
mutation NN O
would MD O
indicate VB O
a DT O
need NN O
to TO O
screen VB O
the DT O
patient NN O
and CC O
affected VBN O
relatives NNS O
for IN O
polyps NNS O
and CC O
tumors NNS O
of IN O
the DT O
large JJ O
intestine NN O
. . O

The DT O
inflammatory JJ B-C
effects NNS I-C
of IN O
crystalline JJ B-C
silica NN I-C
are VBP O
apparently RB O
mediated VBN O
by IN O
the DT O
NALP3 NNP O
inflammasome NN O
. . O

People NNS B-C
suffer VBP I-C
from IN I-C
hunger NN I-C
because IN O
of IN O
a DT O
lack NN B-C
of IN I-C
food NN I-C
and CC O
the DT O
nutrients NNS O
which WDT O
accompany VBP O
food NN O
in IN O
the DT O
short JJ O
term NN O
. . O

Individual JJ B-C
susceptibility NN I-C
to IN I-C
fluorosis NN I-C
is VBZ O
also RB O
influenced VBN O
by IN O
genetic JJ B-C
factors NNS I-C
. . O

Haiti NNP B-C
's POS I-C
overall JJ I-C
low JJ I-C
ranking NN I-C
for IN I-C
life NN I-C
expectancy NN I-C
stems VBZ O
from IN O
a DT O
multitude NN O
of IN O
factors NNS O
that WDT O
includes VBZ O
high JJ B-C
HIV NN I-C
/ , I-C
AIDS NN I-C
prevalence NN I-C
, , O
lack NN B-C
of IN I-C
access NN I-C
to IN I-C
basic JJ I-C
healthcare NN I-C
services NNS I-C
, , O
malnutrition NN B-C
, , O
and CC O
poor JJ B-C
sanitation NN I-C
. . O

The DT O
resulting VBG O
increase NN B-C
in IN I-C
subsurface JJ I-C
pore NN I-C
pressure NN I-C
can MD O
trigger VB O
movement NN B-C
along IN I-C
faults NNS I-C
, , O
resulting VBG O
in IN O
earthquakes NNS B-C
. . O

Diabetes NNS B-C
mellitus NN I-C
is VBZ O
a DT O
risk NN O
factor NN O
for IN O
pancreatic JJ B-C
cancer NN I-C
and CC O
( -LRB- O
as IN O
noted VBN O
in IN O
the DT O
Signs NNS O
and CC O
symptoms NNS O
section NN O
) -RRB- O
new JJ B-C
- HYPH I-C
onset NN I-C
diabetes NNS I-C
may MD O
also RB O
be VB O
an DT O
early JJ O
sign NN O
of IN O
the DT O
disease NN O
. . O

The DT O
injection NN B-C
of IN I-C
supercritical JJ I-C
CO2 NN I-C
will MD O
change VB B-C
the DT I-C
stresses NNS I-C
in IN I-C
the DT I-C
reservoir NN I-C
as IN O
it PRP O
expands VBZ O
, , O
causing VBG O
potential JJ B-C
failure NN I-C
on IN I-C
nearby JJ I-C
faults NNS I-C
. . O

More JJR O
than IN O
80 CD O
% NN O
of IN O
all DT O
cases NNS O
of IN O
HHT NN B-C
are VBP O
due IN O
to IN O
mutations NNS B-C
in IN I-C
either CC I-C
ENG NNP I-C
or CC I-C
ACVRL1 NNP I-C
. . O

There EX O
are VBP O
three CD O
specific JJ O
types NNS O
of IN O
radiodermatitis NN O
: : O
acute JJ O
radiodermatitis NN O
, , O
chronic NN O
radiodermatitis NN O
, , O
and CC O
eosinophilic JJ O
, , O
polymorphic JJ O
, , O
and CC O
pruritic JJ B-C
eruption NN I-C
associated VBN O
with IN O
radiotherapy NN B-C
. . O

This DT O
increase NN B-C
in IN I-C
temperature NN I-C
can MD O
cause VB O
scaling NN B-C
, , O
especially RB O
in IN O
cases NNS O
such JJ O
as IN O
hot JJ O
water NN O
heaters NNS O
. . O

A DT O
common JJ O
cause NN O
for IN O
Weaver NNP B-C
syndrome NN I-C
is VBZ O
mutations NNS B-C
in IN I-C
the DT I-C
EZH2 NN I-C
gene NN I-C
on IN O
chromosome NN O
7q36 CD O
. . O

Women NNS B-C
are VBP I-C
often RB I-C
started VBN I-C
on IN I-C
alternative JJ I-C
medications NNS I-C
due IN O
to IN O
valproic NN B-C
acid NN I-C
's POS I-C
high JJ I-C
incidence NN I-C
of IN I-C
fetal JJ I-C
malformations NNS I-C
. . O

This DT O
process NN O
takes VBZ O
place NN O
when WRB O
the DT O
body NN B-C
releases VBZ I-C
adrenaline NN I-C
into IN I-C
the DT I-C
blood NN I-C
stream NN I-C
causing VBG O
a DT O
chain NN B-C
of IN I-C
reactions NNS I-C
to TO O
occur VB O
. . O

Asian JJ B-C
glacial JJ I-C
melting NN I-C
could MD O
lead VB O
to IN O
water NN B-C
shortages NNS I-C
and CC O
floods NNS B-C
for IN O
the DT O
hundreds NNS O
of IN O
millions NNS O
of IN O
people NNS O
who WP O
live VBP O
downstream RB O
. . O

Beta NNP B-C
burns NNS I-C
are VBP O
frequently RB O
the DT O
result NN O
of IN O
exposure NN O
to IN O
radioactive JJ B-C
fallout NN I-C
after IN O
nuclear JJ B-C
explosions NNS I-C
or CC O
nuclear JJ B-C
accidents NNS I-C
. . O

This DT O
constitutes VBZ O
our PRP$ O
powerful JJ O
system NN B-C
of IN I-C
empathy NN I-C
, , O
which WDT O
leads VBZ O
to IN O
our PRP$ B-C
thinking NN I-C
that IN I-C
we PRP I-C
should MD I-C
do VB I-C
something NN I-C
to TO I-C
relieve VB I-C
the DT I-C
suffering NN I-C
of IN I-C
others NNS I-C
. . O

Radiation NN B-C
therapy NN I-C
sources NNS I-C
can MD O
cause VB O
beta NN B-C
burns NNS I-C
during IN O
exposure NN O
of IN O
the DT O
patients NNS O
. . O

A DT O
result NN O
of IN O
this DT O
rubbing NN B-C
is VBZ O
often RB O
swelling VBG B-C
, , O
inflammation NN B-C
or CC O
other JJ O
painful JJ B-C
symptoms NNS I-C
. . O

Ingestion NN B-C
of IN I-C
under IN I-C
- HYPH I-C
cooked VBN I-C
egg NN I-C
may MD O
trigger VB O
more RBR O
severe JJ B-C
clinical JJ I-C
reactions NNS I-C
than IN O
well RB O
- HYPH O
cooked VBN O
egg NN O
. . O

Late JJ B-C
- HYPH I-C
phase NN I-C
responses NNS I-C
seen VBN O
in IN O
asthma NN O
are VBP O
slightly RB O
different JJ O
from IN O
those DT O
seen VBN O
in IN O
other JJ O
allergic JJ O
responses NNS O
, , O
although IN O
they PRP O
are VBP O
still RB O
caused VBN O
by IN O
release NN B-C
of IN I-C
mediators NNS I-C
from IN I-C
eosinophils NNS I-C
. . O

Dental JJ B-C
fluorosis NN I-C
is VBZ O
caused VBN O
by IN O
a DT O
higher JJR B-C
than IN I-C
normal JJ I-C
amount NN I-C
of IN I-C
fluoride NN I-C
ingestion NN I-C
whilst IN I-C
teeth NNS I-C
are VBP I-C
forming VBG I-C
. . O

One CD O
factor NN O
being VBG O
the DT O
positive JJ O
correlation NN O
between IN O
higher JJR B-C
intensity NN I-C
resistance NN I-C
training NN I-C
and CC O
greater JJR O
improvements NNS O
in IN O
muscle NN B-C
strength NN I-C
. . O

Though IN O
blisters NNS B-C
are VBP O
the DT O
most RBS O
common JJ O
skin NN O
manifestations NNS O
of IN O
PCT NN B-C
, , O
other JJ O
skin NN O
manifestations NNS O
like IN O
hyperpigmentation NN B-C
( -LRB- O
as IN O
if IN O
they PRP O
are VBP O
getting VBG O
a DT O
tan NN O
) -RRB- O
and CC O
hypertrichosis NN B-C
( -LRB- O
mainly RB O
on IN O
top NN O
of IN O
the DT O
cheeks NNS O
) -RRB- O
also RB O
occur VB O
. . O

Temporal JJ B-C
lobe NN I-C
epilepsy NN I-C
is VBZ O
associated VBN O
with IN O
memory NN B-C
disorders NNS I-C
and CC O
loss NN B-C
of IN I-C
memory NN I-C
. . O

Less RBR O
commonly RB O
, , O
systemic JJ B-C
envenoming NN I-C
is VBZ O
heralded VBN O
by IN O
swollen JJ B-C
or CC I-C
tender JJ I-C
regional JJ I-C
lymph NN I-C
nodes NNS I-C
; , O
associated VBN O
features NNS O
include VBP O
malaise NN B-C
, , O
nausea NN B-C
, , O
vomiting NN B-C
, , O
abdominal JJ B-C
or CC O
chest NN B-C
pain NN I-C
, , O
generalised VBN B-C
sweating NN I-C
, , O
headache NN B-C
, , O
fever NN B-C
, , O
hypertension NN B-C
and CC O
tremor NN B-C
. . O

Ions NNS B-C
causing VBG O
permanent JJ B-C
hardness NN I-C
of IN O
water NN O
can MD O
be VB O
removed VBN O
using VBG O
a DT O
water NN O
softener NN O
, , O
or CC O
ion NN O
exchange NN O
column NN O
. . O

Reactive JJ O
armor NN O
attempts VBZ O
to TO O
defeat VB O
this DT O
with IN O
an DT O
outward JJ B-C
directed VBN I-C
explosion NN I-C
under IN I-C
the DT I-C
impact NN I-C
point NN I-C
, , O
causing VBG O
the DT O
jet NN B-C
to TO I-C
deform VB I-C
and CC O
so RB O
greatly RB B-C
reducing VBG I-C
penetrating VBG I-C
power NN I-C
. . O

A DT O
study NN O
conducted VBN O
in IN O
1995 CD O
identified VBN O
of IN O
HLA NNP B-C
- HYPH I-C
B51 NNP I-C
, , O
HLA NNP B-C
- HYPH I-C
DR11 NNP I-C
, , O
and CC O
HLA NNP B-C
- HYPH I-C
DQ3 NN I-C
of IN O
unknown JJ O
serotypes NNS O
to TO O
be VB O
associated VBN O
with IN O
development NN O
of IN O
AGEP NNP B-C
but CC O
the DT O
results NNS O
have VBP O
not RB O
been VBN O
confirmed VBN O
, , O
expanded VBN O
to TO O
identify VB O
the DT O
serotypes NNS O
involved VBN O
, , O
nor CC O
therefore RB O
useful JJ O
in IN O
identifying VBG O
individuals NNS O
predisposed JJ O
to TO O
develop VB O
AGEP NNP O
in IN O
response NN O
to IN O
any DT O
drug NN O
. . O

In IN O
conjunction NN O
with IN O
degenerative JJ O
disc NN O
disease NN O
, , O
a DT O
distinct JJ O
but CC O
functionally RB O
related JJ O
condition NN O
, , O
facet NN B-C
syndrome NN I-C
is VBZ O
believed VBN O
to TO O
be VB O
one CD O
of IN O
the DT O
most RBS O
common JJ O
causes NNS O
of IN O
lower JJR B-C
back NN I-C
pain NN I-C
. . O

Pneumatic NN B-C
drilling NN I-C
in IN I-C
mines NNS I-C
and CC O
less RBR O
commonly RB O
, , O
mining NN B-C
using VBG I-C
explosives NNS I-C
, , O
would MD O
raise VB O
fine JJ B-C
- HYPH I-C
ultra JJ I-C
fine JJ I-C
crystalline JJ I-C
silica NN I-C
dust NN I-C
( -LRB- O
rock NN O
dust NN O
) -RRB- O
. . O

A DT O
widespread JJ O
tendency NN B-C
to TO I-C
avoid VB I-C
government NN I-C
- HYPH I-C
run VBN I-C
public JJ I-C
facilities NNS I-C
is VBZ O
frequently RB O
credited VBN O
to IN O
those DT O
institutions NNS B-C
' POS I-C
low JJ I-C
quality NN I-C
of IN I-C
care NN I-C
, , O
such JJ O
as IN O
a DT O
lack NN B-C
of IN I-C
equipment NN I-C
and CC I-C
services NNS I-C
, , O
and CC O
unfriendly JJ B-C
attitude NN I-C
of IN I-C
the DT I-C
medical JJ I-C
staff NN I-C
. . O

The DT O
results NNS O
showed VBD O
that IN O
students NNS O
that WDT O
had VBD O
higher JJR B-C
levels NNS I-C
of IN I-C
stress NN I-C
were VBD O
more RBR O
likely JJ O
to TO O
have VB O
NES NNP B-C
due IN O
to IN O
the DT O
inability NN O
to TO O
adapt VB O
. . O

Alcohol NN B-C
also RB O
makes VBZ O
it PRP O
harder JJR O
for IN O
the DT O
body NN O
to TO O
burn VB O
fat NN O
that WDT O
is VBZ O
already RB O
present JJ O
by IN O
slowing VBG B-C
down RP I-C
a DT I-C
process NN I-C
called VBN I-C
" `` I-C
liquid NN I-C
oxidation NN I-C
. . I-C
" '' I-C

Even RB O
though IN O
only RB O
5 CD O
% NN O
of IN O
the DT O
test NN O
subjects NNS O
showed VBD O
a DT O
weight NN O
gain NN O
of IN O
15 CD O
lbs NNS O
or CC O
greater JJR O
the DT O
authors NNS O
of IN O
this DT O
study NN O
concluded VBD O
that IN O
the DT O
freshmen NNS B-C
year NN I-C
in IN O
college NN O
has VBZ O
potential NN O
for IN O
weight NN B-C
gain NN I-C
and CC O
can MD O
even RB O
lead VB O
to IN O
obesity NN B-C
later RB O
on RB O
in IN O
life NN O
. . O

Surgical JJ B-C
excision NN I-C
has VBZ I-C
now RB I-C
essentially RB I-C
been VBN I-C
abandoned VBN I-C
due IN O
to IN O
the DT O
success NN B-C
of IN I-C
embolotherapy NN I-C
. . O

It PRP O
is VBZ O
generally RB O
believed VBN O
that IN O
the DT O
hypomineralization NN B-C
of IN I-C
affected VBN I-C
enamel NN I-C
is VBZ O
mainly RB O
due IN O
to IN O
in IN B-C
- HYPH I-C
situ NN I-C
toxic JJ I-C
effects NNS I-C
of IN I-C
the DT I-C
fluoride NN I-C
on IN I-C
the DT I-C
ameloblasts NNS I-C
in IN I-C
the DT I-C
enamel NN I-C
formation NN I-C
, , O
and CC O
not RB O
caused VBN O
by IN O
the DT O
general JJ B-C
effects NNS I-C
of IN I-C
fluoride NN I-C
on IN I-C
the DT I-C
calcium NN I-C
metabolism NN I-C
, , O
or CC O
by IN O
the DT O
poisoning NN B-C
effects NNS I-C
that WDT I-C
suppress VBP I-C
the DT I-C
fluoride NN I-C
metabolism NN I-C
, , O
. . O

When WRB O
a DT O
student NN B-C
is VBZ I-C
stressed VBN I-C
, , O
the DT O
stress NN B-C
hormone NN I-C
cortisol NN I-C
is VBZ O
released VBN O
, , O
which WDT O
causes VBZ O
some DT O
individuals NNS O
to TO O
excessively RB B-C
eat VB I-C
. . O

The DT O
number NN O
of IN O
flight NN B-C
delays NNS I-C
has VBZ O
increased VBN O
as IN O
staff NN B-C
has VBZ I-C
been VBN I-C
cut VBN I-C
back RB I-C
as IN O
a DT O
result NN O
of IN O
the DT O
financial JJ B-C
woes NNS I-C
following VBG O
the DT O
September NNP B-C
11 CD I-C
attacks NNS I-C
. . O

Complicated JJ O
silicosis NN O
: : O
Silicosis NN B-C
can MD O
become VB O
" `` O
complicated JJ O
" '' O
by IN O
the DT O
development NN O
of IN O
severe JJ B-C
scarring NN I-C
( -LRB- O
progressive JJ O
massive JJ O
fibrosis NN O
, , O
or CC O
also RB O
known VBN O
as IN O
conglomerate NN O
silicosis NN O
) -RRB- O
, , O
where WRB O
the DT O
small JJ O
nodules NNS O
gradually RB O
become VB O
confluent JJ O
, , O
reaching VBG O
a DT O
size NN O
of IN O
1 CD O
cm NNS O
or CC O
greater JJR O
. . O

Silicosis NN B-C
is VBZ O
seen VBN O
in IN O
horses NNS O
associated VBN O
with IN O
inhalation NN B-C
of IN I-C
dust NN I-C
from IN I-C
certain JJ I-C
cristobalite NN I-C
- HYPH I-C
containing VBG I-C
soils NNS I-C
in IN O
California NNP O
. . O

If IN O
such JJ O
events NNS B-C
accelerate VBP I-C
through IN I-C
mountain NN I-C
passes NNS I-C
, , O
dangerously RB B-C
accelerated VBN I-C
mountain NN I-C
- HYPH I-C
gap NN I-C
winds NNS I-C
can MD O
result VB O
, , O
such JJ O
as IN O
the DT O
Tehuantepecer NNP B-C
and CC I-C
Santa NNP I-C
Ana NNP I-C
winds NNS I-C
. . O

Breast NN B-C
pain NN I-C
is VBZ O
a DT O
medical JJ O
symptom NN O
that WDT O
is VBZ O
most RBS O
often RB O
associated VBN O
with IN O
a DT O
developing VBG B-C
disease NN I-C
or CC I-C
condition NN I-C
of IN I-C
the DT I-C
breast NN I-C
. . O

The DT O
tumor NN O
may MD O
compress VB O
neighboring VBG O
organs NNS O
, , O
disrupting VBG O
digestive JJ O
processes NNS O
and CC O
making VBG O
it PRP O
difficult JJ B-C
for IN I-C
the DT I-C
stomach NN I-C
to TO I-C
empty VB I-C
, , O
which WDT O
may MD O
cause VB O
nausea NN B-C
and CC O
a DT O
feeling NN B-C
of IN I-C
fullness NN I-C
. . O

Piblokto NNP B-C
may MD O
be VB O
linked VBN O
to IN O
repression NN B-C
of IN I-C
the DT I-C
personality NN I-C
of IN I-C
Inuit JJ I-C
women NNS I-C
. . O

The DT O
causative JJ O
agent NN O
of IN O
PHF NNP B-C
is VBZ O
Neorickettsia NNP B-C
risticii NN I-C
( -LRB- O
formerly RB O
Ehrlichia NNP O
risticii NNP O
) -RRB- O
, , O
an DT O
intracellular JJ O
rickettsial NN O
bacterium NN O
. . O

In IN O
the DT O
largest JJS O
study NN O
conducted VBN O
so RB O
far RB O
, , O
bevacizumab NN B-C
infusion NN I-C
was VBD O
associated VBN O
with IN O
a DT O
decrease NN B-C
in IN I-C
cardiac JJ I-C
output NN I-C
and CC O
reduced VBN B-C
duration NN I-C
and CC I-C
number NN I-C
of IN I-C
episodes NNS I-C
of IN I-C
epistaxis NN I-C
in IN O
treated VBN O
HHT NN O
patients NNS O
. . O

Through IN O
epidemiological JJ O
studies NNS O
in IN O
the DT O
USA NNP O
, , O
Henry NNP O
Trendley NNP O
Dean NNP O
helped VBD O
to TO O
identify VB O
a DT O
causal JJ O
link NN O
between IN O
high JJ B-C
concentrations NNS I-C
of IN I-C
fluoride NN I-C
in IN I-C
the DT I-C
drinking NN I-C
water NN I-C
and CC O
mottled JJ B-C
enamel NN I-C
. . O

Stereotypes NNS B-C
lead VBP O
people NNS O
to TO B-C
expect VB I-C
certain JJ I-C
actions NNS I-C
from IN I-C
members NNS I-C
of IN I-C
social JJ I-C
groups NNS I-C
. . O

Certain JJ O
genetic JJ B-C
conditions NNS I-C
can MD O
also RB O
lead VB O
to IN O
post NN B-C
- HYPH I-C
lingual JJ I-C
deafness NN I-C
. . O

The DT O
problem NN O
of IN O
radiation NN B-C
injuries NNS I-C
due IN O
to IN O
fluoroscopy NN B-C
has VBZ O
been VBN O
further RB O
investigated VBN O
in IN O
review NN O
articles NNS O
in IN O
2000 CD O
, , O
2001 CD O
, , O
2009 CD O
and CC O
2010 CD O
. . O

Therefore RB O
, , O
alcohol NN B-C
consumption NN I-C
may MD O
increase VB O
the DT O
production NN B-C
of IN I-C
uroporphyrinogen NN I-C
, , O
exacerbating VBG B-C
symptoms NNS I-C
in IN I-C
individuals NNS I-C
with IN I-C
porphyria JJ I-C
cutanea NN I-C
tarda NN I-C
. . O

Therefore RB O
, , O
the DT O
neuronal JJ B-C
degeneration NN I-C
in IN I-C
HSAN NNP I-C
IA NNP I-C
is VBZ O
likely JJ O
due IN O
to IN O
subtler JJR B-C
and CC I-C
rather RB I-C
long JJ I-C
- HYPH I-C
term NN I-C
effects NNS I-C
of IN I-C
the DT I-C
mutations NNS I-C
or CC O
perhaps RB O
accumulation NN B-C
of IN I-C
toxic JJ I-C
lipids NNS I-C
produced VBN I-C
by IN I-C
mutant JJ I-C
enzymes NNS I-C
. . O

The DT O
term NN O
" `` O
blister NN O
beetle NN O
dermatitis NN O
" '' O
is VBZ O
also RB O
occasionally RB O
and CC O
inappropriately RB O
used VBN O
as IN O
a DT O
synonym NN O
for IN O
Paederus NNP O
dermatitis NN O
, , O
a DT O
somewhat RB O
different JJ O
dermatitis NN B-C
caused VBN O
by IN O
contact NN B-C
with IN I-C
pederin NN I-C
, , O
an DT O
irritant NN O
in IN O
the DT O
hemolymph NN O
of IN O
a DT O
different JJ O
group NN O
of IN O
beetles NNS O
, , O
the DT O
rove NN O
beetles NNS O
. . O

Other JJ O
studies NNS O
have VBP O
shown VBN O
that IN O
when WRB O
people NNS B-C
are VBP I-C
stressed VBN I-C
, , O
they PRP O
have VBP O
cravings NNS B-C
for IN I-C
foods NNS I-C
that WDT O
are VBP O
high JJ O
in IN O
calories NNS O
such JJ O
as IN O
sweet JJ O
, , O
salty JJ O
, , O
and CC O
processed VBN O
foods NNS O
. . O

Radiation NN B-C
burns NNS I-C
are VBP O
caused VBN O
by IN O
exposure NN B-C
to IN I-C
high JJ I-C
levels NNS I-C
of IN I-C
radiation NN I-C
. . O

The DT O
2008 CD B-C
Sichuan NNP I-C
earthquake NN I-C
, , O
which WDT O
caused VBD O
approximately RB O
68,000 CD B-C
deaths NNS I-C
, , O
is VBZ O
another DT O
possible JJ O
example NN O
. . O

On IN O
April NNP O
21 CD O
, , O
2015 CD O
, , O
the DT O
Oklahoma NNP O
Geological NNP O
Survey NNP O
released VBD O
a DT O
statement NN O
reversing VBG O
its PRP$ O
stance NN O
on IN O
induced VBN O
earthquakes NNS O
in IN O
Oklahoma NNP O
: : O
" `` O
The DT O
OGS NNP O
considers VBZ O
it PRP O
very RB O
likely JJ O
that IN O
the DT O
majority NN O
of IN O
recent JJ O
earthquakes NNS B-C
, , O
particularly RB O
those DT O
in IN O
central JJ O
and CC O
north JJ O
- HYPH O
central JJ O
Oklahoma NNP O
, , O
are VBP O
triggered VBN O
by IN O
the DT O
injection NN B-C
of IN I-C
produced VBN I-C
water NN I-C
in IN I-C
disposal NN I-C
wells NNS I-C
. . O
" '' O

If IN O
a DT O
tape NN B-C
with IN I-C
sticky JJ I-C
- HYPH I-C
shed NN I-C
syndrome NN I-C
is VBZ I-C
played VBN I-C
, , O
the DT O
reels NNS O
will MD O
make VB O
screeching NN B-C
or CC I-C
squeaking NN I-C
sounds NNS I-C
, , O
and CC O
the DT O
tape NN O
will MD O
leave VB O
dusty JJ B-C
, , I-C
rusty JJ I-C
particles NNS I-C
on IN O
the DT O
guides NNS O
and CC O
heads NNS O
. . O

Large JJ B-C
hemangiomas NNS I-C
can MD O
leave VB O
visible JJ B-C
skin NN I-C
changes NNS I-C
secondary JJ O
to IN O
severe JJ O
stretching NN O
that IN O
results NNS O
in IN O
altered VBN B-C
surface NN I-C
texture NN I-C
. . O

Acute JJ B-C
generalized VBN I-C
exanthematous JJ I-C
pustulosis NN I-C
( -LRB- O
AGEP NNP O
) -RRB- O
( -LRB- O
also RB O
known VBN O
as IN O
pustular JJ O
drug NN O
eruption NN O
and CC O
toxic JJ O
pustuloderma NN O
) -RRB- O
is VBZ O
a DT O
rare JJ O
skin NN O
reaction NN O
that WDT O
in IN O
90 CD O
% NN O
of IN O
cases NNS O
is VBZ O
related JJ O
to IN O
medication NN B-C
administration NN I-C
. . O

Once IN O
infection NN O
occurs VBZ O
, , O
these DT O
ulcerations NNS B-C
can MD O
result VB O
in IN O
severe JJ B-C
complications NNS I-C
that WDT O
lead VBP O
to IN O
foot NN B-C
deformity NN I-C
, , O
such JJ O
as IN O
inflammation NN B-C
of IN I-C
the DT I-C
underlying JJ I-C
bones NNS I-C
, , O
spontaneous JJ B-C
bone NN I-C
fractures NNS I-C
, , O
and CC O
progressive JJ B-C
degeneration NN I-C
of IN I-C
weight NN I-C
- HYPH I-C
bearing NN I-C
joints NNS I-C
. . O

Prenatal NN O
testing NN O
for IN O
pregnancies NNS O
at IN O
increased VBN O
risk NN O
is VBZ O
possible JJ O
if IN O
the DT O
disease NN B-C
- HYPH O
causing VBG O
mutation NN B-C
has VBZ O
been VBN O
identified VBN O
in IN O
the DT O
family NN O
. . O

In IN O
the DT O
past JJ O
several JJ O
years NNS O
, , O
some DT O
energy NN B-C
technologies NNS I-C
that WDT O
inject VBP O
or CC O
extract VBP O
fluid NN O
from IN O
the DT O
Earth NNP O
, , O
such JJ O
as IN O
oil NN B-C
and CC I-C
gas NN I-C
extraction NN I-C
and CC O
geothermal JJ B-C
energy NN I-C
development NN I-C
, , O
have VBP O
been VBN O
found VBN O
or CC O
suspected VBN O
to TO O
cause VB O
seismic JJ B-C
events NNS I-C
. . O

The DT O
prevalence NN B-C
of IN I-C
silicosis NN I-C
led VBD O
some DT O
men NNS O
to TO O
grow VB O
what WP O
is VBZ O
called VBN O
a DT O
miner NN B-C
's POS I-C
mustache NN I-C
, , O
in IN O
an DT O
attempt NN O
to TO O
intercept VB O
as RB O
much JJ O
dust NN O
as IN O
possible JJ O
. . O

As IN O
a DT O
result NN O
of IN O
the DT O
continuous JJ B-C
raids NNS I-C
a DT O
huge JJ B-C
number NN I-C
of IN I-C
Russian JJ I-C
prisoners NNS I-C
were VBD I-C
taken VBN I-C
to IN I-C
Crimea NNP I-C
. . O

The DT O
cause NN O
of IN O
the DT O
crash NN B-C
was VBD O
determined VBN O
to TO O
be VB O
a DT O
stall NN B-C
caused VBN O
by IN O
the DT O
leading VBG B-C
edge NN I-C
slats NNS I-C
( -LRB- O
strictly RB O
speaking VBG O
, , O
Krueger NNP O
flaps NNS O
) -RRB- O
having VBG O
been VBN O
left VBN O
in IN O
the DT O
retracted VBN O
position NN O
. . O

Aberrant JJ B-C
mossy NN I-C
fibre NN I-C
sprouting NN I-C
may MD O
create VB O
excitatory JJ B-C
feedback NN I-C
circuits NNS I-C
that WDT O
lead VBP O
to IN O
temporal JJ B-C
lobe NN I-C
seizures NNS I-C
. . O

UROD NNP B-C
makes VBZ O
an DT O
enzyme NN O
called VBN O
uroporphyrinogen NN B-C
III CD I-C
decarboxylase NN I-C
, , O
which WDT O
is VBZ O
critical JJ O
to IN O
the DT O
chemical JJ B-C
process NN I-C
that WDT O
leads VBZ O
to IN O
heme NN B-C
production NN I-C
. . O

Patients NNS O
often RB O
report VBP O
quick JJ B-C
jerking NN I-C
movements NNS I-C
in IN O
the DT O
morning NN O
that IN O
results NNS O
in IN O
knocking VBG B-C
over RP I-C
objects NNS I-C
around IN O
them PRP O
. . O

The DT O
HEAT NN B-C
warhead NN I-C
has VBZ I-C
become VBN I-C
less RBR I-C
effective JJ I-C
against IN I-C
tanks NNS I-C
and CC O
other JJ O
armored JJ O
vehicles NNS O
due IN O
to IN O
the DT O
use NN O
of IN O
composite JJ B-C
armor NN I-C
, , O
explosive JJ B-C
- HYPH I-C
reactive JJ I-C
armor NN I-C
, , O
and CC O
active JJ B-C
protection NN I-C
systems NNS I-C
which WDT O
destroy VBP O
the DT O
HEAT NN O
warhead NN O
before IN O
it PRP O
hits VBZ O
the DT O
tank NN O
. . O

The DT O
blood NN B-C
hemoglobin NN I-C
levels NNS I-C
of IN I-C
these DT I-C
treated VBN I-C
patients NNS I-C
rose VBD I-C
as IN O
a DT O
result NN O
of IN O
reduced VBN B-C
hemorrhage NN I-C
and CC O
enhanced VBN B-C
blood NN I-C
vessel NN I-C
stabilization NN I-C
. . O

Knightley NNP B-C
was VBD I-C
reported VBN I-C
to TO I-C
have VB I-C
" `` I-C
cried VBN I-C
every DT I-C
single JJ I-C
day NN I-C
" '' I-C
due IN O
to IN O
finding VBG B-C
the DT I-C
wardrobe NN I-C
uncomfortable JJ I-C
. . O

When WRB O
drinking VBG B-C
alcohol NN I-C
on IN O
a DT O
regular JJ O
basis NN O
, , O
certain JJ O
vitamin NN B-C
and CC I-C
mineral JJ I-C
deficiencies NNS I-C
can MD O
follow VB O
. . O

Because IN O
a DT O
major JJ O
function NN O
of IN O
the DT O
temporal JJ O
lobe NN O
is VBZ O
short JJ O
- HYPH O
term NN O
memory NN O
, , O
a DT O
focal NN B-C
with IN I-C
impaired VBN I-C
awareness NN I-C
seizure NN I-C
, , O
and CC O
a DT O
focal NN B-C
to IN I-C
bilateral JJ I-C
seizure NN I-C
can MD O
cause VB O
amnesia NN B-C
for IN O
the DT O
period NN O
of IN O
the DT O
seizure NN O
, , O
meaning VBG O
that IN O
the DT O
seizure NN O
may MD O
not RB O
be VB O
remembered VBN O
. . O

Chronic NN B-C
inflammation NN I-C
and CC I-C
obstruction NN I-C
of IN I-C
the DT I-C
bile NN I-C
ducts NNS I-C
, , O
and CC O
the DT O
resulting VBG O
impaired VBN B-C
bile NN I-C
flow NN I-C
, , O
are VBP O
thought VBN O
to TO O
play VB O
a DT O
role NN O
in IN O
this DT O
progression NN O
. . O

The DT O
MAK NNP O
report NN O
found VBD O
the DT O
" `` O
immediate JJ O
cause NN O
" '' O
of IN O
the DT O
accident NN B-C
was VBD O
the DT O
failure NN B-C
of IN I-C
the DT I-C
crew NN I-C
to TO O
make VB O
a DT O
timely JJ O
decision NN O
to TO O
proceed VB O
to IN O
an DT O
alternate JJ O
airport NN O
despite IN O
being VBG O
warned VBN O
multiple JJ O
times NNS O
of IN O
the DT O
poor JJ O
weather NN O
conditions NNS O
at IN O
Smolensk NNP O
. . O

Lucas NNP B-C
decided VBD I-C
to TO I-C
make VB I-C
elaborate JJ I-C
costumes NNS I-C
because IN O
the DT O
film NN B-C
's POS I-C
society NN I-C
was VBD I-C
more RBR I-C
sophisticated JJ I-C
than IN O
the DT O
one NN O
depicted VBN O
in IN O
the DT O
original JJ O
trilogy NN O
. . O

Some DT O
breast NN B-C
pain NN I-C
is VBZ O
normal JJ O
and CC O
associated VBN O
with IN O
the DT O
changes NNS B-C
that WDT I-C
accompany VBP I-C
puberty NN I-C
, , O
pregnancy NN B-C
, , O
lactation NN B-C
and CC O
menopause NN B-C
. . O

The DT O
primary JJ O
cause NN O
of IN O
catastrophic JJ B-C
bush NN I-C
fires NNS I-C
during IN O
this DT O
period NN O
lies VBZ O
in IN O
a DT O
poor JJ B-C
understanding NN I-C
of IN I-C
local JJ I-C
fire NN I-C
regimes NNS I-C
and CC O
in IN O
inappropriate JJ B-C
landscape NN I-C
management NN I-C
by IN I-C
settlers NNS I-C
. . O

Though IN O
twitching NN B-C
is VBZ O
sometimes RB O
a DT O
symptom NN O
of IN O
serious JJ B-C
diseases NNS I-C
such JJ O
as IN O
spinal JJ B-C
injury NN I-C
, , O
muscular JJ B-C
dystrophy NN I-C
, , O
Lyme NNP B-C
disease NN I-C
, , O
Creutzfeldt NNP B-C
– , I-C
Jakob NNP I-C
disease NN I-C
( -LRB- O
CJD NN O
) -RRB- O
, , O
neurofibromatosis JJ B-C
or CC O
amyotrophic JJ B-C
lateral JJ I-C
sclerosis NN I-C
( -LRB- O
ALS NN O
) -RRB- O
, , O
causes VBZ O
like IN O
over IN B-C
- HYPH I-C
exertion NN I-C
are VBP O
more RBR O
common JJ O
. . O

The DT O
diagnosis NN O
of IN O
HHT NNP O
remained VBD O
a DT O
clinical JJ O
one NN O
until IN O
the DT O
genetic JJ B-C
defects NNS I-C
that WDT O
cause VBP O
HHT NNP B-C
were VBD O
identified VBN O
by IN O
a DT O
research NN O
group NN O
at IN O
Duke NNP O
University NNP O
Medical NNP O
Center NNP O
, , O
in IN O
1994 CD O
and CC O
1996 CD O
respectively RB O
. . O

Since IN O
the DT O
complications NNS O
mimic VBP O
foot NN B-C
ulcers NNS I-C
caused VBN O
by IN O
diabetic JJ B-C
neuropathy NN I-C
, , O
the DT O
treatment NN O
for IN O
foot NN O
ulcers NNS O
and CC O
infections NNS O
can MD O
follow VB O
the DT O
guidelines NNS O
given VBN O
for IN O
diabetic JJ O
foot NN O
care NN O
which WDT O
starts VBZ O
with IN O
early JJ O
and CC O
accurate JJ O
counseling NN O
of IN O
patients NNS O
about IN O
risk NN O
factors NNS O
for IN O
developing VBG O
foot NN O
ulcerations NNS O
. . O

Dynapenia NNP B-C
can MD O
contribute VB O
to IN O
increased VBN B-C
risk NN I-C
of IN I-C
falling NN I-C
as RB O
well RB O
as IN O
feeling VBG B-C
weak JJ I-C
and CC I-C
/ , I-C
or CC I-C
fatigued JJ I-C
. . O

The DT O
risk NN O
of IN O
pancreatic JJ B-C
cancer NN I-C
in IN O
individuals NNS O
with IN O
familial JJ B-C
pancreatitis NN I-C
is VBZ O
particularly RB O
high JJ O
. . O

Hypothesized VBD O
psychological JJ O
reasons NNS O
for IN O
the DT O
increase NN B-C
in IN I-C
kodokushi NN I-C
include VBP O
social JJ B-C
apathy NN I-C
and CC O
life NN B-C
stress NN I-C
. . O

The DT O
report NN O
noted VBD O
that IN O
hydraulic NN B-C
fracturing NN I-C
has VBZ O
a DT O
low JJ O
risk NN O
for IN O
inducing VBG O
earthquakes NNS B-C
that WDT O
can MD O
be VB O
felt VBN O
by IN O
people NNS O
, , O
but CC O
underground JJ B-C
injection NN I-C
of IN I-C
wastewater NN I-C
produced VBN O
by IN O
hydraulic JJ B-C
fracturing NN I-C
and CC O
other JJ O
energy NN B-C
technologies NNS I-C
has VBZ O
a DT O
higher JJR O
risk NN O
of IN O
causing VBG O
such JJ O
earthquakes NNS B-C
. . O

MG NNP O
is VBZ O
characteristically RB O
variable JJ O
in RB O
course RB O
, , O
with IN O
the DT O
frequency NN B-C
of IN I-C
diplopia NN I-C
and CC I-C
ptosis NN I-C
affected VBN O
by IN O
environmental JJ O
, , O
emotional JJ O
and CC O
physical JJ O
factors NNS O
such JJ O
as IN O
bright JJ B-C
sunlight NN I-C
, , O
stress NN B-C
, , O
viral NN B-C
illness NN I-C
, , O
menstruation NN B-C
, , O
pregnancy NN B-C
, , O
etc FW O
. . O

While IN O
a DT O
deficiency NN B-C
in IN I-C
this DT I-C
enzyme NN I-C
is VBZ O
the DT O
direct JJ O
cause NN O
leading VBG O
to IN O
this DT O
disorder NN B-C
, , O
there EX O
are VBP O
a DT O
number NN O
of IN O
both CC O
genetic JJ B-C
and CC I-C
environmental JJ I-C
risk NN I-C
factors NNS I-C
that WDT O
are VBP O
associated VBN O
with IN O
PCT NNP B-C
. . O

HLA NN B-C
serotypes NNS I-C
associated VBN O
with IN O
AGEP NNP B-C
and CC O
specific JJ O
drugs NNS O
have VBP O
not RB O
been VBN O
identified VBN O
. . O

Temporary JJ B-C
hardness NN I-C
is VBZ O
a DT O
type NN O
of IN O
water NN O
hardness NN O
caused VBN O
by IN O
the DT O
presence NN B-C
of IN I-C
dissolved VBN I-C
bicarbonate NN I-C
minerals NNS I-C
( -LRB- O
calcium NN O
bicarbonate NN O
and CC O
magnesium NN O
bicarbonate NN O
) -RRB- O
. . O

Examples NNS O
of IN O
symptoms NNS O
of IN O
childbirth NN B-C
- HYPH I-C
related JJ I-C
posttraumatic JJ I-C
stress NN I-C
disorder NN I-C
include VBP O
: : O
intrusive JJ B-C
symptoms NNS I-C
such JJ O
as IN O
flashbacks NNS B-C
and CC O
nightmares NNS B-C
, , O
as RB O
well RB O
as IN O
symptoms NNS B-C
of IN I-C
avoidance NN I-C
( -LRB- O
including VBG O
amnesia NN O
for IN O
the DT O
whole JJ O
or CC O
parts NNS O
of IN O
the DT O
event NN O
) -RRB- O
, , O
problems NNS B-C
in IN I-C
developing VBG I-C
a DT I-C
mother NN I-C
- HYPH I-C
child NN I-C
attachment NN I-C
, , O
not RB B-C
having VBG I-C
sexual JJ I-C
intercourse NN I-C
in IN O
order NN O
to TO O
prevent VB O
another DT O
pregnancy NN O
, , O
and CC O
avoidance NN B-C
of IN I-C
birth NN I-C
and CC O
pregnancy NN B-C
related JJ I-C
issues NNS I-C
. . O

Loneliness NN B-C
and CC O
depression NN B-C
can MD O
arise VB O
as IN O
a DT O
result NN O
of IN O
isolation NN B-C
( -LRB- O
from IN O
the DT O
inability NN O
to TO O
communicate VB O
with IN O
friends NNS O
and CC O
loved VBN O
ones NNS O
) -RRB- O
and CC O
difficulty NN B-C
in IN I-C
accepting VBG I-C
their PRP$ I-C
disability NN I-C
. . O

Aside RB O
from IN O
asymmetric JJ B-C
ptosis NN I-C
( -LRB- O
which WDT O
becomes VBZ O
worse JJR O
with IN O
fatigue NN O
, , O
sustained VBN O
upgaze NN O
, , O
and CC O
at IN O
the DT O
end NN O
of IN O
the DT O
day NN O
) -RRB- O
and CC O
variable JJ B-C
limitation NN I-C
of IN I-C
extraocular JJ I-C
muscles NNS I-C
/ , O
diplopia NN B-C
, , O
other JJ O
clinical JJ O
signs NNS O
of IN O
ocular JJ B-C
MG NN I-C
include VBP O
gaze NN B-C
- HYPH I-C
evoked VBN I-C
nystagmus NN I-C
( -LRB- O
rapid JJ O
, , O
involuntary JJ O
, , O
oscillatory JJ O
motion NN O
of IN O
the DT O
eyeball NN O
) -RRB- O
and CC O
Cogan NNP B-C
’s POS I-C
lid NN I-C
twitch NN I-C
( -LRB- O
upper JJ O
lid NN O
twitch NN O
present NN O
when WRB O
patient NN O
looks VBZ O
straight RB O
ahead RB O
after IN O
looking VBG O
down RB O
for IN O
10 CD O
– SYM O
15 CD O
seconds NNS O
) -RRB- O
. . O

The DT O
nervous JJ B-C
system NN I-C
’s POS I-C
lowered VBN I-C
ability NN I-C
to TO I-C
stimulate VB I-C
a DT I-C
full JJ I-C
muscle NN I-C
contraction NN I-C
subsequently RB O
leads VBZ O
to IN O
loss NN B-C
of IN I-C
muscle NN I-C
strength NN I-C
and CC I-C
power NN I-C
. . O

Lesions NNS B-C
adjacent VBP I-C
to IN I-C
bone NN I-C
may MD O
cause VB O
erosion NN B-C
of IN I-C
the DT I-C
bone NN I-C
. . O

The DT O
great JJ O
majority NN O
of IN O
breast NN B-C
cancer NN I-C
cases NNS O
do VBP O
not RB O
present VB O
with IN O
symptoms NNS O
of IN O
pain NN B-C
, , O
though IN O
breast NN B-C
pain NN I-C
in IN I-C
older JJR I-C
women NNS I-C
is VBZ O
more RBR O
likely JJ O
to TO O
be VB O
associated VBN O
with IN O
cancer NN B-C
. . O

Brief JJ B-C
or CC I-C
casual JJ I-C
exposure NN I-C
to IN I-C
low JJ I-C
levels NNS I-C
of IN I-C
crystalline JJ I-C
silica NN I-C
dust NN O
are VBP O
said VBN O
to TO O
not RB O
produce VB O
clinically RB B-C
significant JJ I-C
lung NN I-C
disease NN I-C
. . O

Some DT O
tapes NNS B-C
may MD I-C
deteriorate VB I-C
because IN O
of IN O
a DT O
breakdown NN B-C
in IN I-C
the DT I-C
binder NN I-C
( -LRB- O
the DT O
glue NN O
) -RRB- O
that WDT O
holds VBZ O
the DT O
oxide NN O
particles NNS O
on IN O
the DT O
tape NN O
, , O
or CC O
the DT O
back NN O
coating NN O
on IN O
the DT O
reverse JJ O
side NN O
, , O
if IN O
the DT O
tape NN O
was VBD O
from IN O
any DT O
of IN O
the DT O
tape NN O
manufacturers NNS O
who WP O
had VBD O
inadvertently RB O
used VBN O
an DT O
unstable JJ O
binder NN O
formulation NN O
. . O

The DT O
damaging VBG O
effects NNS O
of IN O
porphyrins NNS O
interacting VBG O
with IN O
iron NN O
, , O
absorbing VBG O
photons NNS O
to TO O
then RB O
emit VB O
reactive JJ O
oxygen NN O
species NNS O
is VBZ O
the DT O
mechanism NN O
of IN O
action NN O
that WDT O
results VBZ O
in IN O
the DT O
itchy JJ B-C
, , I-C
painful JJ I-C
blisters NNS I-C
that WDT O
are VBP O
common JJ O
with IN O
PCT NN B-C
. . O

It PRP O
may MD O
lead VB O
to IN O
nosebleeds NNS O
, , O
acute JJ O
and CC O
chronic JJ O
digestive JJ O
tract NN O
bleeding NN O
, , O
and CC O
various JJ B-C
problems NNS I-C
due IN O
to IN O
the DT O
involvement NN B-C
of IN I-C
other JJ I-C
organs NNS I-C
. . O

Typically RB O
, , O
Dupuytren NNP B-C
's POS I-C
contracture NN I-C
first RB O
presents VBZ O
as IN O
a DT O
thickening NN B-C
or CC I-C
nodule NN I-C
in IN I-C
the DT I-C
palm NN I-C
, , O
which WDT O
initially RB O
can MD O
be VB O
with IN O
or CC O
without IN O
pain NN B-C
. . O

Natural JJ O
gas NN O
remained VBD O
at IN O
the DT O
same JJ O
price NN O
during IN O
the DT O
inflationary JJ B-C
process NN I-C
caused VBN O
by IN O
the DT O
devaluation NN B-C
of IN I-C
the DT I-C
Argentine JJ I-C
peso NN I-C
, , O
while IN O
the DT O
prices NNS O
of IN O
gasoline NN O
and CC O
diesel NN O
were VBD O
adjusted VBN O
upwards RB O
, , O
which WDT O
increased VBD O
the DT O
demand NN O
for IN O
gas NN O
as IN O
a DT O
cheap JJ O
alternative JJ O
fuel NN O
and CC O
at IN O
the DT O
same JJ O
time NN O
discouraged VBD O
its PRP$ O
production NN O
. . O

People NNS O
who WP O
have VBP O
these DT O
mutations NNS B-C
are VBP O
also RB O
at IN O
an DT O
increased VBN O
risk NN O
of IN O
developing VBG O
porphyria JJ B-C
cutanea NN I-C
tarda NN I-C
. . O

Vascular JJ B-C
malformations NNS I-C
in IN I-C
the DT I-C
lungs NNS I-C
may MD O
cause VB O
a DT O
number NN B-C
of IN I-C
problems NNS I-C
. . O

However RB O
, , O
breast NN B-C
pain NN I-C
is VBZ O
not RB O
a DT O
common JJ O
symptom NN O
of IN O
cancer NN B-C
. . O

Several JJ O
severe JJ B-C
injuries NNS I-C
or CC O
diseases NNS B-C
can MD O
cause VB O
IA NNP B-C
in IN O
a DT O
wide JJ O
range NN O
of IN O
patients NNS O
. . O

Due IN O
to IN O
habitat NN B-C
loss NN I-C
and CC O
colonisation NN B-C
of IN I-C
their PRP$ I-C
natural JJ I-C
habitat NN I-C
by IN I-C
other JJ I-C
exotic JJ I-C
spiders NNS I-C
, , O
the DT O
katipo NN B-C
is VBZ I-C
threatened VBN I-C
with IN I-C
extinction NN I-C
. . O

The DT O
most RBS O
severe JJ O
risks NNS O
of IN O
insulin NN B-C
coma NN I-C
therapy NN O
were VBD O
death NN B-C
and CC O
brain NN B-C
damage NN I-C
, , O
resulting VBG O
from IN O
irreversible JJ O
or CC O
prolonged VBN O
coma NN B-C
respectively RB O
. . O

Patients NNS O
with IN O
chronic JJ B-C
liver NN I-C
disease NN I-C
, , O
whether IN O
in IN O
the DT O
form NN O
of IN O
viral NN B-C
hepatitis NN I-C
( -LRB- O
e.g. FW O
hepatitis NN O
B NN O
or CC O
hepatitis NN O
C NN O
) -RRB- O
, , O
alcoholic JJ B-C
liver NN I-C
disease NN I-C
, , O
or CC O
cirrhosis NN B-C
of IN I-C
the DT I-C
liver NN I-C
due IN O
to IN O
other JJ O
causes NNS O
, , O
are VBP O
at IN O
significantly RB O
increased VBN O
risk NN O
of IN O
cholangiocarcinoma NN B-C
. . O

Finally RB O
, , O
hemorrhage NN B-C
from IN I-C
an DT I-C
AVM NN I-C
may MD O
lead VB O
to IN O
intracerebral JJ B-C
hemorrhage NN I-C
( -LRB- O
bleeding VBG O
into IN O
the DT O
brain NN O
) -RRB- O
, , O
which WDT O
causes VBZ O
any DT O
of IN O
the DT O
symptoms NNS B-C
of IN I-C
stroke NN I-C
such JJ O
as IN O
weakness NN B-C
in IN I-C
part NN I-C
of IN I-C
the DT I-C
body NN I-C
or CC O
difficulty NN B-C
speaking VBG I-C
. . O

It PRP O
is VBZ O
extremely RB O
common JJ O
to TO O
find VB O
incidental JJ B-C
nodules NNS I-C
on IN O
liver NN O
scans NNS O
, , O
most RBS O
commonly RB O
due IN O
to IN O
focal JJ B-C
nodular JJ I-C
hyperplasia NN I-C
( -LRB- O
FNH NN O
) -RRB- O
, , O
as IN O
these DT O
are VBP O
a DT O
hundredfold NN O
times NNS O
more RBR O
common JJ O
in IN O
HHT NN O
compared VBN O
to IN O
the DT O
general JJ O
population NN O
. . O

In IN O
TLE NN B-C
, , O
granule NN B-C
cells NNS I-C
are VBP I-C
lost VBN I-C
, , O
the DT O
structure NN O
is VBZ O
no RB O
longer RBR O
closely RB O
packed VBN O
and CC O
there EX O
are VBP O
changes NNS B-C
in IN I-C
the DT I-C
orientation NN I-C
of IN I-C
dendrites NNS I-C
. . O

Nongenetic NNP O
factors NNS O
such JJ O
as IN O
alcohol NN B-C
abuse NN I-C
, , O
excess JJ B-C
iron NN I-C
, , O
and CC O
others NNS O
listed VBN O
above RB O
can MD O
increase VB O
the DT O
demand NN B-C
for IN I-C
heme NN I-C
and CC O
the DT O
enzymes NNS O
required VBN O
to TO O
make VB O
heme NN O
. . O

Pancreatic JJ B-C
cancer NN I-C
is VBZ O
the DT O
fifth JJ O
most RBS O
common JJ O
cause NN O
of IN O
death NN B-C
from IN I-C
cancer NN I-C
in IN O
the DT O
United NNP O
Kingdom NNP O
, , O
and CC O
the DT O
fourth JJ O
most RBS O
common JJ O
in IN O
the DT O
United NNP O
States NNP O
. . O

The DT O
collapse NN B-C
resulted VBD O
in IN O
14 CD B-C
deaths NNS I-C
and CC O
25 CD B-C
injuries NNS I-C
to IN O
the DT O
construction NN O
workers NNS O
working VBG O
on IN O
the DT O
building NN O
. . O

The DT O
intensity NN B-C
of IN I-C
these DT I-C
fires NNS I-C
is VBZ O
due IN O
in IN O
part NN O
to IN O
natural JJ B-C
fuels NNS I-C
, , O
such JJ O
as IN O
sclerophyll JJ B-C
forests NNS I-C
in IN O
the DT O
region NN O
. . O

Symptoms NNS O
of IN O
increasing VBG B-C
stress NN I-C
can MD O
be VB O
sweating VBG B-C
, , O
trembling VBG B-C
, , O
being VBG B-C
irritated VBN I-C
, , O
and CC O
sleep NN B-C
disturbances NNS I-C
. . O

Evidence NN O
to TO O
support VB O
its PRP$ O
use NN O
as IN O
of IN O
2017 CD O
, , O
however RB O
, , O
is VBZ O
poor JJ O
; , O
efforts NNS B-C
to TO I-C
gather VB I-C
evidence NN I-C
are VBP I-C
complicated JJ I-C
due IN O
to IN O
a DT O
poor JJ B-C
understanding NN I-C
of IN I-C
the DT I-C
how WRB I-C
the DT I-C
condition NN I-C
develops VBZ I-C
over IN I-C
time NN I-C
. . O

The DT O
undigested JJ B-C
fat NN I-C
leads VBZ O
to IN O
foul NN B-C
- HYPH I-C
smelling NN I-C
, , I-C
fatty NN I-C
feces NNS I-C
that WDT O
are VBP O
difficult JJ O
to TO O
flush VB O
away RP O
. . O

The DT O
BAD NNP B-C
is VBZ O
mainly RB O
caused VBN O
by IN O
the DT O
high JJ B-C
temperature NN I-C
and CC O
velocity NN B-C
armor NN I-C
and CC O
slug NN B-C
fragments NNS I-C
being VBG I-C
injected VBN I-C
into IN I-C
the DT I-C
interior JJ I-C
space NN I-C
and CC O
also RB O
overpressure NN B-C
( -LRB- O
blast NN O
) -RRB- O
caused VBN O
by IN O
the DT O
impact NN B-C
. . O

Generally RB O
, , O
the DT O
cords NNS O
or CC O
contractures NNS O
are VBP O
painless JJ O
, , O
but CC O
, , O
rarely RB O
, , O
tenosynovitis NN B-C
can MD O
occur VB O
and CC O
produce VB O
pain NN B-C
. . O

Potential JJ O
complications NNS O
of IN O
surgical JJ B-C
correction NN I-C
of IN I-C
a DT I-C
rectocele NN I-C
include VBP O
bleeding NN B-C
, , O
infection NN B-C
, , O
dyspareunia NN B-C
( -LRB- O
pain NN O
during IN O
intercourse NN O
) -RRB- O
, , O
as RB O
well RB O
as IN O
recurrence NN B-C
or CC I-C
even RB I-C
worsening NN I-C
of IN I-C
the DT I-C
rectocele JJ I-C
symptoms NNS I-C
. . O

Its PRP$ O
cause NN O
is VBZ O
unknown JJ O
, , O
but CC O
coagulation NN B-C
defects NNS I-C
caused VBN O
by IN O
amyloid NN B-C
are VBP O
thought VBN O
to TO O
contribute VB O
. . O

During IN O
the DT O
war NN O
there EX O
was VBD O
an DT O
increase NN B-C
in IN I-C
infectious JJ I-C
diseases NNS I-C
caused VBN O
by IN O
the DT O
general JJ B-C
malnutrition NN I-C
among IN O
the DT O
Polish JJ O
population NN O
. . O

In IN O
approximately RB O
15 CD O
% NN O
to IN O
30 CD O
% NN O
of IN O
people NNS O
, , O
urushiol NNP B-C
does VBZ O
not RB O
trigger VB O
an DT O
immune JJ B-C
system NN I-C
response NN I-C
, , O
while IN O
at RB O
least RBS O
25 CD O
% NN O
of IN O
people NNS O
have VBP O
a DT O
very RB B-C
strong JJ I-C
immune JJ I-C
response NN I-C
resulting VBG O
in IN O
severe JJ B-C
symptoms NNS I-C
. . O

The DT O
risk NN O
was VBD O
best RBS O
seen VBN O
in IN O
cases NNS O
with IN O
underlying JJ O
silicosis NN B-C
, , O
with IN O
relative JJ O
risks NNS O
for IN O
lung NN B-C
cancer NN I-C
of IN O
2–4 CD O
. . O

The DT O
signs NNS O
and CC O
symptoms NNS O
of IN O
HMG NN B-C
- HYPH I-C
CoA NN I-C
lyase NN I-C
deficiency NN I-C
include VBP O
vomiting NN B-C
, , O
dehydration NN B-C
, , O
lethargy NN B-C
, , O
convulsions NNS B-C
, , O
and CC O
coma NN B-C
. . O

High JJ B-C
- HYPH I-C
explosive JJ I-C
anti-tank JJ I-C
rounds NNS I-C
caused VBD O
a DT O
revolution NN B-C
in IN I-C
anti-tank JJ I-C
warfare NN I-C
when WRB O
they PRP O
were VBD O
first RB O
introduced VBN O
in IN O
the DT O
later JJ O
stages NNS O
of IN O
World NNP O
War NNP O
II CD O
. . O

It PRP O
is VBZ O
usually RB O
found VBN O
in IN O
association NN O
with IN O
fatty NN O
liver NN O
, , O
an DT O
early JJ O
stage NN O
of IN O
alcoholic JJ O
liver NN O
disease NN O
, , O
and CC O
may MD O
contribute VB O
to IN O
the DT O
progression NN B-C
of IN I-C
fibrosis NN I-C
, , O
leading VBG O
to IN O
cirrhosis NN B-C
. . O

The DT O
New NNP O
Zealand NNP O
AA NN O
has VBZ O
submitted VBN O
to IN O
the DT O
Government NN O
that IN O
New NNP B-C
Zealands NNP I-C
poor JJ I-C
road NN I-C
safety NN I-C
performance NN I-C
is VBZ O
due IN O
to IN O
misplaced VBN B-C
overemphasis NN I-C
on IN I-C
speed NN I-C
and CC I-C
alcohol NN I-C
when WRB O
emergent JJ O
risks NNS O
are VBP O
now RB O
more RBR O
significant JJ O
( -LRB- O
Saving VBG O
Ourselves PRP O
publication NN O
, , O
submissions NNS O
to IN O
2020 CD O
plan NN O
consultation NN O
) -RRB- O
. . O

Additionally RB O
, , O
exposure NN B-C
to IN I-C
chlorinated VBN I-C
cyclic JJ I-C
hydrocarbons NNS I-C
can MD O
lead VB O
to IN O
a DT O
deficiency NN B-C
in IN I-C
the DT I-C
activity NN I-C
of IN I-C
uroporphyrinogen NN I-C
decarboxylase NN I-C
, , O
causing VBG O
the DT O
buildup NN O
of IN O
excess JJ B-C
uroporphyrinogen NN I-C
. . O

Unlike IN O
other JJ O
causes NNS O
of IN O
acute JJ B-C
colitis NN I-C
in IN I-C
horses NNS I-C
, , O
such JJ O
as IN O
Salmonella NNP B-C
and CC O
Clostridium NNP B-C
, , O
PHF NNP B-C
is VBZ O
not RB O
spread VBN O
directly RB O
from IN O
horse NN O
to IN O
horse NN O
. . O

Ulcerations NNS B-C
are VBP O
prone JJ O
to IN O
secondary JJ B-C
bacterial NN I-C
infections NNS I-C
which WDT O
can MD O
present VB O
with IN O
yellow JJ B-C
crusting NN I-C
, , O
drainage NN B-C
, , O
pain NN B-C
or CC O
odor NN B-C
. . O

Cold JJ B-C
air NN I-C
damming NN I-C
causes VBZ O
the DT O
cold JJ B-C
air NN I-C
to TO I-C
bank VB I-C
up RP I-C
along IN I-C
the DT I-C
eastern JJ I-C
Cascade NNP I-C
slopes NNS I-C
, , O
especially RB O
into IN O
the DT O
lower JJR O
passes NNS O
, , O
such JJ O
as IN O
Snoqualmie NNP O
Pass NNP O
and CC O
Stevens NNP O
Pass NNP O
. . O

" `` O
Beta NNP B-C
burns NNS I-C
" '' O
are VBP O
shallow JJ O
surface NN O
burns NNS O
, , O
usually RB O
of IN O
skin NN O
and CC O
less RBR O
often RB O
of IN O
lungs NNS O
or CC O
gastrointestinal JJ O
tract NN O
, , O
caused VBN O
by IN O
beta NN B-C
particles NNS I-C
, , O
typically RB O
from IN O
hot JJ O
particles NNS O
or CC O
dissolved VBN O
radionuclides NNS O
that WDT O
came VBD O
to IN O
direct JJ O
contact NN O
with IN O
or CC O
close JJ O
proximity NN O
to IN O
the DT O
body NN O
. . O

HHT NNP B-C
is VBZ O
the DT O
most RBS O
common JJ O
cause NN O
of IN O
lung NN B-C
AVMs NNS I-C
: : O
out IN O
of IN O
all DT O
people NNS O
found VBN O
to TO O
have VB O
lung NN B-C
AVMs NNS I-C
, , O
70 CD O
– SYM O
80 CD O
% NN O
are VBP O
due IN O
to IN O
HHT NN B-C
. . O

Individually RB O
, , O
structural JJ B-C
violence NN I-C
creates VBZ O
barriers NNS B-C
that WDT O
prevent VBP O
one CD O
from IN O
properly RB O
receiving VBG O
and CC O
utilizing VBG O
health NN O
care NN O
systems NNS O
. . O

If IN O
a DT O
hemangioma NN B-C
develops VBZ I-C
in IN I-C
the DT I-C
larynx NN I-C
, , O
breathing NN B-C
can MD I-C
be VB I-C
compromised VBN I-C
. . O

Minor JJ B-C
injuries NNS I-C
in IN I-C
the DT I-C
painless JJ I-C
area NN I-C
can MD O
result VB O
in IN O
slow JJ B-C
- HYPH I-C
healing NN I-C
wounds NNS I-C
which WDT O
, , O
if IN O
not RB O
immediately RB O
recognized VBN O
, , O
can MD O
develop VB O
into IN O
chronic JJ B-C
ulcerations NNS I-C
. . O

Herbal NN B-C
medications NNS I-C
, , O
spider NN B-C
bites NNS I-C
, , O
iopamidol NN B-C
( -LRB- O
used VBN O
for IN O
radiocontrast NN O
) -RRB- O
, , O
lacquers NNS B-C
, , O
mercury NN B-C
, , O
psoralen NN B-C
( -LRB- O
combined VBN O
with IN O
ultraviolet NN O
A NN O
to TO O
treat VB O
psoriasis NN O
) -RRB- O
, , O
and CC O
xenobiotics NNS B-C
have VBP O
been VBN O
associated VBN O
with IN O
the DT O
development NN O
of IN O
AGEP NNP B-C
in IN O
case NN O
reports NNS O
. . O

The DT O
fat JJ B-C
graft NN I-C
results NNS O
in IN O
supple JJ B-C
skin NN I-C
. . O

When WRB O
cold JJ B-C
air NN I-C
damming NN I-C
occurs VBZ I-C
, , O
it PRP O
allows VBZ O
for IN O
cold JJ B-C
air NN I-C
to TO I-C
surge VB I-C
toward IN I-C
the DT I-C
equator NN I-C
in IN O
the DT O
affected VBN O
area NN O
. . O

Systemic NN B-C
- HYPH I-C
onset JJ I-C
juvenile JJ I-C
idiopathic JJ I-C
arthritis NN I-C
( -LRB- O
also RB O
known VBN O
as IN O
systemic JJ O
juvenile JJ O
idiopathic JJ O
arthritis NN O
( -LRB- O
s AFX O
JIA NN O
) -RRB- O
or CC O
the DT O
juvenile JJ O
onset NN O
form NN O
of IN O
Still RB O
's POS O
disease NN O
) -RRB- O
is VBZ O
a DT O
type NN O
of IN O
juvenile JJ O
idiopathic JJ O
arthritis NN O
( -LRB- O
JIA NN O
) -RRB- O
with IN O
extra-articular JJ O
manifestations NNS O
like IN O
fever NN B-C
and CC O
rash NN B-C
apart RB O
from IN O
arthritis NN B-C
. . O

That IN O
White NNP B-C
was VBD I-C
not RB I-C
convicted VBN I-C
of IN I-C
first JJ I-C
- HYPH I-C
degree NN I-C
murder NN I-C
( -LRB- O
of IN O
which WDT O
he PRP O
was VBD O
originally RB O
charged VBN O
) -RRB- O
had VBD O
so RB O
outraged VBN B-C
the DT I-C
city's NNS I-C
gay JJ I-C
community NN I-C
that IN O
it PRP O
set VBD O
off RP O
the DT O
most RBS O
violent JJ B-C
reaction NN I-C
by IN I-C
gay JJ I-C
Americans NNPS I-C
since IN O
the DT O
1969 CD O
Stonewall NNP O
Riots NNPS O
in IN O
New NNP O
York NNP O
City NNP O
( -LRB- O
which WDT O
is VBZ O
credited VBN O
as IN O
the DT O
beginning NN O
of IN O
the DT O
modern JJ O
gay JJ O
rights NNS O
movement NN O
in IN O
the DT O
United NNP O
States NNP O
) -RRB- O
. . O

Lack NN B-C
of IN I-C
support NN I-C
, , O
or CC O
insufficient JJ B-C
coping NN I-C
strategies NNS I-C
to TO I-C
deal VB I-C
with IN I-C
the DT I-C
pain NN I-C
are VBP O
examples NNS O
of IN O
situations NNS O
that WDT O
can MD O
cause VB O
psychological JJ B-C
trauma NN I-C
. . O

Coupled VBN O
with IN O
a DT O
continuing VBG O
series NN O
of IN O
political JJ B-C
upheavals NNS I-C
that WDT O
have VBP O
led VBN O
to IN O
an DT O
unending JJ O
social JJ B-C
instability NN I-C
, , O
Haiti NNP O
's POS O
inefficient JJ O
healthcare NN O
framework NN O
has VBZ O
crippled VBN O
the DT O
nation NN O
's POS O
ability NN O
to TO O
provide VB O
good JJ O
healthcare NN O
access NN O
to IN O
its PRP$ O
people NNS O
. . O

While IN O
piblokto NN O
can MD O
often RB O
be VB O
confused VBN O
with IN O
other JJ O
conditions NNS O
, , O
( -LRB- O
including VBG O
epilepsy NN O
) -RRB- O
in IN O
which WDT O
failure NN B-C
to TO I-C
intervene VB I-C
can MD O
lead VB O
to IN O
the DT O
victim NN B-C
coming VBG I-C
to TO I-C
harm VB I-C
, , O
most JJS O
cases NNS O
tend VBP O
to TO O
be VB O
more RBR O
typical JJ O
. . O

Prominent JJ O
signs NNS O
and CC O
symptoms NNS O
of IN O
cholangiocarcinoma NN B-C
include VBP O
abnormal JJ B-C
liver NN I-C
function NN I-C
tests NNS I-C
, , O
abdominal JJ B-C
pain NN I-C
, , O
jaundice NN B-C
, , O
and CC O
weight NN B-C
loss NN I-C
. . O

The DT O
most RBS O
common JJ O
cause NN O
of IN O
CVI NN B-C
is VBZ O
superficial JJ B-C
venous JJ I-C
reflux NN I-C
which WDT O
is VBZ O
a DT O
treatable JJ O
condition NN O
. . O

Numerous JJ B-C
isolations NNS I-C
of IN I-C
both CC I-C
SPONV NNP I-C
strain NN I-C
types NNS I-C
along IN I-C
with IN I-C
evidence NN I-C
of IN I-C
antibodies NNS I-C
to IN I-C
the DT I-C
two CD I-C
strains NNS I-C
were VBD I-C
not RB I-C
detected VBN I-C
in IN I-C
any DT I-C
rodent NN I-C
or CC I-C
birds NNS I-C
collected VBN I-C
in IN I-C
South NNP I-C
Africa NNP I-C
in IN I-C
1958 CD I-C
, , O
leading VBG O
to IN O
the DT O
speculation NN B-C
that IN I-C
these DT I-C
species NNS I-C
were VBD I-C
unlikely JJ I-C
amplification NN I-C
and CC I-C
transmission NN I-C
of IN I-C
the DT I-C
virus NN I-C
. . O

Alcoholic JJ B-C
hepatitis NN I-C
can MD O
vary VB O
from IN O
mild JJ O
with IN O
only RB O
liver NN B-C
enzyme NN I-C
elevation NN I-C
to IN O
severe JJ B-C
liver NN I-C
inflammation NN I-C
with IN O
development NN O
of IN O
jaundice NN B-C
, , O
prolonged VBN B-C
prothrombin NN I-C
time NN I-C
, , O
and CC O
even RB O
liver NN B-C
failure NN I-C
. . O

The DT O
syndrome NN B-C
causes VBZ O
no DT O
known VBN O
long JJ B-C
- HYPH I-C
term NN I-C
physical JJ I-C
damage NN I-C
. . O

Procaine NNP B-C
causes VBZ O
loss NN B-C
of IN I-C
feeling NN I-C
in IN O
the DT O
skin NN O
and CC O
surrounding VBG O
tissues NNS O
. . O

Along IN O
with IN O
Rhizoctonia NN O
solani NN O
, , O
attacks NNS B-C
by IN I-C
Pythium NN I-C
are VBP O
most RBS O
associated VBN O
with IN O
producing VBG B-C
roughly RB I-C
circular JJ I-C
patches NNS I-C
of IN I-C
dead JJ I-C
seedlings NNS I-C
. . O

Relatives NNS B-C
of IN I-C
the DT I-C
prisoners NNS I-C
gathered VBD I-C
outside IN I-C
the DT I-C
facility NN I-C
to TO O
discover VB O
the DT O
fate NN O
of IN O
the DT O
incarcerated VBN O
, , O
eventually RB O
leading VBG O
to IN O
clashes NNS B-C
with IN I-C
the DT I-C
police NN I-C
. . O

The DT O
disease NN B-C
is VBZ O
caused VBN O
by IN O
deposition NN B-C
of IN I-C
this DT I-C
dust NN I-C
in IN I-C
the DT I-C
lung NN I-C
. . O

They PRP O
are VBP O
therefore RB O
counselled VBN O
to TO O
avoid VB O
scuba NN O
diving NN O
, , O
during IN O
which WDT O
small JJ B-C
air NN I-C
bubbles NNS I-C
may MD O
form VB O
in IN O
the DT O
bloodsteam NN O
that WDT O
may MD O
migrate VB O
to IN O
the DT O
brain NN O
and CC O
cause VB O
stroke NN B-C
. . O

Weakened VBN B-C
pelvic JJ I-C
structures NNS I-C
occur VBP O
as IN O
a DT O
result NN O
of IN O
an DT O
episiotomy NN B-C
during IN I-C
previous JJ I-C
births NNS I-C
, , O
even RB O
decades NNS O
later RB O
. . O

In IN O
the DT O
TLE NN B-C
brain NN O
, , O
where WRB O
granule NN B-C
cells NNS I-C
are VBP I-C
damaged VBN I-C
or CC I-C
lost VBN I-C
, , O
axons NNS O
, , O
the DT O
mossy NN O
fibres NNS O
, , O
' `` O
sprout NN O
' '' O
in IN O
order NN O
to TO O
reconnect VB O
to IN O
other JJ O
granule NN O
cell NN O
dendrites NNS O
. . O

HSAN NNP B-C
IE NN I-C
is VBZ O
associated VBN O
with IN O
heterozygous JJ B-C
missense NN I-C
mutations NNS I-C
in IN I-C
the DT I-C
DNMT1 NN I-C
gene NN I-C
which WDT O
encodes VBZ O
DNA NN O
methyltransferase NN O
1 CD O
( -LRB- O
Dnmt NN O
1 CD O
) -RRB- O
. . O

Due IN O
to IN O
the DT O
highly RB B-C
conjugated VBN I-C
structure NN I-C
of IN I-C
porphyrins NNS I-C
involving VBG O
alternating VBG O
single JJ O
and CC O
double JJ O
carbon NN O
bonds NNS O
, , O
these DT O
compounds NNS B-C
exhibit VBP I-C
a DT I-C
deep JJ I-C
purple JJ I-C
color NN I-C
, , O
resulting VBG O
in IN O
the DT O
discoloration NN B-C
observed VBN I-C
in IN I-C
the DT I-C
skin NN I-C
. . O

This DT O
information NN O
may MD O
identify VB O
ineffective JJ B-C
platelet NN I-C
production NN I-C
as IN O
the DT O
cause NN O
of IN O
thrombocytopenia NN B-C
and CC O
rule VB O
out RP O
a DT O
malignant JJ B-C
disease NN I-C
process NN I-C
at IN O
the DT O
same JJ O
time NN O
. . O

In IN O
November NNP O
2006 CD O
the DT O
American NNP O
Dental NNP O
Association NNP O
published VBD O
information NN O
stating VBG O
that IN O
water NN O
fluoridation NN O
is VBZ O
safe JJ O
, , O
effective JJ O
and CC O
healthy JJ O
; , O
that IN O
enamel NN B-C
fluorosis NN I-C
, , O
usually RB O
mild JJ O
and CC O
difficult JJ O
for IN O
anyone NN O
except IN O
a DT O
dental JJ O
health NN O
care NN O
professional JJ O
to TO O
see VB O
, , O
can MD O
result VB O
from IN O
ingesting VBG B-C
more JJR I-C
than IN I-C
optimal JJ I-C
amounts NNS I-C
of IN I-C
fluoride NN I-C
in IN I-C
early JJ I-C
childhood NN I-C
; , O
that IN O
it PRP O
is VBZ O
safe JJ O
to TO O
use VB O
fluoridated VBN O
water NN O
to TO O
mix VB O
infant NN O
formula NN O
; , O
and CC O
that IN O
the DT O
probability NN O
of IN O
babies NNS O
developing VBG O
fluorosis NN O
can MD O
be VB O
reduced VBN O
by IN O
using VBG O
ready JJ O
- HYPH O
to IN O
- HYPH O
feed NN O
infant NN O
formula NN O
or CC O
using VBG O
water NN O
that WDT O
is VBZ O
either CC O
free JJ O
of IN O
fluoride NN O
or CC O
low JJ O
in IN O
fluoride NN O
to TO O
prepare VB O
powdered VBN O
or CC O
liquid NN O
concentrate NN O
formula NN O
. . O

Rectoceles NNS B-C
in IN I-C
men NNS I-C
are VBP O
uncommon JJ O
, , O
and CC O
associated VBN O
with IN O
prostatectomy NN B-C
. . O

Confusion NN B-C
emerged VBD I-C
when WRB O
a DT O
German JJ O
traveler NN O
recorded VBD O
that IN O
the DT O
ruler NN O
of IN O
Muscovy NNP O
at IN O
the DT O
time NN O
was VBD O
titled VBN O
' `` O
czar NN O
, , O
' '' O
due IN O
to IN O
a DT O
linguistic JJ B-C
mistake NN I-C
derived VBN O
from IN O
the DT O
fact NN O
that IN O
the DT O
Polish NNP O
, , O
with IN O
which WDT O
the DT O
German NNP O
had VBD O
greater JJR O
familiarity NN O
, , O
titled VBD O
some DT O
leaders NNS O
' POS O
czars NNS O
. . O
' `` O

Benign JJ B-C
fasciculation NN I-C
syndrome NN I-C
( -LRB- O
BFS NN O
) -RRB- O
is VBZ O
a DT O
neurological JJ O
disorder NN O
characterized VBN O
by IN O
fasciculation NN B-C
( -LRB- O
twitching NN O
) -RRB- O
of IN O
various JJ O
voluntary JJ O
muscles NNS O
in IN O
the DT O
body NN O
. . O

The DT O
crystallization NN B-C
of IN I-C
uric NN I-C
acid NN I-C
, , O
often RB O
related JJ O
to IN O
relatively RB O
high JJ O
levels NNS O
in IN O
the DT O
blood NN O
, , O
is VBZ O
the DT O
underlying JJ O
cause NN O
of IN O
gout NN B-C
. . O

In IN O
2010 CD O
, , O
Haiti NNP O
also RB O
experienced VBD O
a DT O
cholera NN B-C
outbreak NN I-C
that WDT O
spread VBD O
quickly RB O
throughout IN O
the DT O
nation NN O
due IN O
to IN O
difficulties NNS B-C
in IN I-C
healthcare JJ I-C
access NN I-C
and CC O
the DT O
lack NN B-C
of IN I-C
experience NN I-C
of IN I-C
the DT I-C
healthcare NN I-C
system NN I-C
. . O

With IN O
retraction NN B-C
of IN I-C
the DT I-C
lung NN I-C
tissue NN I-C
, , O
there EX O
is VBZ O
compensatory JJ B-C
emphysema NN I-C
. . O

Poverty NN B-C
is VBZ O
one CD O
of IN O
the DT O
biggest JJS O
by-products NNS O
of IN O
structural JJ B-C
violence NN I-C
, , O
as IN O
structural JJ O
violence NN O
inhibits VBZ O
individuals NNS O
and CC O
communities NNS O
from IN O
achieving VBG O
basic JJ O
human JJ O
rights NNS O
. . O

Dispersion NN B-C
of IN I-C
the DT I-C
granule NN I-C
cell NN I-C
layer NN I-C
in IN I-C
the DT I-C
hippocampal JJ I-C
dentate JJ I-C
gyrus NN I-C
is VBZ O
occasionally RB O
seen VBN O
in IN O
temporal JJ B-C
lobe NN I-C
epilepsy NN I-C
and CC O
has VBZ O
been VBN O
linked VBN O
to IN O
the DT O
downregulation NN B-C
of IN I-C
reelin NN I-C
, , O
a DT O
protein NN O
that WDT O
normally RB O
keeps VBZ O
the DT O
layer NN O
compact JJ O
by IN O
containing VBG O
neuronal JJ O
migration NN O
. . O

Body NN O
image NN O
in IN O
college NN O
students NNS O
is VBZ O
of IN O
great JJ O
concern NN O
because IN O
body NN B-C
dissatisfaction NN I-C
is VBZ O
one CD O
of IN O
the DT O
most RBS O
consistent JJ O
risk NN O
factors NNS O
for IN O
eating NN B-C
disorders NNS I-C
and CC O
is VBZ O
a DT O
significant JJ O
predictor NN O
of IN O
low JJ B-C
self NN I-C
- HYPH I-C
esteem NN I-C
, , O
depression NN B-C
, , O
and CC O
obesity NN B-C
. . O

These DT O
results NNS O
demonstrate VBP O
that IN O
resistance NN B-C
training NN I-C
is VBZ O
an DT O
effective JJ O
way NN O
to TO O
improve VB B-C
the DT I-C
muscular JJ I-C
strength NN I-C
capacity NN I-C
of IN I-C
older JJR I-C
adults NNS I-C
. . O

Infections NNS B-C
with IN I-C
Parvovirus NN I-C
B19 NN I-C
, , O
mycoplasma NN B-C
, , O
cytomegalovirus NN B-C
, , O
coxsackie NN B-C
B4 NN I-C
virus NN I-C
, , O
Chlamydophila NN B-C
pneumoniae NN I-C
, , O
E. NNP B-C
Coli NNP I-C
, , O
and CC O
Echinococcus NNP B-C
have VBP O
been VBN O
reported VBN O
to TO O
be VB O
associated VBN O
with IN O
the DT O
development NN O
of IN O
AGEP NNP B-C
in IN O
the DT O
absence NN O
of IN O
an DT O
apparent JJ O
drug NN O
- HYPH O
induced VBN O
cause NN O
. . O

In IN O
HHT NN O
, , O
some DT O
lesions NNS B-C
( -LRB- O
high JJ O
- HYPH O
flow NN O
arteriovenous JJ O
fistulae NN O
) -RRB- O
tend VBP O
to TO O
cause VB O
more JJR B-C
problems NNS I-C
, , O
and CC O
treatment NN O
is VBZ O
warranted VBN O
. . O

A DT O
diagnosis NN B-C
of IN I-C
binge NN I-C
eating NN I-C
is VBZ O
associated VBN O
with IN O
feelings NNS B-C
of IN I-C
loss NN I-C
of IN I-C
control NN I-C
. . O

Acute JJ B-C
radiodermatitis NN I-C
, , O
while IN O
presenting VBG O
as IN O
red JJ B-C
patches NNS I-C
, , O
may MD O
sometimes RB O
also RB O
present VB O
with IN O
desquamation NN B-C
or CC O
blistering NN B-C
. . O

A DT O
similarly RB O
small JJ O
proportion NN O
experiences NNS O
pulmonary JJ B-C
hypertension NN I-C
, , O
a DT O
state NN O
in IN O
which WDT O
the DT O
pressure NN O
in IN O
the DT O
lung NN O
arteries NNS O
is VBZ O
increased VBN O
, , O
exerting VBG O
pressure NN O
on IN O
the DT O
right JJ O
side NN O
of IN O
the DT O
heart NN O
and CC O
causing VBG O
peripheral JJ B-C
edema NN I-C
( -LRB- O
swelling NN O
of IN O
the DT O
legs NNS O
) -RRB- O
, , O
fainting NN B-C
and CC O
attacks NNS B-C
of IN I-C
chest NN I-C
pain NN I-C
. . O

When WRB O
we PRP O
see VBP B-C
another DT I-C
person NN I-C
suffering VBG I-C
, , O
it PRP O
can MD O
also RB O
cause VB O
us PRP O
pain NN B-C
. . O

Macrophomina NNP B-C
phaseoli VBD I-C
a DT O
plant NN O
pathogen NN O
that WDT O
causes VBZ O
charcoal NN B-C
rot NN I-C
on IN I-C
many JJ I-C
plant NN I-C
species NNS I-C
including VBG O
Zea NNP O
mays NNP O
and CC O
Pinus NNP O
elliottii NN O
. . O

Other JJ O
conditions NNS O
sometimes RB O
confused JJ O
with IN O
AGEP NNP O
include VBP O
pustular JJ B-C
eruptions NNS I-C
caused VBN O
by IN O
bacteria NN B-C
, , O
funguses NNS B-C
, , O
Herpesviridae NN B-C
, , O
and CC O
the DT O
Varicella JJ B-C
zoster NN I-C
virus NN I-C
( -LRB- O
i.e. FW O
causative JJ O
agent NN O
of IN O
chicken NN O
pox NN O
) -RRB- O
. . O

The DT O
first JJ O
case NN O
of IN O
hyperinsulinism NN B-C
due IN O
to IN O
a DT O
tumor NN B-C
of IN O
this DT O
type NN O
was VBD O
reported VBN O
in IN O
1927 CD O
. . O

About RB O
20 CD O
% NN O
are VBP O
affected VBN O
by IN O
symptomatic JJ O
digestive JJ O
tract NN O
lesions NNS B-C
, , O
although IN O
a DT O
higher JJR O
percentage NN O
have VBP O
lesions NNS O
that WDT O
do VBP O
not RB O
cause VB O
symptoms NNS B-C
. . O

In IN O
the DT O
context NN O
of IN O
dementia NN O
, , O
apraxia NN B-C
is VBZ O
a DT O
major JJ O
cause NN O
of IN O
morbidity NN B-C
, , O
and CC O
progresses VBZ O
with IN O
the DT O
underlying JJ O
disease NN O
sometimes RB O
to IN O
the DT O
extent NN O
that IN O
patients NNS O
may MD O
be VB O
unable JJ O
to TO O
feed VB O
themselves PRP O
or CC O
use VB O
simple JJ O
utensils NNS O
. . O

Together RB O
, , O
these DT O
studies NNS O
provide VBP O
evidence NN O
that IN O
dynapenia NN B-C
in IN I-C
older JJR I-C
adults NNS I-C
is VBZ O
strongly RB O
correlated VBN O
with IN O
increased VBN B-C
risk NN I-C
of IN I-C
physical JJ I-C
disabilities NNS I-C
and CC O
mortality NN B-C
, , O
and CC O
decreased VBN B-C
physical JJ I-C
function NN I-C
. . O

A DT O
sudden JJ B-C
death NN I-C
in IN I-C
a DT I-C
young JJ I-C
person NN I-C
can MD O
be VB O
caused VBN O
by IN O
heart NN B-C
disease NN I-C
( -LRB- O
including VBG O
cardiomyopathy NN B-C
, , O
congenital JJ B-C
heart NN I-C
disease NN I-C
, , O
myocarditis NN B-C
, , O
genetic JJ B-C
connective JJ I-C
tissue NN I-C
disorders NNS I-C
, , O
or CC O
conduction NN B-C
disease NN I-C
) -RRB- O
, , O
medication NN B-C
- HYPH I-C
related JJ I-C
causes NNS I-C
or CC O
other JJ O
causes NNS O
. . O

Congenital JJ B-C
liver NN I-C
abnormalities NNS I-C
, , O
such JJ O
as IN O
Caroli NNP B-C
's POS I-C
syndrome NN I-C
( -LRB- O
a DT O
specific JJ O
type NN O
of IN O
five CD O
recognized VBN O
choledochal JJ O
cysts NNS O
) -RRB- O
, , O
have VBP O
been VBN O
associated VBN O
with IN O
an DT O
approximately RB O
15 CD O
% NN O
lifetime NN O
risk NN O
of IN O
developing VBG O
cholangiocarcinoma NN B-C
. . O

Subendothelial NNP B-C
deposits NNS I-C
of IN I-C
amyloid NN I-C
may MD O
weaken VB O
blood NN O
vessels NNS O
and CC O
lead VB O
to IN O
the DT O
extravasation NN B-C
of IN I-C
blood NN I-C
. . O

Some DT O
experts NNS O
worry VBP O
that IN O
the DT O
Three NNP B-C
Gorges NNPS I-C
Dam NNP I-C
in IN O
China NNP O
may MD O
cause VB O
an DT O
increase NN B-C
in IN I-C
the DT I-C
frequency NN I-C
and CC I-C
intensity NN I-C
of IN I-C
earthquakes NNS I-C
. . O

Pupillary NN B-C
symptoms NNS I-C
of IN O
Holmes NNP O
– , O
Adie NNP O
syndrome NN O
are VBP O
thought VBN O
to TO O
be VB O
the DT O
result NN O
of IN O
a DT O
viral NN B-C
or CC I-C
bacterial JJ I-C
infection NN I-C
that WDT O
causes VBZ O
inflammation NN B-C
and CC O
damage NN B-C
to IN I-C
neurons NNS I-C
in IN I-C
the DT I-C
ciliary JJ I-C
ganglion NN I-C
, , O
located VBN O
in IN O
the DT O
posterior JJ O
orbit NN O
, , O
that WDT O
provides VBZ O
parasympathetic JJ O
control NN O
of IN O
eye NN O
constriction NN O
. . O

As IN O
a DT O
result NN O
, , O
a DT O
hemorrhage NN B-C
can MD O
occur VB O
in IN O
the DT O
corpus JJ O
luteum NN O
due IN O
to IN O
the DT O
fragility NN B-C
of IN I-C
blood NN I-C
vessels NNS I-C
, , O
causing VBG O
a DT O
hematoma NN B-C
. . O

In IN O
some DT O
cases NNS O
, , O
the DT O
loss NN B-C
is VBZ O
extremely RB O
sudden JJ O
and CC O
can MD O
be VB O
traced VBN O
to IN O
specific JJ B-C
diseases NNS I-C
, , O
such JJ O
as IN O
meningitis NN B-C
, , O
or CC O
to IN O
ototoxic JJ B-C
medications NNS I-C
, , O
such JJ O
as IN O
Gentamicin NNP B-C
. . O

Mutations NNS B-C
in IN I-C
the DT I-C
HMGCL NN I-C
gene NN I-C
cause VBP O
3-hydroxy-3-methylglutaryl JJ B-C
- HYPH I-C
CoA NN I-C
lyase NN I-C
deficiency NN I-C
. . O

Also RB O
, , O
the DT O
mining NN O
establishment NN O
of IN O
Delamar NNP O
Ghost NNP O
Town NNP O
, , O
Nevada NNP O
was VBD O
ruined VBN O
by IN O
a DT O
dry-mining NN B-C
process NN I-C
that WDT O
produced VBD O
a DT O
silicosis NN B-C
- HYPH O
causing VBG O
dust NN B-C
. . O

If IN O
someone NN B-C
causes VBZ O
harm NN B-C
to IN O
another DT O
, , O
and CC O
then RB O
feels VBZ O
guilt NN O
and CC O
demonstrates VBZ O
regret NN O
and CC O
sorrow NN O
, , O
the DT O
person NN O
harmed VBN O
is VBZ O
likely JJ O
to TO O
forgive VB O
. . O

Studies NNS O
have VBP O
suggested VBN O
that IN O
the DT O
cytopathic JJ B-C
effect NN I-C
of IN O
the DT O
virus NN O
on IN O
hepatocytes NNS O
can MD O
lead VB O
to IN O
the DT O
release NN O
of IN O
free JJ B-C
iron NN I-C
. . O

PMF NN B-C
is VBZ I-C
associated VBN O
with IN O
more RBR O
severe JJ B-C
symptoms NNS I-C
and CC O
respiratory JJ B-C
impairment NN I-C
than IN O
simple JJ O
disease NN O
. . O

Enamel NN O
fluorosis NN O
has VBZ O
a DT O
white JJ B-C
opaque JJ I-C
appearance NN I-C
which WDT O
is VBZ O
due IN O
to IN O
the DT O
surface NN B-C
of IN I-C
the DT I-C
enamel NN I-C
being VBG I-C
hypomineralised VBN I-C
. . O

AGE NN B-C
typically RB O
shows VBZ O
a DT O
mild JJ O
course NN O
: : O
usually RB O
, , O
it PRP O
is VBZ O
not RB O
associated VBN O
with IN O
life NN B-C
- , I-C
threatening VBG I-C
complicates VBZ I-C
although IN O
superinfections NNS O
of IN O
skin NN O
lesions NNS O
may MD O
be VB O
serious JJ O
or CC O
even RB O
life NN O
- HYPH O
threatening VBG O
. . O

Since IN O
geological JJ B-C
sequestration NN I-C
of IN I-C
carbon NN I-C
dioxide NN I-C
has VBZ O
the DT O
potential NN O
to TO O
induce VB O
seismicity NN B-C
, , O
researchers NNS O
have VBP O
developed VBN O
methods NNS O
to TO O
monitor VB O
and CC O
model VB O
the DT O
risk NN O
of IN O
injection NN B-C
- HYPH O
induced VBN O
seismicity NN B-C
, , O
in IN O
order NN O
to TO O
better RBR O
manage VB O
the DT O
risks NNS O
associated VBN O
with IN O
this DT O
phenomenon NN O
. . O

This DT O
temperature NN O
profile NN O
, , O
known VBN O
as IN O
a DT O
temperature NN B-C
inversion NN I-C
, , O
will MD O
lead VB O
to IN O
the DT O
development NN O
of IN O
drizzle NN B-C
, , O
rain NN B-C
, , O
freezing NN B-C
rain NN I-C
, , O
sleet NN B-C
, , O
or CC O
snow NN B-C
. . O

Edema NN B-C
of IN I-C
dermal JJ I-C
papillae NNS I-C
, , O
if IN O
present JJ O
within IN O
48 CD O
hours NNS O
since IN O
the DT O
exposition NN O
, , O
is VBZ O
followed VBN O
by IN O
transepidermal NN B-C
necrosis NN I-C
. . O

The DT O
rash NN B-C
takes VBZ O
one CD O
to IN O
two CD O
weeks NNS O
to TO O
run VB O
its PRP$ O
course NN O
and CC O
may MD O
cause VB O
scars NNS B-C
, , O
depending VBG O
on IN O
the DT O
severity NN O
of IN O
the DT O
exposure NN O
. . O

It PRP O
is VBZ O
caused VBN O
by IN O
damage NN O
to IN O
the DT O
postganglionic JJ O
fibers NNS O
of IN O
the DT O
parasympathetic JJ O
innervation NN O
of IN O
the DT O
eye NN O
, , O
usually RB O
by IN O
a DT O
viral NN B-C
or CC I-C
bacterial JJ I-C
infection NN I-C
which WDT O
causes VBZ O
inflammation NN B-C
, , O
and CC O
affects VBZ O
the DT O
pupil NN O
of IN O
the DT O
eye NN O
and CC O
the DT O
autonomic JJ O
nervous JJ O
system NN O
. . O

During IN O
underground JJ O
nuclear JJ O
testing NN O
in IN O
Nevada NNP O
, , O
several JJ O
workers NNS O
developed VBD O
burns NNS B-C
and CC O
skin NN B-C
ulcers NNS I-C
, , O
in IN O
part NN O
attributed VBN O
to IN O
exposure NN B-C
to IN I-C
tritium NN I-C
. . O

Echocardiography NN O
is VBZ O
also RB O
used VBN O
if IN O
there EX O
is VBZ O
a DT O
suspicion NN O
of IN O
pulmonary JJ B-C
hypertension NN I-C
or CC O
high JJ B-C
- HYPH I-C
output NN I-C
cardiac JJ I-C
failure NN I-C
due IN O
to IN O
large JJ B-C
liver NN I-C
lesions NNS I-C
, , O
sometimes RB O
followed VBN O
by IN O
cardiac JJ O
catheterization NN O
to TO O
measure VB O
the DT O
pressures NNS O
inside IN O
the DT O
various JJ O
chambers NNS O
of IN O
the DT O
heart NN O
. . O

Because IN O
of IN O
work NN B-C
- HYPH I-C
exposure NN I-C
to IN I-C
silica NN I-C
dust NN O
, , O
silicosis NN B-C
is VBZ I-C
an DT I-C
occupational JJ I-C
hazard NN I-C
to IN I-C
mining NN I-C
, , O
sandblasting NN O
, , O
quarry NN O
, , O
ceramics NNS O
and CC O
foundry NN O
workers NNS O
, , O
as RB O
well RB O
as IN O
grinders NNS O
, , O
stone NN O
cutters NNS O
, , O
stone NN O
countertops NNS O
, , O
refractory JJ O
brick NN O
workers NNS O
, , O
tombstone NN O
workers NNS O
, , O
workers NNS O
in IN O
the DT O
oil NN O
and CC O
gas NN O
industry NN O
, , O
pottery NN O
workers NNS O
, , O
fiberglass NN O
manufacturing NN O
, , O
glass NN O
manufacturing NN O
, , O
flint NN O
knappers NNS O
and CC O
others NNS O
. . O

Typically RB O
, , O
patients NNS O
who WP O
are VBP O
ultimately RB O
diagnosed VBN O
with IN O
PCT NNP B-C
first RB O
seek VBP O
treatment NN O
following VBG O
the DT O
development NN O
of IN O
photosensitivities NNS B-C
in IN O
the DT O
form NN O
of IN O
blisters NNS B-C
and CC O
erosions NNS B-C
on IN O
commonly RB O
exposed VBN O
areas NNS O
of IN O
the DT O
skin NN O
. . O

On IN O
snow—even NN O
at IN O
concentrations NNS O
below IN O
five CD O
parts NNS O
per IN O
billion CD O
– , O
dark JJ B-C
carbon NN I-C
triggers NNS O
melting NN B-C
, , O
and CC O
may MD O
be VB O
responsible JJ O
for IN O
as RB O
much JJ O
as IN O
94 CD O
percent NN O
of IN O
Arctic JJ B-C
warming NN I-C
. . O

These DT O
include VBP O
: : O
Chronic NN B-C
simple JJ I-C
silicosis NN I-C
: : O
Usually RB O
resulting VBG O
from IN O
long JJ B-C
- HYPH I-C
term NN I-C
exposure NN I-C
( -LRB- I-C
10 CD I-C
years NNS I-C
or CC I-C
more JJR I-C
) -RRB- I-C
to IN I-C
relatively RB I-C
low JJ I-C
concentrations NNS I-C
of IN I-C
silica NN I-C
dust NN I-C
and CC O
usually RB O
appearing VBG O
10 CD O
– SYM O
30 CD O
years NNS O
after IN O
first JJ O
exposure NN O
. . O

Second RB O
, , O
the DT O
breathing NN B-C
tube NN I-C
may MD I-C
be VB I-C
removed VBN I-C
before IN I-C
the DT I-C
patient NN I-C
is VBZ I-C
strong JJ I-C
enough JJ I-C
to TO I-C
breathe VB I-C
properly RB I-C
, , O
potentially RB O
causing VBG O
respiratory JJ B-C
arrest NN I-C
. . O

Mutations NNS B-C
in IN I-C
the DT I-C
HADH NN I-C
gene NN I-C
lead NN O
to IN O
inadequate JJ B-C
levels NNS I-C
of IN I-C
an DT I-C
enzyme NN I-C
called VBN I-C
3-hydroxyacyl NN I-C
- HYPH I-C
coenzyme NN I-C
A DT I-C
dehydrogenase NN I-C
. . O

Dietary NN B-C
causes VBZ O
account NN O
for IN O
about RB O
12 CD O
% NN O
of IN O
gout NN B-C
, , O
and CC O
include VBP O
a DT O
strong JJ O
association NN O
with IN O
the DT O
consumption NN B-C
of IN I-C
alcohol NN I-C
, , O
fructose NN B-C
- HYPH I-C
sweetened VBN I-C
drinks NNS I-C
, , O
meat NN B-C
, , O
and CC O
seafood NN B-C
. . O

Third RB O
, , O
there EX O
are VBP O
no DT O
underlying JJ O
illnesses NNS B-C
that WDT O
are VBP O
more RBR O
likely JJ O
to TO O
be VB O
causing VBG O
the DT O
abnormalities NNS B-C
. . O

The DT O
detergent NN B-C
causes VBZ O
the DT O
vessel NN B-C
to TO I-C
collapse VB I-C
and CC I-C
harden VB I-C
, , O
resulting VBG O
in IN O
scar NN B-C
tissue NN I-C
residue NN I-C
. . O

Silicosis NN B-C
( -LRB- O
particularly RB O
the DT O
acute JJ O
form NN O
) -RRB- O
is VBZ O
characterized VBN O
by IN O
shortness NN B-C
of IN I-C
breath NN I-C
, , O
cough NN B-C
, , O
fever NN B-C
, , O
and CC O
cyanosis NN B-C
( -LRB- O
bluish JJ O
skin NN O
) -RRB- O
. . O

In IN O
the DT O
United NNP O
States NNP O
, , O
a DT O
1930 CD O
epidemic NN B-C
of IN I-C
silicosis NN I-C
due IN O
to IN O
the DT O
construction NN B-C
of IN I-C
the DT I-C
Hawk NNP I-C
's POS I-C
Nest NNP I-C
Tunnel NNP I-C
near IN I-C
Gauley NNP I-C
Bridge NNP I-C
, , I-C
West NNP I-C
Virginia NNP I-C
caused VBD O
the DT O
death NN B-C
of IN O
at RB O
least RBS O
400 CD O
workers NNS O
. . O

MADH4 NN B-C
mutations NNS I-C
, , O
which WDT O
cause VBP O
colonic JJ B-C
polyposis NN I-C
in IN O
addition NN O
to IN O
HHT NN B-C
, , O
comprise VBP O
about RB O
2 CD O
% NN O
of IN O
disease NN B-C
- HYPH O
causing VBG O
mutations NNS B-C
. . O

Temporal JJ B-C
lobe NN I-C
epilepsy NN I-C
is VBZ O
not RB O
the DT O
result NN O
of IN O
psychiatric JJ B-C
illness NN I-C
or CC O
fragility NN B-C
of IN I-C
the DT I-C
personality NN I-C
. . O

When WRB O
episodes NNS O
occur VBP O
in IN O
an DT O
infant NN O
or CC O
child NN O
, , O
blood NN O
sugar NN O
becomes VBZ O
extremely RB O
low JJ O
( -LRB- O
hypoglycemia NN O
) -RRB- O
, , O
and CC O
harmful JJ B-C
compounds NNS I-C
can MD O
build VB O
up RP O
and CC O
cause VB O
the DT O
blood NN B-C
to TO I-C
become VB I-C
too RB I-C
acidic JJ I-C
( -LRB- O
metabolic NN O
acidosis NN O
) -RRB- O
. . O

By IN O
far RB O
the DT O
most RBS O
common JJ O
cause NN O
of IN O
mixed VBN B-C
- HYPH I-C
field NN I-C
agglutination NN I-C
is VBZ O
false JJ B-C
chimerism NN I-C
. . O

Causes NNS O
that WDT O
have VBP O
resulted VBN O
in IN O
higher JJR O
levels NNS O
of IN O
structural JJ B-C
violence NN I-C
within IN O
Haiti NNP O
include VBP O
political JJ B-C
instability NN I-C
and CC O
corruption NN B-C
, , O
as RB O
well RB O
as IN O
the DT O
impact NN B-C
of IN I-C
post-colonialism JJ I-C
, , O
which WDT O
has VBZ O
established VBN O
a DT O
caste NN O
- HYPH O
based VBN O
class NN O
system NN O
within IN O
Haiti NNP O
. . O

The DT O
abnormally RB B-C
widened VBN I-C
trachea NN I-C
and CC I-C
mainstem NN I-C
bronchi NN I-C
are VBP O
associated VBN O
with IN O
recurrent JJ B-C
lower JJR I-C
respiratory JJ I-C
tract NN I-C
infection NN I-C
and CC O
copious JJ B-C
purulent JJ I-C
sputum NN I-C
production NN I-C
, , O
eventually RB O
leading VBG O
to IN O
bronchiectasis NN B-C
and CC O
other JJ O
respiratory JJ B-C
complications NNS I-C
. . O

Weaver NNP B-C
syndrome NN I-C
and CC I-C
Sotos NNP I-C
syndrome NN I-C
are VBP I-C
often RB I-C
mistaken JJ I-C
for IN I-C
one CD I-C
another DT I-C
due IN O
to IN O
their PRP$ O
significant JJ O
phenotypic JJ B-C
overlap NN I-C
and CC O
similarities NNS B-C
. . O

The DT O
SA NNP B-C
Ar NNP I-C
94 CD I-C
strain NN I-C
had VBD O
been VBN O
observed VBN O
causing VBG O
dissemination NN B-C
infection NN I-C
in IN I-C
Ae. NNP I-C

Primary JJ B-C
erythema NN I-C
lasting VBG O
more JJR O
than IN O
72 CD O
hours NNS O
is VBZ O
an DT O
indication NN O
of IN O
injury NN O
severe JJ O
enough JJ O
to TO O
cause VB O
chronic JJ B-C
radiation NN I-C
dermatitis NN I-C
. . O

According VBG O
to IN O
Daniel NNP O
Katz NNP O
and CC O
Kenneth NNP O
Braly NNP O
, , O
stereotyping NN B-C
leads VBZ O
to IN O
racial JJ B-C
prejudice NN I-C
when WRB O
people NNS O
emotionally RB O
react VBP O
to IN O
the DT O
name NN O
of IN O
a DT O
group NN O
, , O
ascribe VBP O
characteristics NNS O
to IN O
members NNS O
of IN O
that DT O
group NN O
, , O
and CC O
then RB O
evaluate VB O
those DT O
characteristics NNS O
. . O

Although IN O
people NNS O
who WP O
do VBP O
not RB O
have VB O
any DT O
eating NN O
disorder NN O
may MD O
occasionally RB O
experience VB O
episodes NNS O
of IN O
overeating NN O
, , O
frequent JJ B-C
binge NN I-C
eating NN I-C
is VBZ O
often RB O
a DT O
symptom NN O
of IN O
an DT O
eating NN B-C
disorder NN I-C
. . O

The DT O
loss NN B-C
of IN I-C
the DT I-C
GABA NN I-C
- HYPH I-C
mediated VBN I-C
inhibitory JJ I-C
interneurons NNS I-C
may MD O
increase VB O
the DT O
hyperexcitability NN B-C
of IN I-C
neurons NNS I-C
of IN I-C
the DT I-C
hippocampus NN I-C
leading VBG O
to IN O
recurrent JJ B-C
seizures NNS I-C
. . O

The DT O
wildfire NN B-C
resulted VBD O
in IN O
evacuation NN B-C
of IN I-C
500 CD I-C
campers NNS I-C
from IN O
the DT O
Pio NNP O
Pico NNP O
RV NNP O
Resort NNP O
and CC O
Campground NNP O
. . O

Polish JJ O
demographer NN O
Piotr NNP O
Eberhardt NNP O
maintains VBZ O
that IN O
it PRP O
is VBZ O
commonly RB O
agreed VBN O
that IN O
the DT O
criterion NN B-C
of IN I-C
declared VBN I-C
language NN I-C
to TO I-C
classify VB I-C
ethnic JJ I-C
groups NNS I-C
led VBD O
to IN O
an DT O
overestimation NN B-C
of IN I-C
the DT I-C
number NN I-C
of IN I-C
Poles NNS I-C
in IN I-C
pre-war JJ I-C
Poland NNP I-C
. . O

Enhanced VBN B-C
geothermal JJ I-C
systems NNS I-C
( -LRB- O
EGS NNP O
) -RRB- O
, , O
a DT O
new JJ O
type NN O
of IN O
geothermal JJ O
power NN O
technologies NNS O
that WDT O
do VBP O
not RB O
require VB O
natural JJ O
convective JJ O
hydrothermal JJ O
resources NNS O
, , O
are VBP O
known VBN O
to TO O
be VB O
associated VBN O
with IN O
induced VBN B-C
seismicity NN I-C
. . O

There EX O
is VBZ O
one CD O
known VBN O
mutation NN B-C
in IN O
this DT O
gene NN O
that WDT O
is VBZ O
associated VBN O
with IN O
JME NNP B-C
, , O
A322D NNP B-C
, , O
which WDT O
is VBZ O
located VBN O
in IN O
the DT O
third JJ O
segment NN O
of IN O
the DT O
protein NN O
. . O

Large JJ B-C
hemangiomas NNS I-C
can MD O
leave VB O
visible JJ B-C
skin NN I-C
changes NNS I-C
secondary JJ O
to IN O
significant JJ O
stretching NN O
of IN O
the DT O
skin NN O
or CC O
alteration NN O
of IN O
surface NN O
texture NN O
. . O

Due IN O
to IN O
diffusion NN B-C
of IN I-C
exogenous JJ I-C
ions NNS I-C
( -LRB- O
ex NN O
, , O
iron NN O
and CC O
copper NN O
) -RRB- O
, , O
the DT O
stains NNS B-C
would MD I-C
develop VB I-C
into IN I-C
the DT I-C
abnormally RB I-C
porous JJ I-C
enamel NN I-C
. . O

These DT O
stereotype NN B-C
- HYPH I-C
based VBN I-C
expectations NNS I-C
may MD O
lead VB O
to IN O
self NN B-C
- HYPH I-C
fulfilling VBG I-C
prophecies NNS I-C
, , O
in IN O
which WDT O
one NN O
's POS O
inaccurate JJ O
expectations NNS O
about IN O
a DT O
person NN O
's POS O
behavior NN O
, , O
through IN O
social JJ O
interaction NN O
, , O
prompt VB O
that DT O
person NN O
to TO O
act VB O
in IN O
stereotype NN O
- HYPH O
consistent JJ O
ways NNS O
, , O
thus RB O
confirming VBG O
one PRP O
's POS O
erroneous JJ O
expectations NNS O
and CC O
validating VBG O
the DT O
stereotype NN O
. . O

Trousseau NNP B-C
's POS I-C
syndrome NN I-C
, , O
in IN O
which WDT O
blood NN B-C
clots VBZ I-C
form NN I-C
spontaneously RB I-C
in IN I-C
the DT I-C
portal JJ I-C
blood NN I-C
vessels NNS I-C
, , O
the DT O
deep JJ O
veins NNS O
of IN O
the DT O
extremities NNS O
, , O
or CC O
the DT O
superficial JJ O
veins NNS O
anywhere RB O
on IN O
the DT O
body NN O
, , O
may MD O
be VB O
associated VBN O
with IN O
pancreatic JJ B-C
cancer NN I-C
, , O
and CC O
is VBZ O
found VBN O
in IN O
about RB O
10 CD O
% NN O
of IN O
cases NNS O
. . O

A DT O
study NN O
done VBN O
on IN O
60 CD O
students NNS O
at IN O
Cornell NNP O
University NNP O
showed VBD O
that IN O
20 CD O
% NN O
of IN O
the DT O
weight NN B-C
gained VBN I-C
by IN O
the DT O
test NN O
subjects NNS O
was VBD O
directly RB O
caused VBN O
by IN O
the DT O
dining NN B-C
halls NNS I-C
' POS I-C
all DT I-C
- HYPH I-C
you PRP I-C
- HYPH I-C
can MD I-C
- HYPH I-C
eat VB I-C
nature NN I-C
. . O

Weaver NNP B-C
syndrome NN I-C
( -LRB- O
also RB O
called VBN O
Weaver NNP O
- HYPH O
Smith NNP O
syndrome NN O
) -RRB- O
is VBZ O
an DT O
extremely RB O
rare JJ O
congenital JJ O
disorder NN O
associated VBN O
with IN O
rapid JJ B-C
growth NN I-C
beginning VBG I-C
in IN I-C
the DT I-C
prenatal NN I-C
period NN I-C
and CC O
continuing VBG O
through IN O
the DT O
toddler NN O
and CC O
youth JJ O
years NNS O
. . O

The DT O
Black JJ B-C
Thursday NNP I-C
bushfires NNS I-C
, , O
were VBD O
caused VBN O
in IN O
part NN O
by IN O
an DT O
intense JJ B-C
drought NN I-C
that WDT O
occurred VBD O
throughout IN O
1850 CD O
when WRB O
the DT O
continent NN O
suffered VBD O
from IN O
extreme JJ O
heat NN O
. . O

At IN O
high JJ O
levels NNS O
, , O
uric NN B-C
acid NN I-C
crystallizes VBZ I-C
and CC O
the DT O
crystals NNS B-C
deposit NN I-C
in IN I-C
joints NNS I-C
, , I-C
tendons NNS I-C
, , I-C
and CC I-C
surrounding VBG I-C
tissues NNS I-C
, , O
resulting VBG O
in IN O
an DT O
attack NN B-C
of IN I-C
gout NN I-C
. . O

Porphyria NN B-C
cutanea NN I-C
tarda NN I-C
is VBZ O
primarily RB O
caused VBN O
by IN O
uroporphyrinogen NN B-C
decarboxylase NN I-C
deficiency NN I-C
( -LRB- O
UROD NNP O
) -RRB- O
. . O

Interferon NNP B-C
- HYPH I-C
alpha JJ I-C
2 CD I-C
a NN I-C
and CC I-C
2 CD I-C
b NN I-C
, , O
given VBN O
via IN O
subcutaneous JJ O
injection NN O
, , O
has VBZ O
shown VBN O
efficacy NN O
against IN O
hemangiomas NNS O
, , O
but CC O
may MD O
result VB O
in IN O
spastic JJ B-C
diplegia NN I-C
in IN O
up IN O
to IN O
20 CD O
% NN O
of IN O
treated VBN O
children NNS O
. . O

The DT O
fishing NN O
ship NN O
Daigo NNP O
Fukuryu NNP O
Maru NNP O
was VBD O
affected VBN O
by IN O
the DT O
fallout NN O
as RB O
well RB O
; , O
the DT O
crew NN O
suffered VBD O
skin NN O
doses NNS O
between IN O
1.7 CD O
– SYM O
6.0 CD O
Gy NN O
, , O
with IN O
beta NN B-C
burns NNS I-C
manifesting VBG O
as IN O
severe JJ B-C
skin NN I-C
lesions NNS I-C
, , O
erythema NN B-C
, , O
erosions NNS B-C
, , O
sometimes RB O
necrosis JJ B-C
, , O
and CC O
skin NN B-C
atrophy NN I-C
. . O

This DT O
allergic JJ B-C
reaction NN I-C
may MD O
be VB O
triggered VBN O
by IN O
small JJ B-C
amounts NNS I-C
of IN I-C
egg NN I-C
, , O
even RB O
egg NN B-C
incorporated VBN I-C
into IN I-C
cooked VBN I-C
foods NNS I-C
, , O
such JJ O
as IN O
cake NN O
. . O

Although IN O
the DT O
theory NN O
is VBZ O
controversial JJ O
, , O
there EX O
is VBZ O
a DT O
link NN O
between IN O
febrile JJ B-C
seizures NNS I-C
( -LRB- O
seizures NNS O
coinciding VBG O
with IN O
episodes NNS O
of IN O
fever NN O
in IN O
young JJ O
children NNS O
) -RRB- O
and CC O
subsequent JJ O
temporal JJ B-C
lobe NN I-C
epilepsy NN I-C
, , O
at RB O
least RBS O
epidemiologically RB O
. . O

Also RB O
, , O
shingles NNS B-C
can MD O
cause VB O
a DT O
painful JJ B-C
blistering JJ I-C
rash NN I-C
on IN I-C
the DT I-C
skin NN I-C
of IN I-C
the DT I-C
breasts NNS I-C
. . O

Beta NNP O
burns NNS O
were VBD O
a DT O
serious JJ O
medical JJ O
issue NN O
for IN O
some DT O
victims NNS O
of IN O
the DT O
Chernobyl NNP O
disaster NN O
; , O
from IN O
115 CD O
patients NNS O
treated VBN O
in IN O
Moscow NNP O
, , O
30 CD O
% NN O
had VBD O
burns NNS O
covering VBG O
10– CD O
50 CD O
% NN O
of IN O
body NN O
surface NN O
, , O
11 CD O
% NN O
were VBD O
affected VBN O
on IN O
50 CD O
– SYM O
100 CD O
% NN O
of IN O
skin NN O
; , O
the DT O
massive JJ B-C
exposure NN I-C
was VBD O
often RB O
caused VBN O
by IN O
clothes NNS B-C
drenched VBN I-C
with IN I-C
radioactive JJ I-C
water NN I-C
. . O

Given VBN O
that IN O
liver NN B-C
AVMs NNS I-C
generally RB O
cause VB O
high JJ B-C
- HYPH I-C
output NN I-C
cardiac JJ I-C
failure NN I-C
, , O
the DT O
emphasis NN O
is VBZ O
on IN O
treating VBG O
this DT O
with IN O
diuretics NNS O
to TO O
reduce VB O
the DT O
circulating VBG O
blood NN O
volume NN O
, , O
restriction NN O
of IN O
salt NN O
and CC O
fluid NN O
intake NN O
, , O
and CC O
antiarrhythmic JJ O
agents NNS O
in IN O
case NN O
of IN O
irregular JJ O
heart NN O
beat VBP O
. . O

Those DT O
with IN O
nevus JJ O
depigmentosus NN O
may MD O
be VB O
prone JJ O
to IN O
sunburn NN B-C
due IN O
to IN O
the DT O
lack NN B-C
of IN I-C
pigment NN I-C
, , O
and CC O
the DT O
patient NN O
should MD O
use VB O
good JJ O
sun NN O
protection NN O
. . O

When WRB O
fine JJ B-C
particles NNS I-C
of IN I-C
crystalline JJ I-C
silica NN I-C
dust NN I-C
are VBP I-C
deposited VBN I-C
in IN I-C
the DT I-C
lungs NNS I-C
, , O
macrophages NNS B-C
that WDT I-C
ingest VBP I-C
the DT I-C
dust NN I-C
particles NNS I-C
will MD I-C
set VB I-C
off RP I-C
an DT I-C
inflammation NN I-C
response NN I-C
by IN O
releasing VBG O
tumor NN O
necrosis NN O
factors NNS O
, , O
interleukin NN O
- HYPH O
1 CD O
, , O
leukotriene NN O
B4 NN O
and CC O
other JJ O
cytokines NNS O
. . O

Why WRB O
some DT O
proteins NNS B-C
trigger VBP O
allergic JJ B-C
reactions NNS I-C
while IN O
others NNS O
do VBP O
not RB O
is VBZ O
not RB O
entirely RB O
clear JJ O
, , O
although IN O
in IN O
part NN O
thought VBD O
to TO O
be VB O
due IN O
to IN O
resistance NN O
to IN O
digestion NN O
. . O

Hereditary JJ B-C
pancreatitis NN I-C
gives VBZ O
a DT O
greatly RB O
increased VBN O
lifetime NN O
risk NN O
of IN O
pancreatic JJ B-C
cancer NN I-C
of IN O
30–40 CD O
% NN O
to IN O
the DT O
age NN O
of IN O
70 CD O
. . O

Mutations NNS B-C
in IN I-C
the DT I-C
NSD1 NN I-C
gene NN I-C
may MD O
also RB O
be VB O
another DT O
cause NN O
for IN O
confusion NN B-C
. . O

A DT O
flaw NN B-C
in IN I-C
the DT I-C
construction NN I-C
design NN I-C
was VBD O
cited VBN O
as IN O
the DT O
cause NN O
for IN O
collapse NN B-C
. . O

With IN O
interventional JJ B-C
fluoroscopy NN I-C
, , O
because IN O
of IN O
the DT O
high JJ O
skin NN O
doses NNS O
that WDT O
can MD O
be VB O
generated VBN O
in IN O
the DT O
course NN O
of IN O
the DT O
intervention NN O
, , O
some DT O
procedures NNS O
have VBP O
resulted VBN O
in IN O
early RB O
( -LRB- O
less JJR O
than IN O
two CD O
months NNS O
after IN O
exposure NN O
) -RRB- O
and CC O
/ , O
or CC O
late RB O
( -LRB- O
two CD O
months NNS O
or CC O
more JJR O
after IN O
exposure NN O
) -RRB- O
skin NN B-C
reactions NNS I-C
, , O
including VBG O
necrosis NN B-C
in IN O
some DT O
cases NNS O
. . O

By IN O
definition NN O
, , O
primary JJ B-C
immune JJ I-C
deficiencies NNS I-C
are VBP O
due IN O
to IN O
genetic JJ B-C
causes NNS I-C
. . O

The DT O
resulting VBG B-C
asymmetrical JJ I-C
lift NN I-C
caused VBD O
an DT O
uncommanded JJ B-C
roll NN I-C
to IN I-C
the DT I-C
left NN I-C
. . O

Indications NNS O
for IN O
treatment NN O
of IN O
a DT O
hemangioma NN B-C
include VBP O
functional JJ B-C
impairment NN I-C
( -LRB- O
ie FW O
visual JJ O
or CC O
feeding NN O
compromise NN O
) -RRB- O
, , O
bleeding NN B-C
, , O
potentially RB B-C
life NN I-C
- HYPH I-C
threatening VBG I-C
complications NNS I-C
( -LRB- O
ie FW O
airway NN O
, , O
cardiac JJ O
or CC O
hepatic JJ O
disease NN O
) -RRB- O
, , O
and CC O
risk NN O
of IN O
long JJ B-C
- HYPH I-C
term NN I-C
or CC I-C
permanent JJ I-C
disfigurement NN I-C
. . O

The DT O
end NN B-C
of IN I-C
Prohibition NNP I-C
prompted VBD O
the DT O
opening NN B-C
of IN I-C
several JJ I-C
gay JJ I-C
bars NNS I-C
along IN I-C
North NNP I-C
Beach NNP I-C
. . O

Restated VBN B-C
, , I-C
chronic JJ I-C
radiodermatitis NN I-C
, , O
squamous JJ B-C
and CC O
basal JJ B-C
cell NN I-C
carcinomas NNS I-C
may MD O
develop VB O
months NNS O
to IN O
years NNS O
after IN O
radiation NN B-C
exposure NN I-C
. . O

Radiation NN B-C
dermatitis NN I-C
occurs VBZ O
to IN O
some DT O
degree NN O
in IN O
most JJS O
patients NNS O
receiving VBG O
radiation NN B-C
therapy NN I-C
, , O
with IN O
or CC O
without IN O
chemotherapy NN O
. . O

The DT O
presence NN O
of IN O
combined VBN B-C
injury NN I-C
( -LRB- O
exposure NN O
to IN O
radiation NN O
plus CC O
trauma NN O
or CC O
radiation NN O
burn NN O
) -RRB- O
increases VBZ O
the DT O
likelihood NN O
of IN O
generalized VBN B-C
sepsis NN I-C
. . O

The DT O
lack NN B-C
of IN I-C
development NN I-C
and CC I-C
spending NN I-C
on IN I-C
healthcare NN I-C
resources NNS I-C
and CC I-C
services NNS I-C
makes VBZ O
Haitians NNPS O
, , O
particularly RB O
those DT O
living VBG O
in IN O
rural JJ O
areas NNS O
, , O
susceptible JJ B-C
to IN I-C
diseases NNS I-C
that WDT O
are VBP O
otherwise RB O
manageable JJ O
. . O

Symptoms NNS O
of IN O
acute JJ B-C
silicosis NN I-C
include VBP O
more RBR O
rapid JJ O
onset NN O
of IN O
severe JJ O
disabling NN O
shortness NN B-C
of IN I-C
breath NN I-C
, , O
cough NN B-C
, , O
weakness NN B-C
, , O
and CC O
weight NN B-C
loss NN I-C
, , O
often RB O
leading VBG O
to IN O
death NN B-C
. . O

Recent JJ O
evidence NN O
suggests VBZ O
that IN O
aging NN B-C
is VBZ O
also RB O
related JJ O
to IN O
the DT O
loss NN B-C
of IN I-C
myelinated VBN I-C
nerve NN I-C
fiber NN I-C
length NN I-C
and CC I-C
the DT I-C
mass NN I-C
of IN I-C
white JJ I-C
matter NN I-C
, , O
with IN O
individuals NNS O
losing VBG O
approximately RB O
45 CD O
% NN O
of IN O
total JJ O
nerve NN O
fiber NN O
length NN O
as IN O
they PRP O
age VBP O
. . O

Even RB O
if IN O
they PRP O
had VBD O
, , O
an DT O
excessive JJ B-C
rate NN I-C
of IN I-C
descent NN I-C
and CC I-C
excessive JJ I-C
airspeed NN I-C
can MD O
cause VB O
the DT O
TAWS NN B-C
to TO I-C
issue VB I-C
a DT I-C
" `` I-C
SINK NN I-C
RATE NN I-C
" '' I-C
warning NN I-C
followed VBN I-C
by IN I-C
a DT I-C
" `` I-C
PULL VB I-C
UP NN I-C
" '' I-C
warning NN I-C
. . O

Eventually RB O
, , O
Anakin NNP B-C
's POS I-C
younger JJR I-C
age NN I-C
led VBD O
Lucas NNP B-C
to TO I-C
rewrite VB I-C
his PRP$ I-C
participation NN I-C
in IN I-C
the DT I-C
movie NN I-C
's POS I-C
major JJ I-C
scenes NNS I-C
. . O

Chronic NN B-C
lead NN I-C
exposure NN I-C
and CC O
lead NN B-C
- HYPH I-C
contaminated VBN I-C
alcohol NN I-C
are VBP O
risk NN O
factors NNS O
for IN O
gout NN B-C
due IN O
to IN O
the DT O
harmful JJ O
effect NN O
of IN O
lead NN O
on IN O
kidney NN O
function NN O
. . O

After IN O
the DT O
Trinity NNP O
test NN O
, , O
the DT O
fallout NN B-C
caused VBD O
localized VBN B-C
burns NNS I-C
on IN O
the DT O
backs NNS O
of IN O
cattle NNS O
in IN O
the DT O
area NN O
downwind RB O
. . O

It PRP O
is VBZ O
possible JJ O
that IN O
the DT O
displacement NN B-C
of IN I-C
the DT I-C
katipo NN I-C
by IN I-C
S. NNP I-C
capensis NN I-C
is VBZ O
due IN O
to IN O
its PRP$ O
ability NN B-C
to TO I-C
recolonise VB I-C
areas NNS I-C
from IN O
which WDT O
the DT O
katipo NN O
had VBD O
been VBN O
displaced VBN O
after IN O
storms NNS O
or CC O
other JJ O
dune NN O
modifications NNS O
. . O

Social JJ O
realist JJ O
artist NN O
Noel NNP O
Counihan NNP O
depicted VBD O
men NNS O
who WP O
worked VBD O
in IN O
industrial JJ O
mines NNS O
in IN O
Australia NNP O
in IN O
the DT O
1940s NNS O
dying VBG B-C
of IN O
silicosis NN B-C
in IN O
his PRP$ O
series NN O
of IN O
six CD O
prints NNS O
, , O
' `` O
The DT O
miners NNS O
' '' O
( -LRB- O
1947 CD O
linocuts NNS O
) -RRB- O
. . O

Pulmonary NN O
complications NNS O
of IN O
silicosis NN B-C
also RB O
include VBP O
chronic JJ B-C
bronchitis NN I-C
and CC O
airflow NN B-C
limitation NN I-C
( -LRB- O
indistinguishable JJ O
from IN O
that DT O
caused VBN O
by IN O
smoking NN O
) -RRB- O
, , O
non-tuberculous JJ B-C
Mycobacterium NN I-C
infection NN I-C
, , O
fungal JJ B-C
lung NN I-C
infection NN I-C
, , O
compensatory JJ B-C
emphysema NN I-C
, , O
and CC O
pneumothorax NN B-C
. . O

Tumors NNS B-C
in IN I-C
the DT I-C
head NN I-C
of IN I-C
the DT I-C
pancreas NN I-C
typically RB O
also RB O
cause VBP O
jaundice NN B-C
, , O
pain NN B-C
, , O
loss NN B-C
of IN I-C
appetite NN I-C
, , O
dark JJ B-C
urine NN I-C
, , O
and CC O
light NN B-C
- HYPH I-C
colored VBN I-C
stools NNS I-C
. . O

Ideational JJ B-C
apraxia NN I-C
( -LRB- O
IA NN O
) -RRB- O
is VBZ O
a DT O
neurological JJ O
disorder NN O
which WDT O
explains VBZ O
the DT O
loss NN B-C
of IN I-C
ability NN I-C
to TO I-C
conceptualize VB I-C
, , O
plan NN O
, , O
and CC O
execute VB O
the DT O
complex JJ O
sequences NNS O
of IN O
motor NN O
actions NNS O
involved VBN O
in IN O
the DT O
use NN O
of IN O
tools NNS O
or CC O
otherwise RB O
interacting VBG O
with IN O
objects NNS O
in IN O
everyday JJ O
life NN O
. . O

After IN O
hundreds NNS O
of IN O
deaths NNS B-C
from IN O
silicosis NN B-C
, , O
the DT O
town NN O
was VBD O
nicknamed VBN O
The DT O
Widowmaker NNP O
. . O

In IN O
mice NNS O
, , O
a DT O
naturally RB O
occurring VBG O
null JJ B-C
mutation NN I-C
leads VBZ O
to IN O
the DT O
" `` B-C
lethargic JJ I-C
" '' I-C
phenotype NN I-C
, , O
which WDT O
is VBZ O
similar JJ O
to IN O
JME NNP O
. . O

Injection NN B-C
of IN I-C
fluids NNS I-C
also RB O
increases VBZ O
the DT O
pore NN B-C
pressures NNS I-C
in IN I-C
the DT I-C
reservoir NN I-C
, , O
triggering VBG O
slip NN B-C
on IN I-C
existing VBG I-C
rock NN I-C
weakness NN I-C
planes NNS I-C
. . O

Usually RB O
intentional JJ B-C
movement NN I-C
of IN O
the DT O
involved VBN O
muscle NN O
causes VBZ O
the DT O
fasciculations NNS B-C
to TO O
cease VB O
immediately RB O
, , O
but CC O
they PRP O
may MD O
return VB O
once IN O
the DT O
muscle NN O
is VBZ O
at IN O
rest NN O
again RB O
. . O

Radiation NN B-C
therapy NN I-C
can MD O
also RB O
cause VB O
radiation NN B-C
cancer NN I-C
. . O

If IN O
hunger NN B-C
proceeds VBZ I-C
for IN I-C
an DT I-C
extended VBN I-C
period NN I-C
of IN I-C
time NN I-C
there EX O
is VBZ O
a DT O
good JJ O
chance NN O
that IN O
it PRP O
will MD O
lead VB O
to IN O
malnutrition NN B-C
. . O

HSAN NNP B-C
IC NN I-C
is VBZ O
associated VBN O
with IN O
heterozygous JJ B-C
missense NN I-C
mutations NNS I-C
in IN I-C
the DT I-C
SPTLC2 NN I-C
gene NN I-C
. . O

The DT O
lungs NNS O
normally RB O
" `` O
filter VB O
out RP O
" '' O
bacteria NN O
and CC O
blood NN O
clots NNS O
from IN O
the DT O
bloodstream NN O
; , O
AVMs NNS O
bypass VBP O
the DT O
capillary JJ O
network NN O
of IN O
the DT O
lungs NNS O
and CC O
allow VB O
these DT O
to TO O
migrate VB O
to IN O
the DT O
brain NN O
, , O
where WRB O
bacteria NN B-C
may MD O
cause VB O
a DT O
brain NN B-C
abscess NN I-C
and CC O
blood NN B-C
clots NNS I-C
may MD O
lead VB O
to IN O
stroke NN B-C
. . O

Most JJS O
anticonvulsants NNS B-C
function NN O
by IN O
decreasing VBG B-C
the DT I-C
excitation NN I-C
of IN I-C
neurons NNS I-C
, , O
for IN O
example NN O
, , O
by IN O
blocking VBG B-C
fast JJ I-C
or CC I-C
slow JJ I-C
sodium NN I-C
channels NNS I-C
or CC O
by IN O
modulating VBG B-C
calcium NN I-C
channels NNS I-C
; , O
or CC O
by IN O
enhancing VBG B-C
the DT I-C
inhibition NN I-C
of IN I-C
neurons NNS I-C
, , O
for IN O
example NN O
by IN O
potentiating VBG B-C
the DT I-C
effects NNS I-C
of IN I-C
inhibitory JJ I-C
neurotransmitters NNS I-C
like IN O
GABA NNP O
. . O

Since IN O
a DT O
SCARs NNS B-C
- , O
inducing VBG O
drug NN B-C
or CC O
metabolite NN B-C
interacts VBZ O
with IN O
only RB O
one CD O
or CC O
a DT O
few JJ O
HLA NN O
serotypes NNS O
, , O
their PRP$ O
ability NN O
to TO O
induce VB O
SCARs NNS O
is VBZ O
limited VBN O
to IN O
those DT O
individuals NNS O
who WP O
express VBP O
HLA NN O
serotypes NNS O
targeted VBN O
by IN O
the DT O
drug NN O
or CC O
metabolite NN O
. . O

Both DT O
modeling NN O
and CC O
monitoring NN O
are VBP O
useful JJ O
tools NNS O
to TO O
quantify VB O
, , O
and CC O
thus RB O
better RBR O
understand VB O
and CC O
mitigate VB O
the DT O
risks NNS B-C
associated VBN O
with IN O
injection NN B-C
- HYPH O
induced VBN O
seismicity NN B-C
. . O

Additionally RB O
, , O
thalidomide NN B-C
can MD O
cause VB O
neuropathy NN B-C
. . O

Several JJ B-C
died VBD I-C
of IN O
fourth JJ B-C
degree NN I-C
beta NN I-C
burns NNS I-C
between IN O
9 CD O
– SYM O
28 CD O
days NNS O
after IN O
dose NN O
of IN O
6–16 CD O
Gy NNP O
. . O

Telangiectasias NNP B-C
and CC I-C
arteriovenous JJ I-C
malformations NNS I-C
in IN I-C
HHT NNP I-C
are VBP O
thought VBN O
to TO O
arise VB O
because IN O
of IN O
changes NNS B-C
in IN I-C
angiogenesis NN I-C
, , O
the DT O
development NN O
of IN O
blood NN O
vessels NNS O
out IN O
of IN O
existing VBG O
ones NNS O
. . O

Radiation NN B-C
burns NNS I-C
are VBP O
often RB O
associated VBN O
with IN O
radiation NN B-C
- HYPH O
induced VBN O
cancer NN B-C
due IN O
to IN O
the DT O
ability NN O
of IN O
ionizing NN B-C
radiation NN I-C
to TO O
interact VB O
with IN O
and CC O
damage NN B-C
DNA NN I-C
, , O
occasionally RB O
inducing VBG O
a DT O
cell NN B-C
to TO I-C
become VB I-C
cancerous JJ I-C
. . O

Radiation NN B-C
- HYPH O
induced VBN O
erythema NN B-C
multiforme NN O
may MD O
occur VB O
when WRB O
phenytoin NN B-C
is VBZ I-C
given VBN I-C
prophylactically RB I-C
to IN O
neurosurgical JJ O
patients NNS O
who WP O
are VBP O
receiving VBG O
whole JJ O
- HYPH O
brain NN O
therapy NN O
and CC O
systemic JJ O
steroids NNS O
. . O

A DT O
study NN O
done VBN O
by IN O
Jatturong NNP O
R. NNP O
Wichianson NNP O
and CC O
colleagues NNS O
at IN O
the DT O
University NNP O
of IN O
Southern NNP O
California NNP O
showed VBD O
a DT O
direct JJ O
relationship NN O
between IN O
eating VBG B-C
late RB I-C
at IN I-C
night NN I-C
( -LRB- O
Night NN O
eating NN O
syndrome NN O
) -RRB- O
and CC O
stress NN B-C
levels NNS O
with IN O
college NN O
students NNS O
. . O

Skin NN B-C
infections NNS I-C
, , O
which WDT O
may MD O
lead VB O
to IN O
sepsis NN B-C
, , O
are VBP O
potentially RB O
lethal JJ O
complications NNS O
of IN O
AGEP NN B-C
; , O
preventative JJ O
methods NNS O
and CC O
rapid JJ O
treatment NN O
of IN O
such JJ O
infections NNS O
with IN O
appropriate JJ O
antibiotics NNS O
and CC O
, , O
where WRB O
needed VBN O
, , O
further RB O
supportive JJ O
measures NNS O
are VBP O
critical JJ O
in IN O
the DT O
treatment NN O
of IN O
this DT O
complication NN O
. . O

In IN O
2006 CD O
, , O
Pelucchi NNP O
et FW O
al NNP O
. . O
concluded VBD O
, , O
" `` O
The DT O
silicosis NN B-C
- HYPH O
cancer NN B-C
association NN O
is VBZ O
now RB O
established VBN O
, , O
in IN O
agreement NN O
with IN O
other JJ O
studies NNS O
and CC O
meta-analysis JJ O
. . O
" '' O

It PRP O
has VBZ O
also RB O
been VBN O
suggested VBN O
that IN O
nutritional JJ B-C
factors NNS I-C
may MD O
contribute VB O
to IN O
the DT O
onset NN O
of IN O
dynapenia NN B-C
. . O

Although IN O
aerosol NN B-C
particles NNS I-C
are VBP O
generally RB O
associated VBN O
with IN O
a DT O
global JJ B-C
cooling NN I-C
effect NN I-C
, , O
recent JJ O
studies NNS O
have VBP O
shown VBN O
that IN O
they PRP O
can MD O
actually RB O
have VB O
a DT O
warming NN B-C
effect NN I-C
in IN O
certain JJ O
regions NNS O
such JJ O
as IN O
the DT O
Himalayas NNP O
. . O

According VBG O
to IN O
a DT O
third JJ O
explanation NN O
, , O
shared VBN B-C
stereotypes NNS I-C
are VBP O
neither RB O
caused VBN O
by IN O
the DT O
coincidence NN B-C
of IN I-C
common JJ I-C
stimuli NNS I-C
, , O
nor CC O
by IN O
socialisation NN B-C
. . O

Clinically RB B-C
, , I-C
chronic JJ I-C
radiodermatitis NN I-C
presents VBZ O
as IN O
atrophic JJ B-C
indurated VBN I-C
plaques NNS I-C
, , O
often RB O
whitish JJ O
or CC O
yellowish JJ O
, , O
with IN O
telangiectasia NN O
, , O
sometimes RB O
with IN O
hyperkeratosis NN O
. . O

Those DT O
who WP O
can MD O
not RB O
tolerate VB O
iron NN O
tablets NNS O
or CC O
solutions NNS O
may MD O
require VB O
administration NN O
of IN O
intravenous JJ O
iron NN O
, , O
and CC O
blood NN O
transfusion NN O
if IN O
the DT O
anemia NN B-C
is VBZ O
causing VBG O
severe JJ B-C
symptoms NNS I-C
that WDT O
warrant VBP O
rapid JJ O
improvement NN O
of IN O
the DT O
blood NN O
count NN O
. . O

While IN O
the DT O
high JJ B-C
maternal JJ I-C
mortality NN I-C
rate NN I-C
in IN I-C
Haiti NNP I-C
can MD O
be VB O
attributed VBN O
to IN O
the DT O
fact NN O
that IN O
women NNS B-C
in IN I-C
developing VBG I-C
countries NNS I-C
tend VBP I-C
to TO I-C
have VB I-C
on IN I-C
average JJ I-C
more JJR I-C
pregnancies NNS I-C
than IN O
those DT O
in IN O
developed VBN O
countries NNS O
, , O
factors NNS O
that WDT O
play VBP O
a DT O
much RB O
larger JJR O
role NN O
include VBP O
poverty NN B-C
, , O
distance NN B-C
, , O
and CC O
inadequate JJ B-C
services NNS I-C
, , O
all DT O
of IN O
which WDT O
are VBP O
negative JJ O
impacts NNS O
of IN O
structural JJ B-C
violence NN I-C
. . O

It PRP O
has VBZ O
been VBN O
found VBN O
that IN O
GABA NNP B-C
reversal NN I-C
potential NN I-C
is VBZ I-C
depolarising VBG I-C
in IN I-C
the DT I-C
subpopulation NN I-C
of IN I-C
the DT I-C
pyramidal JJ I-C
cells NNS I-C
due IN O
to IN O
the DT O
lack NN B-C
of IN I-C
KCC2 NNP I-C
co-transporter NN I-C
. . O

Ocular JJ B-C
myasthenia NN I-C
gravis NN I-C
( -LRB- O
MG NNP O
) -RRB- O
is VBZ O
a DT O
disease NN O
of IN O
the DT O
neuromuscular JJ O
junction NN O
resulting VBG O
in IN O
hallmark JJ B-C
variability NN I-C
in IN O
muscle NN B-C
weakness NN I-C
and CC O
fatigability NN B-C
. . O

Ulcerations NNS B-C
are VBP O
also RB O
at IN O
risk NN O
for IN O
bleeding NN B-C
, , O
particularly RB B-C
deep JJ I-C
lesions NNS I-C
or CC O
in IN O
areas NNS O
of IN O
friction NN O
. . O

Protective JJ O
measures NNS O
such JJ O
as IN O
respirators NNS O
have VBP O
brought VBN O
a DT O
steady JJ O
decline NN O
in IN O
death NN B-C
rates NNS O
due IN O
to IN O
silicosis NN B-C
in IN O
Western JJ O
countries NNS O
. . O

The DT O
death NN B-C
of IN I-C
Chinese JJ I-C
Premier NNP I-C
Zhou NNP I-C
Enlai NNP I-C
on IN O
January NNP O
8 CD O
, , O
1976 CD O
, , O
prompted VBD O
the DT O
protest NN B-C
. . O

After IN O
higher JJR B-C
doses NNS I-C
, , O
the DT O
malpighian JJ B-C
layer NN I-C
cells NNS I-C
die VBP I-C
within IN O
24 CD O
hours NNS O
; , O
lower JJR O
doses NNS O
may MD O
take VB O
10 CD O
– , O
14 CD O
days NNS O
to TO O
show VB O
dead JJ O
cells NNS O
. . O

Immune JJ B-C
deficiencies NNS I-C
can MD O
result VB O
in IN O
persistent JJ B-C
or CC I-C
recurring VBG I-C
infections NNS I-C
, , O
autoinflammatory JJ B-C
disorders NNS I-C
, , O
tumors NNS B-C
, , O
and CC O
disorders NNS B-C
of IN I-C
various JJ I-C
organs NNS I-C
. . O

Deep JJ O
and CC O
superficial JJ B-C
vein NN I-C
thrombosis NN I-C
may MD O
in IN O
turn NN O
be VB O
caused VBN O
by IN O
thrombophilia NN B-C
, , O
which WDT O
is VBZ O
an DT O
increased VBN O
propensity NN O
of IN O
forming VBG O
blood NN O
clots NNS O
. . O

Though IN O
understanding NN O
of IN O
reservoir NN B-C
- HYPH O
induced VBN O
seismic JJ B-C
activity NN I-C
is VBZ O
very RB O
limited JJ O
, , O
it PRP O
has VBZ O
been VBN O
noted VBN O
that IN O
seismicity NN O
appears VBZ O
to TO O
occur VB O
on IN O
dams NNS O
with IN O
heights NNS O
larger JJR O
than IN O
. . O

Other JJ O
tests NNS B-C
on IN I-C
the DT I-C
Nevada NNP I-C
Test NNP I-C
Site NNP I-C
also RB O
caused VBD O
fallout NN B-C
and CC O
corresponding VBG O
beta NN B-C
burns NNS I-C
to IN O
sheep NNS O
, , O
horses NNS O
and CC O
cattle NNS O
. . O

HSAN NNP B-C
IB NNP I-C
is VBZ O
linked VBN O
to IN O
a DT O
3.42 CD B-C
cM NN I-C
interval NN I-C
on IN I-C
chromosome NN I-C
3p22–p24 NN I-C
. . O

Some DT O
studies NNS O
, , O
however RB O
, , O
have VBP O
found VBN O
that IN O
this DT O
effect NN O
only RB O
holds VBZ O
when WRB O
stereotyped VBN O
individuals NNS O
can MD O
be VB O
absolutely RB O
certain JJ O
that IN O
their PRP$ O
negative JJ B-C
outcomes NNS I-C
are VBP O
due IN O
to IN O
the DT O
evaluators NNS B-C
's POS I-C
prejudice NN I-C
. . O

In IN O
dental JJ O
enamel NN O
, , O
fluorosis NN B-C
causes VBZ O
subsurface JJ B-C
porosity NN I-C
or CC O
hypomineralizations NNS B-C
, , O
which WDT O
extend VBP O
toward IN O
the DT O
dentinal JJ O
- HYPH O
enamel NN O
junction NN O
as IN O
severity NN O
increases VBZ O
. . O

Eosinophilic JJ B-C
, , I-C
polymorphic JJ I-C
, , I-C
and CC I-C
pruritic JJ I-C
eruption NN I-C
associated VBN O
with IN O
radiotherapy NN B-C
is VBZ O
a DT O
skin NN O
condition NN O
that WDT O
occurs VBZ O
most RBS O
often RB O
in IN O
women NNS O
receiving VBG O
cobalt NN O
radiotherapy NN O
for IN O
internal JJ O
cancer NN O
. . O

Overall RB O
, , O
IA NNP B-C
is VBZ O
an DT O
autonomous JJ O
syndrome NN O
, , O
linked VBN O
to IN O
damage NN B-C
in IN I-C
the DT I-C
left JJ I-C
hemisphere NN I-C
involving VBG O
semantic JJ B-C
memory NN I-C
disorders NNS I-C
rather RB O
than IN O
a DT O
defect NN O
in IN O
motor NN O
control NN O
. . O

Even RB O
workers NNS O
with IN O
prolonged VBN B-C
silica NN I-C
exposure NN I-C
, , O
but CC O
without IN O
silicosis NN O
, , O
are VBP O
at IN O
a DT O
similarly RB O
increased VBN O
risk NN O
for IN O
TB NNP B-C
. . O

These DT O
deficiencies NNS B-C
can MD O
lead VB O
to IN O
weight NN B-C
issues NNS I-C
caused VBN O
by IN O
malnutrition NN B-C
. . O

In IN O
1961 CD O
, , O
waste NN B-C
water NN I-C
was VBD I-C
injected VBN I-C
into IN I-C
deep JJ I-C
strata NNS I-C
, , O
and CC O
this DT O
was VBD O
later RB O
found VBN O
to TO O
have VB O
caused VBN O
a DT O
series NN B-C
of IN I-C
earthquakes NNS I-C
. . O

Prednisone NNP B-C
is VBZ O
the DT O
most RBS O
commonly RB O
prescribed VBN O
systemic JJ O
treatment NN O
but CC O
can MD O
cause VB O
serious JJ B-C
adrenal JJ I-C
suppression NN I-C
, , O
so RB O
it PRP O
must MD O
be VB O
taken VBN O
carefully RB O
and CC O
tapered VBN O
off RP O
slowly RB O
. . O

Treatment NN O
of IN O
thrombotic JJ B-C
thrombocytopenic JJ I-C
purpura NN I-C
( -LRB- O
TTP NNP O
) -RRB- O
is VBZ O
a DT O
medical JJ O
emergency NN O
, , O
since IN O
the DT O
associated VBN O
hemolytic JJ B-C
anemia NN I-C
and CC O
platelet NN B-C
activation NN I-C
can MD O
lead VB O
to IN O
renal JJ B-C
failure NN I-C
and CC O
changes NNS B-C
in IN I-C
the DT I-C
level NN I-C
of IN I-C
consciousness NN I-C
. . O

Australian JJ O
physicians NNPS O
Fowler NNP O
and CC O
Carter NNP O
first RB O
described VBD O
human JJ B-C
disease NN I-C
caused VBN O
by IN O
amebo-flagellates NNS B-C
in IN O
Adelaide NNP O
in IN O
1965 CD O
. . O

Apart RB O
from IN O
MADH4 NNP O
, , O
it PRP O
is VBZ O
not RB O
clear JJ O
whether IN O
mutations NNS O
in IN O
ENG NNP O
and CC O
ACVRL1 NNP O
lead VBP O
to IN O
particular JJ O
symptoms NNS O
, , O
although IN O
some DT O
reports NNS O
suggest VBP O
that IN O
ENG NN B-C
mutations NNS I-C
are VBP O
more RBR O
likely JJ O
to TO O
cause VB O
lung NN B-C
problems NNS I-C
while IN O
ACVRL1 NNP B-C
mutations NNS I-C
may MD O
cause VB O
more JJR O
liver NN B-C
problems NNS I-C
, , O
and CC O
pulmonary JJ O
hypertension NN O
may MD O
be VB O
a DT O
particular JJ O
problem NN O
in IN O
people NNS O
with IN O
ACVRL1 NNP O
mutations NNS O
. . O

It PRP O
is VBZ O
thought VBN O
to TO O
do VB O
so RB O
by IN O
causing VBG O
oxidative NN B-C
damage NN I-C
to IN I-C
liver NN I-C
cells NNS I-C
, , O
resulting VBG O
in IN O
oxidized VBN B-C
species NNS I-C
of IN I-C
uroporphyrinogen NN I-C
that WDT O
inhibit VBP O
the DT O
activity NN O
of IN O
hepatic JJ O
UROD NN O
. . O

Hereditary JJ B-C
hemorrhagic JJ I-C
telangiectasia NN I-C
( -LRB- O
HHT NNP O
) -RRB- O
, , O
also RB O
known VBN O
as IN O
Osler NNP O
– , O
Weber NNP O
– , O
Rendu NNP O
disease NNP O
and CC O
Osler NNP O
– , O
Weber NNP O
– , O
Rendu NNP O
syndrome NN O
, , O
is VBZ O
a DT O
rare JJ O
autosomal JJ O
dominant JJ O
genetic JJ O
disorder NN O
that WDT O
leads VBZ O
to IN O
abnormal JJ B-C
blood NN I-C
vessel NN I-C
formation NN I-C
in IN O
the DT O
skin NN O
, , O
mucous NN O
membranes NNS O
, , O
and CC O
often RB O
in IN O
organs NNS O
such JJ O
as IN O
the DT O
lungs NNS O
, , O
liver NN O
, , O
and CC O
brain NN O
. . O

Erythema NNP B-C
may MD O
occur VB O
at IN O
a DT O
dose NN B-C
of IN I-C
2 CD I-C
Gy NN I-C
radiation NN I-C
or CC O
greater JJR O
. . O

One CD B-C
died VBD I-C
later RB O
from IN O
second JJ B-C
degree NN I-C
beta NN I-C
burns NNS I-C
and CC O
dose NN O
1 CD O
- SYM O
4 CD O
Gy NN O
. . O

Concentrated VBN O
instances NNS O
of IN O
oxidative NN B-C
stress NN I-C
( -LRB- O
alcohol NN O
, , O
physical JJ O
trauma NN O
, , O
psychological JJ O
stress NN O
, , O
etc FW O
) -RRB- O
cause VBP O
the DT O
liver NN B-C
to TO I-C
hemorrhage VB I-C
these DT I-C
porphyrins NNS I-C
into IN I-C
the DT I-C
blood NN I-C
stream NN I-C
where WRB O
they PRP O
are VBP O
then RB O
susceptible JJ O
to IN O
oxidation NN O
. . O

Enlargement NN B-C
of IN I-C
the DT I-C
hilum NN I-C
is VBZ O
common JJ O
with IN O
chronic NN O
and CC O
accelerated VBN O
silicosis NN B-C
. . O

Exercise NN B-C
can MD O
trigger VB O
hives NNS B-C
and CC O
more RBR O
severe JJ O
symptoms NNS B-C
of IN I-C
an DT I-C
allergic JJ I-C
reaction NN I-C
. . O

The DT O
SLC2A9 NN B-C
, , I-C
SLC22A12 NN I-C
, , I-C
and CC I-C
ABCG2 NN I-C
genes NNS I-C
have VBP O
been VBN O
found VBN O
to TO O
be VB O
commonly RB O
associated VBN O
with IN O
gout NN B-C
and CC O
variations NNS O
in IN O
them PRP O
can MD O
approximately RB O
double VB O
the DT O
risk NN O
. . O

In IN O
the DT O
intergroup JJ O
context NN O
, , O
illusory JJ B-C
correlations NNS I-C
lead VBP O
people NNS O
to TO O
misattribute VB B-C
rare JJ I-C
behaviors NNS I-C
or CC I-C
traits NNS I-C
at IN I-C
higher JJR I-C
rates NNS I-C
to IN I-C
minority JJ I-C
group NN I-C
members NNS I-C
than IN O
to IN O
majority NN O
groups NNS O
, , O
even RB O
when WRB O
both DT O
display VBP O
the DT O
same JJ O
proportion NN O
of IN O
the DT O
behaviors NNS O
or CC O
traits NNS O
. . O

It PRP O
is VBZ O
important JJ O
to TO O
note VB O
from IN O
this DT O
explanation NN O
that IN O
stereotypes NNS B-C
are VBP O
the DT O
consequence NN O
, , O
not RB O
the DT O
cause NN O
, , O
of IN O
intergroup JJ B-C
relations NNS I-C
. . O

A DT O
meta-analysis NN O
published VBD O
the DT O
same JJ O
year NN O
supported VBD O
the DT O
theory NN O
that IN O
early JJ O
introduction NN O
of IN O
eggs NNS O
into IN O
an DT O
infant NN O
's POS O
diet NN O
lowers VBZ O
risk NN O
, , O
and CC O
a DT O
review NN O
of IN O
allergens NNS O
in IN O
general JJ O
stated VBD O
that IN O
introducing VBG B-C
solid JJ I-C
foods NNS I-C
at IN I-C
4 CD I-C
– SYM I-C
6 CD I-C
months NNS I-C
may MD O
result VB O
in IN O
the DT O
lowest JJS B-C
subsequent JJ I-C
allergy NN I-C
risk NN I-C
. . O

Several JJ O
tests NNS O
have VBP O
been VBN O
proposed VBN O
to TO O
be VB O
useful JJ O
for IN O
supporting VBG O
the DT O
diagnosis NN O
of IN O
and CC O
/ , O
or CC O
implicating VBG O
a DT O
particular JJ B-C
drug NN I-C
as IN O
the DT O
cause NN O
of IN O
AGEP NNP B-C
particularly RB O
in IN O
individuals NNS O
who WP O
develop VBP O
skin NN O
lesions NNS O
while IN O
taking VBG O
multiple JJ O
drugs NNS O
. . O

Two CD O
of IN O
the DT O
reviews NNS O
postulate VBP O
that IN O
exercise NN O
is VBZ O
not RB O
essential JJ O
for IN O
the DT O
development NN O
of IN O
symptoms NNS O
, , O
but CC O
rather RB O
that IN O
it PRP O
is VBZ O
one CD O
of IN O
several JJ O
augmentation NN O
factors NNS O
, , O
citing VBG O
evidence NN O
that IN O
the DT O
culprit NN O
food NN B-C
in IN I-C
combination NN I-C
with IN I-C
alcohol NN I-C
or CC I-C
aspirin NN I-C
will MD O
result VB O
in IN O
a DT O
respiratory JJ B-C
anaphylactic JJ I-C
reaction NN I-C
. . O

Stereotypes NNS B-C
can MD O
affect VB O
self NN O
- HYPH O
evaluations NNS O
and CC O
lead NN O
to IN O
self NN B-C
- HYPH I-C
stereotyping NN I-C
. . O

Racial JJ B-C
or CC I-C
ethnic JJ I-C
differentiation NN I-C
has VBZ O
been VBN O
well RB O
acknowledged VBN O
as IN O
a DT O
means NN O
of IN O
depriving VBG B-C
certain JJ I-C
racial JJ I-C
or CC I-C
ethnic JJ I-C
groups NNS I-C
of IN I-C
basic JJ I-C
human JJ I-C
rights NNS I-C
, , O
or CC O
from IN O
receiving VBG B-C
the DT I-C
same JJ I-C
quality NN I-C
of IN I-C
resources NNS I-C
as IN O
others NNS O
. . O

Due IN O
to IN O
the DT O
oppressive JJ B-C
political JJ I-C
and CC O
economic JJ B-C
structures NNS I-C
that WDT O
are VBP O
plaguing VBG O
the DT O
nation NN O
, , O
the DT O
educational JJ B-C
sector NN I-C
in IN I-C
Haiti NNP I-C
has VBZ I-C
limited VBN I-C
financial JJ I-C
support NN I-C
. . O

Additionally RB O
, , O
the DT O
economic JJ B-C
slump NN I-C
in IN O
Japan NNP O
since IN O
1990 CD O
has VBZ O
been VBN O
cited VBN O
as IN O
contributing VBG O
to IN O
the DT O
increase NN B-C
in IN I-C
lonely JJ I-C
deaths NNS I-C
. . O

While IN O
primarily RB O
caused VBN O
through IN O
natural JJ B-C
wear NN I-C
and CC I-C
tear NN I-C
, , O
advanced JJ B-C
facet NN I-C
syndrome NN I-C
can MD O
also RB O
occur VB O
as IN O
a DT O
result NN O
of IN O
injury NN B-C
to IN I-C
the DT I-C
spine NN I-C
, , O
degenerative JJ B-C
disease NN I-C
or CC O
lifestyle NN B-C
choices NNS I-C
. . O

This DT O
stimulation NN O
is VBZ O
decreased VBN O
by IN O
JME NNP B-C
associated VBN O
mutations NNS B-C
. . O

Such JJ O
donor JJ B-C
T-cells NNS I-C
often RB O
cause VBP O
acute JJ B-C
graft NN I-C
- HYPH I-C
versus NN I-C
- HYPH I-C
host NN I-C
disease NN I-C
( -LRB- O
GVHD NN O
) -RRB- O
, , O
a DT O
subject NN O
of IN O
ongoing JJ O
investigation NN O
. . O

The DT O
most RBS O
common JJ O
cause NN O
of IN O
melanosis NN B-C
coli NN I-C
is VBZ O
the DT O
extended VBN B-C
use NN I-C
of IN I-C
laxatives NNS I-C
, , O
and CC O
commonly JJ B-C
anthraquinone NN I-C
containing VBG I-C
laxatives NNS I-C
such JJ O
as IN O
senna NN B-C
, , O
aloe NN B-C
vera NN I-C
and CC O
other JJ O
plant NN B-C
glycosides NNS I-C
. . O

Fluorosis NN B-C
does VBZ O
not RB O
cause VB O
discolouration NN B-C
to IN I-C
the DT I-C
enamel NN I-C
directly RB O
, , O
as IN O
upon IN O
eruption NN O
into IN O
the DT O
mouth NN O
, , O
affected VBN O
permanent JJ O
teeth NNS O
are VBP O
not RB O
discoloured VBN O
yet RB O
. . O

In IN O
March NNP O
2016 CD O
, , O
OSHA NNP O
officially RB O
mandated VBD O
that IN O
companies NNS O
must MD O
provide VB O
certain JJ O
safety NN O
measures NNS O
for IN O
employees NNS O
who WP O
work VBP O
with IN O
or CC O
around IN O
silica NN B-C
, , O
in IN O
order NN O
to TO O
prevent VB O
silicosis NN B-C
, , O
lung NN B-C
cancer NN I-C
, , O
and CC O
other JJ O
silica NN B-C
- HYPH I-C
related JJ I-C
diseases NNS I-C
. . O

Second JJ B-C
degree NN I-C
beta NN I-C
burns NNS I-C
lead VBP O
to IN O
formation NN B-C
of IN I-C
blisters NNS I-C
. . O

Because IN O
the DT O
terrain NN B-C
rises VBZ I-C
up RP I-C
to IN I-C
the DT I-C
runway NN I-C
, , O
this DT O
could MD O
have VB O
had VBN O
the DT O
effect NN O
of IN O
causing VBG O
the DT O
pilot NN B-C
to TO I-C
fly VB I-C
far RB I-C
too RB I-C
low RB I-C
. . O

This DT O
is VBZ O
confirmed VBN O
by IN O
a DT O
report NN O
stating VBG O
that IN O
during IN O
the DT O
period NN O
of IN O
2001 CD O
to IN O
2005 CD O
; , O
about RB O
50 CD O
% NN O
of IN O
deaths NNS B-C
in IN I-C
wild JJ I-C
giant NN I-C
pandas NNS I-C
were VBD O
caused VBN O
by IN O
the DT O
parasite NN B-C
infection NN I-C
. . O

The DT O
incident NN B-C
occurred VBD O
on IN O
the DT O
traditional JJ O
day NN O
of IN O
mourning NN O
, , O
the DT O
Qingming NNP O
Festival NNP O
, , O
after IN O
the DT O
Nanjing NNP O
Incident NNP O
, , O
and CC O
was VBD O
triggered VBN O
by IN O
the DT O
death NN B-C
of IN I-C
Premier NNP I-C
Zhou NNP I-C
Enlai NNP I-C
earlier RB O
that DT O
year NN O
. . O

Overall RB O
, , O
the DT O
disaster NN B-C
resulted VBD O
in IN O
the DT O
death NN B-C
of IN I-C
twelve CD I-C
people NNS I-C
, , O
one CD O
million CD O
sheep NNS O
, , O
and CC O
thousands NNS O
of IN O
cattle NNS O
over RB O
. . O

The DT O
remnants NNS O
of IN O
these DT O
social JJ O
structures NNS O
exist VBP O
today NN O
in IN O
the DT O
form NN O
of IN O
caste NN B-C
- HYPH I-C
based VBN I-C
racial JJ I-C
discrimination NN I-C
, , O
which WDT O
contributes VBZ O
to IN O
the DT O
social JJ B-C
inequality NN I-C
and CC O
economic JJ B-C
disparities NNS I-C
present VBP O
within IN O
Haiti NNP O
. . O

As IN O
small JJ B-C
numbers NNS I-C
of IN I-C
larvae NNS I-C
can MD O
cause VB O
severe JJ B-C
disease NN I-C
, , O
and CC O
larvae NNS O
occur VBP O
randomly RB O
in IN O
tissue NN O
, , O
a DT O
biopsy NN B-C
usually RB I-C
fails VBZ I-C
to TO I-C
include VB I-C
larvae NNS I-C
and CC O
therefore RB O
leads VBZ O
to IN O
negative JJ B-C
results NNS I-C
. . O

Pancreatic JJ B-C
cancer NN I-C
has VBZ O
been VBN O
associated VBN O
with IN O
the DT O
following VBG O
other JJ O
rare JJ O
hereditary JJ O
syndromes NNS O
: : O
Peutz NNP B-C
– , I-C
Jeghers NNP I-C
syndrome NN I-C
due IN O
to IN O
mutations NNS B-C
in IN I-C
the DT I-C
STK11 NN I-C
tumor NN I-C
suppressor NN I-C
gene NN I-C
( -LRB- O
very RB O
rare JJ O
, , O
but CC O
a DT O
very RB O
strong JJ O
risk NN O
factor NN O
) -RRB- O
; , O
dysplastic JJ B-C
nevus JJ I-C
syndrome NN I-C
( -LRB- O
or CC O
familial JJ O
atypical JJ O
multiple JJ O
mole NN O
and CC O
melanoma NN O
syndrome NN O
, , O
FAMMM NN O
- HYPH O
PC NN O
) -RRB- O
due IN O
to IN O
mutations NNS B-C
in IN I-C
the DT I-C
CDKN2A NN I-C
tumor NN I-C
suppressor NN I-C
gene NN I-C
; , O
autosomal JJ B-C
recessive JJ I-C
ataxia- NN I-C
telangiectasia NN I-C
and CC O
autosomal JJ B-C
dominantly RB I-C
inherited VBN I-C
mutations NNS I-C
in IN I-C
the DT I-C
BRCA2 NN I-C
gene NN I-C
and CC I-C
PALB2 NN I-C
gene NN I-C
; , O
hereditary JJ B-C
non-polyposis JJ I-C
colon NN I-C
cancer NN I-C
( -LRB- O
Lynch NN O
syndrome NN O
) -RRB- O
; , O
and CC O
familial JJ B-C
adenomatous JJ I-C
polyposis NN I-C
. . O

Certain JJ O
mutations NNS B-C
in IN I-C
the DT I-C
HFE NN I-C
gene NN I-C
cause VBP O
hemochromatosis NN B-C
( -LRB- O
an DT O
iron NN O
overload NN O
disorder NN O
) -RRB- O
. . O

The DT O
purported VBN O
link NN O
between IN O
TLE NN B-C
and CC O
religiosity NN B-C
has VBZ O
inspired VBN O
work NN O
by IN O
Michael NNP O
Persinger NNP O
and CC O
many JJ O
other JJ O
researchers NNS O
in IN O
the DT O
field NN O
of IN O
neurotheology NN O
, , O
but CC O
some DT O
have VBP O
questioned VBN O
the DT O
evidence NN O
for IN O
a DT O
link NN O
between IN O
temporal JJ B-C
lobe NN I-C
epilepsy NN I-C
and CC O
religiosity NN B-C
. . O

This DT O
uneven JJ B-C
distribution NN I-C
of IN I-C
resources NNS I-C
across IN O
Haiti NNP O
's POS O
society NN O
has VBZ O
created VBN O
numerous JJ O
social JJ B-C
and CC I-C
economic JJ I-C
disparities NNS I-C
, , O
all DT O
of IN O
which WDT O
have VBP O
led VBN O
the DT O
country NN B-C
to TO I-C
perform VB I-C
below IN I-C
other JJ I-C
countries NNS I-C
in IN I-C
a DT I-C
series NN I-C
of IN I-C
health NN I-C
outcomes NNS I-C
. . O

A DT O
computed VBN O
tomography NN O
or CC O
CT NNP O
scan NN O
can MD O
also RB O
provide VB O
a DT O
mode NN O
detailed JJ O
analysis NN O
of IN O
the DT O
lungs NNS O
, , O
and CC O
can MD O
reveal VB O
cavitation NN B-C
due IN O
to IN O
concomitant JJ B-C
mycobacterial JJ I-C
infection NN I-C
. . O

Characteristic JJ O
lung NN O
tissue NN O
pathology NN O
in IN O
nodular JJ B-C
silicosis NN I-C
consists VBZ O
of IN O
fibrotic JJ B-C
nodules NNS I-C
with IN I-C
concentric JJ I-C
" `` I-C
onion NN I-C
- HYPH I-C
skinned VBN I-C
" '' I-C
arrangement NN I-C
of IN I-C
collagen NN I-C
fibers NNS I-C
, , O
central JJ B-C
hyalinization NN I-C
, , O
and CC O
a DT O
cellular JJ B-C
peripheral JJ I-C
zone NN I-C
, , O
with IN O
lightly RB O
birefringent VBG O
particles NNS O
seen VBN O
under IN O
polarized VBN O
light NN O
. . O

Agricola NNP O
, , O
in IN O
the DT O
mid-16th JJ O
century NN O
, , O
wrote VBD O
about IN O
lung NN B-C
problems NNS I-C
from IN O
dust NN B-C
inhalation NN I-C
in IN O
miners NNS O
. . O

The DT O
best JJS O
way NN O
to TO O
prevent VB O
silicosis NN B-C
is VBZ O
to TO O
identify VB O
work NN O
- HYPH O
place NN O
activities NNS O
that WDT O
produce VBP O
respirable JJ B-C
crystalline JJ I-C
silica NN I-C
dust NN I-C
and CC O
then RB O
to TO O
eliminate VB O
or CC O
control VB O
the DT O
dust NN O
( -LRB- O
" `` O
primary JJ O
prevention NN O
" '' O
) -RRB- O
. . O

Due IN O
to IN O
the DT O
inability NN B-C
to TO I-C
receive VB I-C
proper JJ I-C
education NN I-C
and CC I-C
maintain VB I-C
good JJ I-C
health NN I-C
, , O
individuals NNS B-C
impacted VBN I-C
by IN I-C
structural JJ I-C
violence NN I-C
often RB I-C
are VBP I-C
less RBR I-C
well RB I-C
- HYPH I-C
positioned VBN I-C
to TO O
attain VB O
better JJR O
socio-economic JJ O
opportunities NNS O
or CC O
to TO O
be VB O
upwardly RB O
mobile JJ O
. . O

Diuretics NNS B-C
have VBP O
been VBN O
associated VBN O
with IN O
attacks NNS O
of IN O
gout NN B-C
, , O
but CC O
a DT O
low JJ B-C
dose NN I-C
of IN I-C
hydrochlorothiazide NN I-C
does VBZ O
not RB O
seem VB O
to TO O
increase VB O
risk NN O
. . O

Inflamed JJ B-C
facets NNS I-C
can MD O
cause VB O
a DT O
powerful JJ B-C
muscle NN I-C
spasm NN I-C
. . O

The DT O
study NN O
also RB O
found VBD O
that IN O
the DT O
decrease NN B-C
in IN I-C
muscle NN I-C
strength NN I-C
due IN O
to IN O
aging NN B-C
was VBD O
2 CD O
- SYM O
5 CD O
times NNS O
greater JJR O
than IN O
the DT O
loss NN O
of IN O
muscle NN O
size NN O
. . O

Research NN O
on IN O
the DT O
role NN O
of IN O
illusory JJ O
correlations NNS O
in IN O
the DT O
formation NN O
of IN O
stereotypes NNS O
suggests VBZ O
that IN O
stereotypes NNS B-C
can MD O
develop VB O
because IN O
of IN O
incorrect JJ B-C
inferences NNS I-C
about IN I-C
the DT I-C
relationship NN I-C
between IN I-C
two CD I-C
events NNS I-C
( -LRB- O
e.g. FW O
, , O
membership NN O
in IN O
a DT O
social JJ O
group NN O
and CC O
bad JJ O
or CC O
good JJ O
attributes NNS O
) -RRB- O
. . O

An DT O
article NN O
in IN O
Science NNP O
suggested VBD O
that IN O
the DT O
construction NN B-C
and CC I-C
filling NN I-C
of IN I-C
the DT I-C
Zipingpu NNP I-C
Dam NNP I-C
may MD O
have VB O
triggered VBN O
the DT O
earthquake NN B-C
. . O

Discrediting VBG O
earlier JJ O
notions NNS O
of IN O
presbycusis NN O
, , O
Rosen NNP O
demonstrated VBD O
that IN O
long JJ B-C
term NN I-C
hearing NN I-C
loss NN I-C
is VBZ O
usually RB O
the DT O
product NN O
of IN O
chronic JJ B-C
exposure NN I-C
to IN I-C
environmental JJ I-C
noise NN I-C
in IN I-C
industrialized VBN I-C
countries NNS I-C
( -LRB- O
Rosen NNP O
, , O
1965 CD O
) -RRB- O
. . O

Dental JJ O
fluorosis NN O
( -LRB- O
also RB O
termed VBN O
mottled VBN O
enamel NN O
) -RRB- O
is VBZ O
an DT O
extremely RB O
common JJ O
disorder NN O
, , O
characterized VBN O
by IN O
hypomineralization NN B-C
of IN I-C
tooth NN I-C
enamel NN I-C
caused VBN O
by IN O
ingestion NN B-C
of IN I-C
excessive JJ I-C
fluoride NN I-C
during IN I-C
enamel NN I-C
formation NN I-C
. . O

A DT O
lack NN B-C
of IN I-C
retinol NN I-C
- HYPH I-C
binding NN I-C
protein NN I-C
is VBZ O
due IN O
to IN O
a DT O
lack NN B-C
of IN I-C
retinol NN I-C
which WDT O
is VBZ O
required VBN O
to TO O
trigger VB O
its PRP$ O
production NN O
. . O

The DT O
most RBS O
frequently RB O
reported VBN O
drugs NNS O
that WDT O
have VBP O
been VBN O
associated VBN O
with IN O
the DT O
development NN O
of IN O
AGEP NNP B-C
include VBP O
penicillin NN B-C
, , O
aminopenicillins NNS B-C
, , O
macrolides NNS B-C
, , O
quinolones NNS B-C
, , O
sulfonamides NNS B-C
, , O
hydroxychloroquine NN B-C
, , O
terbinafine NN B-C
, , O
and CC O
diltiazem NN B-C
. . O

Deep JJ B-C
hemangiomas NNS I-C
near IN I-C
sensitive JJ I-C
structures NNS I-C
can MD O
cause VB O
compression NN B-C
of IN I-C
softer JJR I-C
surrounding VBG I-C
structures NNS I-C
during IN O
the DT O
proliferative JJ O
phase NN O
, , O
such JJ O
as IN O
the DT O
external JJ O
ear NN O
canal NN O
and CC O
the DT O
eyelid NN O
. . O

Other JJ O
causes NNS O
of IN O
non-cyclical JJ B-C
breast NN I-C
pain NN I-C
include VBP O
alcoholism NN B-C
with IN I-C
liver NN I-C
damage NN I-C
( -LRB- O
likely RB O
due IN O
to IN O
abnormal JJ O
steroid NN O
metabolism NN O
) -RRB- O
, , O
mastitis NN B-C
and CC O
medications NNS B-C
such JJ O
as IN O
digitalis NN B-C
, , O
methyldopa NN B-C
( -LRB- O
an DT O
antihypertensive NN O
) -RRB- O
, , O
spironolactone NN B-C
, , O
certain JJ O
diuretics NNS B-C
, , O
oxymetholone NN B-C
( -LRB- O
an DT O
anabolic JJ O
steroid NN O
) -RRB- O
, , O
and CC O
chlorpromazine NN B-C
( -LRB- O
a DT O
typical JJ O
antipsychotic NN O
) -RRB- O
. . O

Hunger NNP B-C
is VBZ O
a DT O
main JJ O
cause NN O
of IN O
malnutrition NN B-C
because IN O
if IN O
it PRP O
is VBZ O
not RB O
satisfied JJ O
then RB O
malnutrition NN O
is VBZ O
sure JJ O
to TO O
follow VB O
. . O

Extensive JJ B-C
tophi NN I-C
may MD O
lead VB O
to IN O
chronic JJ B-C
arthritis NN I-C
due IN O
to IN O
bone NN B-C
erosion NN I-C
. . O

" `` O
Earthquake NN O
rates NNS O
have VBP O
recently RB O
increased VBN O
markedly RB O
in IN O
multiple JJ O
areas NNS O
of IN O
the DT O
Central NNP O
and CC O
Eastern NNP O
United NNP O
States NNP O
( -LRB- O
CEUS NNP O
) -RRB- O
, , O
especially RB O
since IN O
2010 CD O
, , O
and CC O
scientific JJ O
studies NNS O
have VBP O
linked VBN O
the DT O
majority NN O
of IN O
this DT O
increased VBN B-C
activity NN I-C
to IN O
wastewater NN B-C
injection NN I-C
in IN I-C
deep JJ I-C
disposal NN I-C
wells NNS I-C
. . O
" '' O

Political JJ B-C
instability NN I-C
and CC O
corruption NN B-C
have VBP O
left VBN O
the DT O
country NN O
with IN O
a DT O
number NN O
of IN O
social JJ B-C
problems NNS I-C
, , O
such JJ O
as IN O
a DT O
lack NN B-C
of IN I-C
access NN I-C
to IN I-C
basic JJ I-C
human JJ I-C
rights NNS I-C
, , O
including VBG O
education NN O
and CC O
healthcare NN O
; , O
and CC O
an DT O
economy NN B-C
with IN I-C
a DT I-C
lower JJR I-C
than IN I-C
average JJ I-C
domestic JJ I-C
product NN I-C
. . O

In IN O
cases NNS O
where WRB O
fasciculations NNS B-C
are VBP O
caused VBN O
by IN O
magnesium NN B-C
deficiency NN I-C
, , O
supplementing VBG O
magnesium NN O
can MD O
be VB O
effective JJ O
in IN O
reducing VBG O
symptoms NNS O
. . O

These DT O
experiences NNS O
are VBP O
described VBN O
as IN O
complex JJ O
interactions NNS O
with IN O
their PRP$ O
visions NNS O
, , O
but CC O
lacking VBG O
the DT O
stereotypy NN B-C
, , O
amnestic JJ B-C
periods NNS I-C
, , O
and CC O
automatisms NNS B-C
or CC I-C
generalized VBN I-C
motor NN I-C
events NNS I-C
, , O
which WDT O
are VBP O
characteristic JJ O
of IN O
TLE NN B-C
. . O

Studies NNS O
have VBP O
identified VBN O
several JJ O
HLA NN B-C
serotypes NNS I-C
associated VBN O
with IN O
development NN O
of IN O
DRESS NN B-C
syndrome NN I-C
, , O
SJS NN B-C
, , O
SJS NN B-C
/ , I-C
TEN CD I-C
, , O
and CC O
TEN CD B-C
in IN O
response NN O
to IN O
various JJ O
drugs NNS O
which WDT O
elici VBP O
these DT O
disorders NNS O
, , O
developed VBN O
tests NNS O
to TO O
identify VB O
individuals NNS O
who WP O
express VBP O
these DT O
serotypes NNS O
, , O
and CC O
there RB O
by IN O
determined JJ O
that IN O
these DT O
individuals NNS O
should MD O
avoid VB O
the DT O
offending VBG O
drug NN O
. . O

Mutation NN B-C
in IN I-C
only RB I-C
a DT I-C
single JJ I-C
gene NN I-C
can MD O
lead VB O
to IN O
the DT O
evolution NN B-C
of IN I-C
a DT I-C
resistant JJ I-C
organism NN I-C
. . O

Excessive JJ B-C
scratching NN I-C
may MD O
result VB O
in IN O
infection NN B-C
, , O
commonly RB O
by IN O
staphylococcal JJ O
and CC O
streptococcal JJ O
species NNS O
; , O
these DT O
may MD O
require VB O
antibiotics NNS O
. . O

Recent JJ O
professional JJ O
guidelines NNS O
recommend VBP O
that IN O
all DT O
children NNS O
with IN O
suspected VBN O
or CC O
definite JJ O
HHT NN O
undergo VBP O
a DT O
brain NN O
MRI NN O
early RB O
in IN O
life NN O
to TO O
identify VB O
AVMs NNS B-C
that WDT O
can MD O
cause VB O
major JJ B-C
complications NNS I-C
. . O

Exposure NN B-C
to IN I-C
Thorotrast NNP I-C
, , O
a DT O
form NN O
of IN O
thorium NN O
dioxide NN O
which WDT O
was VBD O
used VBN O
as IN O
a DT O
radiologic JJ O
contrast NN O
medium NN O
, , O
has VBZ O
been VBN O
linked VBN O
to IN O
the DT O
development NN O
of IN O
cholangiocarcinoma NN B-C
as RB O
late RB O
as IN O
30 CD O
– SYM O
40 CD O
years NNS O
after IN O
exposure NN O
; , O
Thorotrast NNP B-C
was VBD I-C
banned VBN I-C
in IN I-C
the DT I-C
United NNP I-C
States NNP I-C
in IN I-C
the DT I-C
1950s NNS I-C
due IN O
to IN O
its PRP$ O
carcinogenicity NN B-C
. . O

Although IN O
simple JJ O
skin NN O
exposure NN O
is VBZ O
most RBS O
common JJ O
, , O
ingestion NN B-C
of IN I-C
urushiol NN I-C
can MD O
lead VB O
to IN O
serious JJ B-C
, , I-C
systemic JJ I-C
reactions NNS I-C
. . O

Both CC O
Alk NNP O
- HYPH O
1 CD O
and CC O
endoglin NN O
are VBP O
expressed VBN O
predominantly RB O
in IN O
endothelium NN O
, , O
perhaps RB O
explaining VBG O
why WRB O
HHT NNP B-C
- HYPH I-C
causing VBG I-C
mutations NNS I-C
in IN O
these DT O
proteins NNS O
lead VBP O
predominantly RB O
to IN O
blood NN B-C
vessel NN I-C
problems NNS I-C
. . O

Individuals NNS O
with IN O
3 CD B-C
- HYPH I-C
hydroxyacyl NN I-C
- HYPH I-C
coenzyme NN I-C
A DT I-C
dehydrogenase NN I-C
deficiency NN I-C
are VBP O
also RB O
at IN O
risk NN O
for IN O
complications NNS O
such JJ O
as IN O
seizures NNS B-C
, , O
life NN B-C
- HYPH I-C
threatening VBG I-C
heart NN I-C
and CC I-C
breathing NN I-C
problems NNS I-C
, , O
coma NN B-C
, , O
and CC O
sudden JJ B-C
unexpected JJ I-C
death NN I-C
. . O

Deep JJ B-C
hemangiomas NNS I-C
rarely RB O
ulcerate VBP O
, , O
but CC O
can MD O
cause VB O
issues NNS B-C
depending VBG O
on IN O
their PRP$ O
location NN O
, , O
size NN O
and CC O
growth NN O
. . O

Similarly RB O
, , O
a DT O
specific JJ O
T NN B-C
cell NN I-C
receptor NN I-C
variant NN I-C
has VBZ O
been VBN O
associated VBN O
with IN O
the DT O
development NN O
of IN O
DRESS NN B-C
syndrome NN I-C
, , O
SJS NNP B-C
, , O
SJS NNP B-C
/ , I-C
TEN CD I-C
, , O
and CC O
TEN CD B-C
but CC O
not RB O
AGEP NNP B-C
. . O

Claims NNS B-C
for IN I-C
priority NN I-C
of IN I-C
invention NN I-C
are VBP I-C
difficult JJ I-C
to TO I-C
resolve VB I-C
due IN O
to IN O
subsequent JJ B-C
historic JJ I-C
interpretations NNS I-C
, , O
secrecy NN B-C
, , O
espionage NN B-C
, , O
and CC O
international JJ B-C
commercial JJ I-C
interest NN I-C
. . O

Also RB O
much JJ O
of IN O
this DT O
comes VBZ O
from IN O
the DT O
transition NN B-C
from IN I-C
living VBG I-C
at IN I-C
home NN I-C
, , I-C
to IN I-C
living VBG I-C
on IN I-C
their PRP$ I-C
own JJ I-C
, , O
which WDT O
results VBZ O
in IN O
students NNS B-C
not RB I-C
knowing VBG I-C
how WRB I-C
to TO I-C
properly RB I-C
eat VB I-C
on IN I-C
their PRP$ I-C
own JJ I-C
. . O

These DT O
results NNS O
demonstrate VBP O
the DT O
age NN B-C
- HYPH O
related JJ O
progression NN B-C
of IN I-C
muscle NN I-C
weakness NN I-C
and CC O
muscular JJ O
fat JJ O
infiltration NN O
regardless RB O
of IN O
changes NNS O
in IN O
muscle NN O
mass JJ O
or CC O
subcutaneous JJ O
fat NN O
, , O
reinforcing VBG O
that IN O
muscle NN B-C
quality NN I-C
is VBZ I-C
lost VBN I-C
with IN O
aging NN B-C
. . O

Individuals NNS O
with IN O
pseudocholinesterase JJ B-C
deficiency NN I-C
also RB O
may MD O
be VB O
at IN O
increased VBN O
risk NN O
of IN O
toxic JJ B-C
reactions NNS I-C
, , O
including VBG O
sudden JJ B-C
cardiac JJ I-C
death NN I-C
, , O
associated VBN O
with IN O
recreational JJ B-C
use NN I-C
of IN I-C
cocaine NN I-C
. . O

However RB O
, , O
aberrant JJ B-C
mossy NN I-C
fiber NN I-C
sprouting NN I-C
may MD O
inhibit VB B-C
excitatory JJ I-C
transmission NN I-C
by IN O
synapsing VBG O
with IN O
basket NN O
cells NNS O
which WDT O
are VBP O
inhibitory JJ O
neurons NNS O
and CC O
by IN O
releasing VBG O
GABA NN O
and CC O
neuropeptide NN O
Y NN O
which WDT O
are VBP O
inhibitory JJ O
neurotransmitters NNS O
. . O

In IN O
the DT O
instances NNS O
where WRB O
people NNS O
experience VBP O
sudden JJ B-C
death NN I-C
, , O
it PRP O
is VBZ O
most RBS O
commonly RB O
found VBN O
that IN O
they PRP O
were VBD O
suffering VBG O
from IN O
CAD NN B-C
( -LRB- O
coronary NN O
artery NN O
disease NN O
) -RRB- O
or CC O
ASCAD NN B-C
( -LRB- O
atherosclerotic JJ O
coronary NN O
artery NN O
disease NN O
) -RRB- O
, , O
or CC O
any DT O
level NN O
of IN O
stress NN O
. . O

Thrombocytopenia NN B-C
that WDT O
starts VBZ O
after IN O
the DT O
first JJ O
72 CD O
hours NNS O
since IN O
birth NN O
is VBZ O
often RB O
the DT O
result NN O
of IN O
underlying JJ B-C
sepsis NN I-C
or CC O
necrotizing NN B-C
enterocolitis NN I-C
( -LRB- O
NEC NN O
) -RRB- O
. . O

In IN O
addition NN O
to IN O
the DT O
symptomatic JJ O
manifestation NN O
of IN O
the DT O
disease NN O
in IN O
the DT O
skin NN O
, , O
chronic JJ B-C
liver NN I-C
problems NNS I-C
are VBP O
extremely RB O
common JJ O
in IN O
patients NNS O
with IN O
the DT O
sporadic JJ O
form NN O
of IN O
PCT NN B-C
. . O

The DT O
resulting VBG O
active JJ B-C
compounds NNS I-C
cause VBP O
damage NN B-C
to IN I-C
the DT I-C
cells NNS I-C
in IN I-C
the DT I-C
lining NN I-C
of IN I-C
the DT I-C
intestine NN O
and CC O
leads NNS O
to IN O
apoptosis NN B-C
( -LRB- O
a DT O
form NN O
of IN O
cell NN O
death NN O
) -RRB- O
. . O

Nuclear JJ B-C
activity NN I-C
can MD O
cause VB O
seismic JJ B-C
activity NN I-C
, , O
but CC O
according VBG O
to IN O
USGS NNP O
, , O
seismic JJ O
activity NN O
is VBZ O
less RBR O
energetic JJ O
than IN O
the DT O
original JJ O
nuclear JJ O
blast NN O
, , O
and CC O
generally RB O
does VBZ O
not RB O
produce VB O
earthquakes NNS O
/ , O
aftershocks NNS O
of IN O
reasonable JJ O
size NN O
. . O

Ideational JJ B-C
apraxia NN I-C
is VBZ O
characterized VBN O
by IN O
the DT O
mechanism NN O
that IN O
the DT O
patient NN B-C
loses VBZ I-C
the DT I-C
“ `` I-C
idea NN I-C
” '' I-C
of IN I-C
how WRB I-C
they PRP I-C
should MD I-C
interact VB I-C
with IN I-C
an DT I-C
object NN I-C
. . O

Another DT O
possible JJ O
area NN O
of IN O
damage NN B-C
leading VBG O
to IN O
IA NNP B-C
is VBZ O
the DT O
submarginal JJ O
gyrus NN O
, , O
which WDT O
is VBZ O
located VBN O
in IN O
the DT O
parietal JJ O
lobe NN O
of IN O
the DT O
brain NN O
. . O

They PRP O
suggest VBP O
that IN O
stereotypes NNS B-C
are VBP O
the DT O
result NN O
of IN O
conflict NN B-C
, , O
poor JJ B-C
parenting NN I-C
, , O
and CC O
inadequate JJ B-C
mental JJ I-C
and CC I-C
emotional JJ I-C
development NN I-C
. . O

Alcoholic JJ B-C
hepatitis NN I-C
by IN O
itself PRP O
does VBZ O
not RB O
lead VB O
to IN O
cirrhosis NN B-C
, , O
but CC O
cirrhosis NN O
is VBZ O
more RBR O
common JJ O
in IN O
patients NNS O
with IN O
long JJ O
term NN O
alcohol NN O
consumption NN O
. . O

Patients NNS O
with IN O
accelerated VBN B-C
silicosis NN I-C
are VBP O
at IN O
greater JJR O
risk NN O
for IN O
complicated JJ B-C
disease NN I-C
, , O
including VBG O
progressive JJ B-C
massive JJ I-C
fibrosis NN I-C
( -LRB- O
PMF NN O
) -RRB- O
. . O

Very RB O
rarely RB O
, , O
extremely RB B-C
large JJ I-C
hemangiomas NNS I-C
can MD O
cause VB O
high JJ B-C
- HYPH I-C
output NN I-C
heart NN I-C
failure NN I-C
due IN O
to IN O
the DT O
amount NN O
of IN O
blood NN O
that WDT O
must MD O
be VB O
pumped VBN O
to IN O
excess JJ O
blood NN O
vessels NNS O
. . O

When WRB O
a DT O
person NN B-C
takes VBZ I-C
in RP I-C
more JJR I-C
or CC I-C
fewer JJR I-C
calories NNS I-C
than IN O
that DT O
set JJ O
limit NN O
, , O
weight NN B-C
is VBZ I-C
either CC I-C
gained VBN I-C
or CC I-C
lost VBN I-C
. . O

Unlike IN O
gamma JJ O
rays NNS O
, , O
beta NN B-C
emissions NNS I-C
are VBP O
stopped VBN O
much RB O
more RBR O
effectively RB O
by IN O
materials NNS O
and CC O
therefore RB O
deposit VB O
all PDT O
their PRP$ O
energy NN O
in IN O
only RB O
a DT O
shallow JJ O
layer NN O
of IN O
tissue NN O
, , O
causing VBG O
more RBR O
intense JJ B-C
but CC I-C
more RBR I-C
localized VBN I-C
damage NN I-C
. . O

Due IN O
to IN O
Haiti NNP B-C
's POS I-C
widespread JJ I-C
economic JJ I-C
hardship NN I-C
, , O
the DT B-C
majority NN I-C
of IN I-C
the DT I-C
nation NN I-C
's POS I-C
population NN I-C
is VBZ I-C
unable JJ I-C
to TO I-C
meet VB I-C
the DT I-C
cost NN I-C
of IN I-C
education NN I-C
. . O

This DT O
is VBZ O
because IN O
when WRB O
the DT O
body NN B-C
is VBZ I-C
stressed VBN I-C
, , O
it PRP O
releases VBZ O
hormones NNS B-C
such JJ O
as IN O
adrenaline NN B-C
or CC O
more RBR O
importantly RB O
cortisol NN B-C
. . O

Wastewater NN B-C
injection NN I-C
, , O
hydraulic NN B-C
fracturing NN I-C
, , O
and CC O
secondary JJ B-C
recovery NN I-C
after IN I-C
oil NN I-C
extraction NN I-C
have VBP O
all RB O
contributed VBN O
significantly RB O
more JJR O
to IN O
induced VBN O
seismic JJ B-C
events NNS I-C
than IN O
carbon NN B-C
capture NN I-C
and CC I-C
storage NN I-C
in IN O
the DT O
last JJ O
several JJ O
years NNS O
. . O

This DT O
depression NN B-C
that WDT O
is VBZ O
caused VBN O
by IN O
prejudice NN B-C
( -LRB- O
i.e. FW O
, , O
" `` O
deprejudice NN O
" '' O
) -RRB- O
can MD O
be VB O
related JJ O
to IN O
a DT O
group NN O
membership NN O
( -LRB- O
e.g. FW O
, , O
Me PRP O
– , O
Gay NNP O
– , O
Bad JJ O
) -RRB- O
or CC O
not RB O
( -LRB- O
e.g. FW O
, , O
Me PRP O
– , O
Bad JJ O
) -RRB- O
. . O

During IN O
the DT O
1970s NNS O
and CC O
1980s NNS O
, , O
when WRB O
an DT O
outbreak NN O
of IN O
this DT O
syndrome NN O
began VBD O
, , O
many JJ O
of IN O
the DT O
Southeast NNP B-C
Asians NNPS I-C
were VBD I-C
not RB I-C
able JJ I-C
to TO I-C
worship VB I-C
properly RB I-C
due IN O
to IN O
the DT O
guerrilla NN B-C
war NN I-C
against IN O
the DT O
government NN O
of IN O
Laos NNP O
with IN O
the DT O
United NNP O
States NNP O
. . O

Macierewicz NNP B-C
repeated VBD I-C
these DT I-C
accusations NNS I-C
as IN I-C
newly RB I-C
appointed VBN I-C
minister NN I-C
of IN I-C
defence NN I-C
in IN I-C
2016 CD I-C
, , O
causing VBG O
a DT O
significant JJ B-C
controversy NN I-C
in IN O
Polish NNP O
, , O
Russian JJ O
and CC O
international JJ O
media NN O
. . O

The DT O
damage NN B-C
caused VBN O
by IN O
calcium NN B-C
carbonate NN I-C
deposits NNS I-C
varies VBZ O
on IN O
the DT O
crystalline JJ O
form NN O
, , O
for IN O
example NN O
, , O
calcite NN O
or CC O
aragonite NN O
. . O

Those DT O
circumstances NNS B-C
led VBD O
to IN O
an DT O
impact NN B-C
on IN I-C
a DT I-C
terrain NN I-C
obstacle NN I-C
resulting VBG O
in IN O
separation NN B-C
of IN I-C
a DT I-C
part NN I-C
of IN I-C
the DT I-C
left VBN I-C
wing NN I-C
with IN I-C
aileron NN I-C
and CC O
consequently RB O
to IN O
the DT O
loss NN B-C
of IN I-C
aircraft NN I-C
control NN I-C
and CC O
eventual JJ O
ground NN B-C
impact NN I-C
" '' O
Circumstances NNS O
contributing VBG O
to IN O
the DT O
accident NN O
: : O
Failure NN O
to TO O
monitor VB O
altitude NN O
by IN O
means NNS O
of IN O
a DT O
pressure NN O
altimeter NN O
during IN O
a DT O
non-precision JJ O
approach NN O
; , O
failure NN O
by IN O
the DT O
crew NN O
to TO O
respond VB O
to IN O
the DT O
PULL NN O
UP RP O
warning NN O
generated VBN O
by IN O
the DT O
TAWS NN O
; , O
attempt NN O
to TO O
execute VB O
the DT O
go NN O
- HYPH O
around IN O
maneuver NN O
under IN O
the DT O
control NN O
of IN O
ABSU NNP O
( -LRB- O
automatic JJ O
go-around NN O
) -RRB- O
; , O
Approach NNP O
Control NNP O
confirming VBG O
to IN O
the DT O
crew NN O
the DT O
correct JJ O
position NN O
of IN O
the DT O
airplane NN O
in IN O
relation NN O
to IN O
the DT O
RWY NNP O
threshold NN O
, , O
glide NN O
slope NN O
, , O
and CC O
course RB O
which WDT O
might MD O
have VB O
affirmed VBN O
the DT O
crew NN O
's POS O
belief NN O
that IN O
the DT O
approach NN O
was VBD O
proceeding VBG O
correctly RB O
although IN O
the DT O
airplane NN O
was VBD O
actually RB O
outside IN O
the DT O
permissible JJ O
deviation NN O
margin NN O
; , O
failure NN O
by IN O
LZC NNP O
to TO O
inform VB O
the DT O
crew NN O
about IN O
descending VBG O
below IN O
the DT O
glide NN O
slope NN O
and CC O
delayed VBN O
issuance NN O
of IN O
the DT O
level NN O
- HYPH O
out NN O
command NN O
; , O
incorrect JJ O
training NN O
of IN O
the DT O
Tu NNP O
- HYPH O
154M NNP O
flight NN O
crews NNS O
in IN O
the DT O
36 CD O
Regiment NNP O
. . O