Radiation NN B-C recall NN I-C reactions NNS I-C occur VBP O months NNS O to IN O years NNS O after IN O radiation NN B-C treatment NN I-C , , O a DT O reaction NN O that WDT O follows VBZ O recent JJ O administration NN O of IN O a DT O chemotherapeutic JJ O agent NN O and CC O occurs VBZ O with IN O the DT O prior JJ O radiation NN O port NN O , , O characterized VBN O by IN O features NNS B-C of IN I-C radiation NN I-C dermatitis NN I-C . . O Other JJ O possible JJ O causes NNS O of IN O ovarian JJ B-C rupture NN I-C include VBP O abdominal JJ B-C trauma NN I-C , , O excessive JJ B-C physical JJ I-C stress NN I-C , , O vigorous JJ B-C sexual JJ I-C intercourse NN I-C , , O horseback NN B-C riding NN I-C , , O etc FW O . . O Also RB O , , O each DT O joint NN O has VBZ O a DT O rich JJ O supply NN O of IN O tiny JJ O nerve NN O fibers NNS O that WDT O provide VBP O a DT O painful JJ B-C stimulus NN I-C when WRB O the DT O joint NN B-C is VBZ I-C injured VBN I-C or CC I-C irritated VBN I-C . . O Baylisascaris JJ B-C larvae NNS I-C in IN O paratenic JJ O hosts NNS O can MD O migrate VB O , , O causing VBG O visceral JJ B-C larva NN I-C migrans NNS I-C ( -LRB- O VLM NNP O ) -RRB- O . . O It PRP O is VBZ O extremely RB O dangerous JJ O to IN O the DT O host NN O due IN O to IN O the DT O ability NN O of IN O the DT O parasite NN B-C 's POS I-C larvae NNS I-C to TO O migrate VB O into IN O brain NN O tissue NN O and CC O cause VB O damage NN B-C . . O Stems NNS B-C of IN I-C seedlings NNS I-C may MD I-C also RB I-C become VB I-C thin JJ I-C and CC I-C tough JJ I-C ( -LRB- O " `` O wire NN O - HYPH O stem NN O " '' O ) -RRB- O resulting VBG O in IN O reduced VBN B-C seedling NN I-C vigor NN I-C . . O Since IN O these DT O channels NNS O are VBP O responsible JJ O for IN O the DT O removal NN O of IN O intracellular JJ O chloride NN O , , O these DT O mutations NNS B-C are VBP O expected VBN O to TO O lead VB O to IN O increased VBN B-C chloride NN I-C concentrations NNS I-C and CC O , , O thus RB O , , O altered VBN B-C chloride NN I-C reversal NN I-C potential NN I-C ( -LRB- O ECl NN O ) -RRB- O . . O The DT O silicotic JJ B-C nodule NN I-C represents VBZ O a DT O specific JJ O tissue NN O response NN O to IN O crystalline JJ B-C silica NN I-C . . O The DT O remaining VBG O cases NNS O of IN O AGEP NNP B-C have VBP O been VBN O associated VBN O with IN O infective JJ B-C and CC I-C other JJ I-C agents NNS I-C . . O Colonization NN B-C with IN I-C the DT I-C liver NN I-C flukes NNS I-C Opisthorchis NNP I-C viverrini NN I-C ( -LRB- O found VBN O in IN O Thailand NNP O , , O Laos NNP O PDR NNP O , , O and CC O Vietnam NNP O ) -RRB- O or CC O Clonorchis NNP B-C sinensis NNP I-C ( -LRB- O found VBN O in IN O China NNP O , , O Taiwan NNP O , , O eastern JJ O Russia NNP O , , O Korea NNP O , , O and CC O Vietnam NNP O ) -RRB- O has VBZ O been VBN O associated VBN O with IN O the DT O development NN O of IN O cholangiocarcinoma NN B-C . . O A DT O study NN O of IN O radiation NN B-C - HYPH O induced VBN O skin NN B-C injuries NNS I-C has VBZ O been VBN O performed VBN O by IN O the DT O Food NNP O and CC O Drug NNP O Administration NNP O ( -LRB- O FDA NNP O ) -RRB- O based VBN O on IN O results NNS O from IN O 1994 CD O , , O followed VBN O by IN O an DT O advisory NN O to TO O minimize VB O further JJ O fluoroscopy JJ B-C - HYPH O induced VBN O injuries NNS B-C . . O These DT O include VBP O both DT O unfavorable JJ O genes NNS O , , O where WRB O high JJ B-C expression NN I-C is VBZ O related JJ O to IN O poor JJ B-C outcome NN I-C , , O for IN O example NN O C NN O - HYPH O Met VBN O and CC O MUC NN O - HYPH O 1 CD O , , O and CC O favorable JJ O genes NNS O where WRB O high JJ B-C expression NN I-C is VBZ O associated VBN O with IN O better JJR B-C survival NN I-C , , O for IN O example NN O the DT O transcription NN O factor NN O PELP1 NN O . . O Affected VBN B-C proteins NNS I-C interfere VBP I-C with IN I-C the DT I-C immune JJ I-C system NN I-C 's POS I-C ability NN I-C to TO I-C recognize VB I-C these DT I-C cells NNS I-C as IN I-C normal JJ I-C parts NNS I-C of IN I-C the DT I-C body NN I-C , , O causing VBG O a DT O T NN B-C - HYPH I-C cell NN I-C - HYPH I-C mediated VBN I-C immune JJ I-C response NN I-C . . O Skin NN B-C lesions NNS I-C in IN I-C the DT I-C fingertips NNS I-C may MD O sometimes RB O bleed VB O and CC O cause VB O pain NN B-C . . O Excess JJ B-C alcohol NN I-C intake NN I-C decreases VBZ O hepcidin NN O production NN O which WDT O leads VBZ O to IN O increased VBN B-C iron NN I-C absorption NN I-C from IN O the DT O gut NN O and CC O an DT O increase NN B-C in IN I-C oxidative NN I-C stress NN I-C . . O Numerous JJ B-C accidents NNS I-C also RB O occur VB O during IN O radiotherapy NN O due IN O to IN O equipment NN B-C failures NNS I-C , , O operator NN B-C errors NNS I-C , , O or CC O wrong JJ B-C dosage NN I-C . . O Cold JJ O air NN O damming NN O , , O or CC O CAD NNP O , , O is VBZ O a DT O meteorological JJ O phenomenon NN O that WDT O involves VBZ O a DT O high JJ B-C - HYPH I-C pressure NN I-C system NN I-C ( -LRB- I-C anticyclone NN I-C ) -RRB- I-C accelerating VBG I-C equatorward JJ I-C east NN I-C of IN I-C a DT I-C north JJ I-C - HYPH I-C south JJ I-C oriented JJ I-C mountain NN I-C range NN I-C due IN O to IN O the DT O formation NN B-C of IN I-C a DT I-C barrier NN I-C jet NN I-C behind IN I-C a DT I-C cold JJ I-C front NN I-C associated VBN O with IN O the DT O poleward JJ O portion NN O of IN O a DT O split JJ O upper JJ O level NN O trough NN O . . O Loss NN B-C - HYPH I-C of IN I-C - HYPH I-C function NN I-C mutations NNS I-C in IN I-C SLC2A9 NNP I-C and CC O SLC22A12 NNP O cause VBP O hereditary JJ B-C hypouricaemia NN I-C by IN O reducing VBG O urate JJ O absorption NN O and CC O unopposed JJ O urate NN O secretion NN O . . O Peutz NNP B-C - HYPH I-C Jeghers NN I-C syndrome NN I-C causes VBZ O pigmentation NN B-C of IN I-C the DT I-C skin NN I-C and CC O mucous NN B-C surfaces NNS I-C with IN I-C melanin NN I-C , , O and CC O polyps NNS B-C in IN I-C the DT I-C digestive JJ I-C tract NN I-C . . O There EX O have VBP O actually RB O not RB O been VBN O any DT O major JJ O seismic JJ B-C events NNS I-C associated VBN O with IN O carbon NN B-C injection NN I-C at IN O this DT O point NN O , , O whereas IN O there EX O have VBP O been VBN O recorded VBN O seismic JJ B-C occurrences NNS I-C caused VBN O by IN O the DT O other JJ B-C injection NN I-C methods NNS I-C . . O Like IN O the DT O ' `` O functioning VBG O ' '' O endocrine JJ O cancers NNS O described VBN O below RB O , , O acinar JJ B-C cell NN I-C carcinomas NNS I-C may MD O cause VB O over- JJ B-C production NN I-C of IN I-C certain JJ I-C molecules NNS I-C , , O in IN O this DT O case NN O digestive JJ B-C enzymes NNS I-C , , O which WDT O may MD O cause VB O symptoms NNS O such JJ O as IN O skin NN B-C rashes NNS I-C and CC O joint JJ B-C pain NN I-C . . O Most RBS O generally RB O , , O failure NN B-C will MD I-C happen VB I-C on IN I-C existing VBG I-C faults NNS I-C due IN O to IN O several JJ O mechanisms NNS O : : O an DT O increase NN B-C in IN I-C shear NN I-C stress NN I-C , , O a DT O decrease NN B-C in IN I-C normal JJ I-C stress NN I-C or CC O a DT O pore NN B-C pressure NN I-C increase NN I-C . . O People NNS O with IN O degenerated VBN B-C joints NNS I-C in IN O the DT O upper JJ O spine NN O will MD O often RB O feel VB O pain NN B-C radiating VBG O throughout IN O the DT O upper JJ O neck NN O and CC O shoulders NNS O . . O As IN O chloride NN O is VBZ O conducted VBN O through IN O the DT O normally RB O inhibitory JJ O GABA NN O receptors NNS O , , O this DT O alteration NN B-C in IN I-C ECl NNP I-C may MD O lead VB O to IN O either CC O decreased VBN B-C GABAergic NN I-C currents NNS I-C or CC O GABAergic NN B-C currents NNS I-C that WDT I-C are VBP I-C actually RB I-C excitatory JJ I-C . . O In IN O TLE NNP B-C , , O there EX O is VBZ O loss NN B-C of IN I-C neurons NNS I-C in IN I-C region NN I-C CA1 NNP I-C and CC I-C CA3 NNP I-C of IN I-C the DT I-C hippocampus NN I-C . . O Cause NN O of IN O accident NN O according VBG O to IN O Polish NNP O report NN O : : O " `` O The DT O immediate JJ O cause NN O of IN O the DT O accident NN B-C was VBD O the DT O descent NN B-C below IN I-C the DT I-C minimum JJ I-C descent NN I-C altitude NN I-C at IN O an DT O excessive JJ O rate NN O of IN O descent NN O in IN O weather NN O conditions NNS O which WDT O prevented VBD O visual JJ O contact NN O with IN O the DT O ground NN O , , O as RB O well RB O as IN O a DT O delayed VBN B-C execution NN I-C of IN I-C the DT I-C go- NN I-C around IN I-C procedure NN I-C . . O Seven CD B-C died VBD I-C after IN O dose NN O of IN O 4 CD O – SYM O 6 CD O Gy NN O and CC O third JJ B-C degree NN I-C beta NN I-C burns NNS I-C in IN O 4 CD O – SYM O 6 CD O weeks NNS O . . O Radiation NN B-C dermatitis NN I-C , , O in IN O the DT O form NN O of IN O intense JJ O erythema NN O and CC O vesiculation NN O of IN O the DT O skin NN O , , O may MD O be VB O observed VBN O in IN O radiation NN B-C ports NNS I-C . . O Conversely RB O , , O a DT O very RB B-C severe JJ I-C bit NN I-C , , O in IN O the DT O right JJ O hands NNS O , , O can MD O transmit VB O extremely RB B-C subtle JJ I-C , , I-C nuanced JJ I-C signals NNS I-C that WDT O cause VBP O no DT O pain NN B-C to IN I-C the DT I-C horse NN I-C . . O Operation NN B-C of IN I-C technologies NNS I-C involving VBG I-C long JJ I-C - HYPH I-C term NN I-C geologic JJ I-C storage NN I-C of IN I-C waste NN I-C fluids NNS I-C have VBP O been VBN O shown VBN O to TO O induce VB O seismic JJ B-C activity NN I-C in IN I-C nearby JJ I-C areas NNS I-C , , O and CC O correlation NN O of IN O periods NNS B-C of IN I-C seismic JJ I-C dormancy NN I-C with IN O minima NNS O in IN O injection NN O volumes NNS O and CC O pressures NNS O has VBZ O even RB O been VBN O demonstrated VBN O for IN O fracking NN B-C wastewater NN I-C injection NN I-C in IN I-C Youngstown NNP I-C , , I-C Ohio NNP I-C . . O Reservoir NN B-C - HYPH O induced VBN O seismic JJ B-C events NNS I-C can MD O be VB O relatively RB O large JJ O compared VBN O to IN O other JJ O forms NNS O of IN O induced VBN O seismicity NN O . . O Autosomal JJ B-C dominant JJ I-C lateral JJ I-C temporal JJ I-C lobe NN I-C epilepsy NN I-C ( -LRB- O ADLTLE NNP O ) -RRB- O is VBZ O a DT O rare JJ O hereditary JJ O condition NN O , , O often RB O associated VBN O with IN O mutations NNS B-C in IN I-C the DT I-C LGI1 NN I-C gene NN I-C . . O As IN O the DT O ionizing NN B-C radiation NN I-C interacts VBZ O with IN O cells NNS O within IN O the DT O body NN O —damaging VBG O them PRP O — , O the DT O body NN O responds VBZ O to IN O this DT O damage NN O , , O typically RB O resulting VBG O in IN O erythema—that NN O is VBZ O , , O redness NN B-C around IN I-C the DT I-C damaged VBN I-C area NN I-C . . O The DT O strong JJ O association NN O of IN O PCT NN B-C with IN O Hepatitis NN B-C C NN I-C infection NN I-C is VBZ O not RB O entirely RB O understood VBN O . . O Additionally RB O , , O the DT O MAK NNP O report NN O found VBD O an DT O " `` O immediate JJ O cause NN O " '' O of IN O the DT O accident NN B-C was VBD O the DT O presence NN B-C in IN I-C the DT I-C cockpit NN I-C of IN I-C the DT I-C Commander NNP I-C - HYPH I-C in IN I-C - HYPH I-C Chief NNP I-C of IN I-C the DT I-C Polish NNP I-C Air NNP I-C Force NNP I-C , , O which WDT O placed VBD O extreme JJ O stress NN O and CC O " `` O psychological JJ O pressure NN O " '' O on IN O the DT O Captain NNP O to TO O " `` O continue VB O descent NN O in IN O conditions NNS O of IN O unjustified JJ O risk NN O with IN O a DT O dominating JJ O aim NN O of IN O landing VBG O at IN O any DT O means NNS O . . O " '' O They PRP O recommend VBP O the DT O alpha JJ O roll NN O should MD O never RB O be VB O used VBN O by IN O inexperienced JJ O trainers NNS O , , O and CC O never RB O to TO O correct JJ O undesired JJ B-C behavior NN I-C caused VBN O by IN O the DT O dog NN B-C 's POS I-C failure NN I-C to TO I-C understand VB I-C a DT I-C command NN I-C . . O Mixed VBN B-C - HYPH I-C field NN I-C agglutination NN I-C is VBZ O an DT O important JJ O cause NN O of IN O ABO NN B-C typing NN I-C and CC O genotype NN B-C discrepancies NNS I-C . . O A DT O radiation NN B-C burn NN I-C is VBZ O damage NN O to IN O the DT O skin NN O or CC O other JJ O biological JJ O tissue NN O as IN O an DT O effect NN O of IN O radiation NN B-C . . O Due IN O to IN O the DT O area NN O 's POS O frequent JJ B-C fires NNS I-C , , O the DT O seeds NNS B-C are VBP I-C usually RB I-C released VBN I-C onto IN I-C a DT I-C recently RB I-C bur NN I-C nt RB I-C seed NN I-C bed NN I-C . . O Most JJS O cases NNS O of IN O Reye NN B-C syndrome NN I-C are VBP O associated VBN O with IN O the DT O use NN B-C of IN I-C aspirin NN I-C during IN O these DT O viral JJ O infections NNS O . . O People NNS O with IN O fluorosis NN O are VBP O relatively RB O resistant JJ O to IN O dental JJ O caries NNS O ( -LRB- O tooth NN B-C decay NN I-C caused VBN O by IN O bacteria NN B-C ) -RRB- O , , O although IN O they PRP O may MD O be VB O of IN O cosmetic JJ O concern NN O . . O The DT O quality NN B-C of IN I-C education NN I-C is VBZ I-C low JJ I-C due IN O to IN O the DT O lack NN B-C of IN I-C organization NN I-C , , I-C expertise NN I-C , , I-C and CC I-C resources NNS I-C . . O This DT O Nazi NNP B-C policy NN I-C caused VBD O a DT O humanitarian JJ B-C crisis NN I-C in IN O Poland NNP O ’s POS O urban JJ O areas NNS O . . O Other JJ O causes NNS O of IN O pelvic JJ B-C floor NN I-C prolapse NN I-C can MD O be VB O advanced VBN B-C age NN I-C , , O multiple JJ B-C vaginal JJ I-C deliveries NNS I-C , , O and CC O birthing NN B-C trauma NN I-C . . O In IN O Dupuytren NNP B-C 's POS I-C contracture NN I-C , , O the DT O palmar JJ B-C fascia NN I-C within IN I-C the DT I-C hand NN I-C becomes VBZ I-C abnormally RB I-C thick JJ I-C , , O which WDT O can MD O cause VB O the DT O fingers NNS B-C to TO I-C curl VB I-C and CC O can MD O impair VB O finger NN O function NN O . . O The DT O mutation NN B-C also RB O results VBZ O in IN O greatly RB B-C decreased VBN I-C sensitivity NN I-C in IN I-C the DT I-C receptor NN I-C for IN I-C activation NN I-C by IN I-C GABA NNP I-C . . O For IN O example NN O , , O the DT O WHO WP O confirmed VBD O that IN O a DT O typhoid NN B-C fever NN I-C outbreak NN I-C that WDT O occurred VBD O in IN O 2003 CD O was VBD O due IN O to IN O a DT O lack NN B-C of IN I-C access NN I-C to IN I-C doctors NNS I-C and CC I-C clean JJ I-C water NN I-C infrastructure NN I-C . . O Urushiol NNP B-C causes VBZ O an DT O eczematous JJ B-C contact NN I-C dermatitis NN I-C characterized VBN O by IN O redness NN B-C , , O swelling NN B-C , , O papules NNS O , , O vesicles NNS B-C , , O blisters NNS B-C , , O and CC O streaking NN B-C . . O Injecting VBG B-C liquids NNS I-C into IN I-C waste NN I-C disposal NN I-C wells NNS I-C , , O most RBS O commonly RB O in IN O disposing NN O of IN O produced VBN O water NN O from IN O oil NN O and CC O natural JJ O gas NN O wells NNS O , , O has VBZ O been VBN O known VBN O to TO O cause VB O earthquakes NNS B-C . . O One CD O explanation NN O for IN O why WRB O stereotypes NNS B-C are VBP O shared VBN O is VBZ O that IN O they PRP O are VBP O the DT O result NN O of IN O a DT O common JJ B-C environment NN I-C that WDT O stimulates VBZ O people NNS O to TO O react VB O in IN O the DT O same JJ O way NN O . . O Radiation NN B-C burns NNS I-C can MD O also RB O occur VB O with IN O high JJ B-C power NN I-C radio NN I-C transmitters NNS I-C at IN O any DT O frequency NN O where WRB O the DT O body NN O absorbs VBZ O radio NN O frequency NN O energy NN O and CC O converts VBZ O it PRP O to IN O heat NN O . . O Their PRP$ O displacement NN O by IN O Europeans NNPS O meant VBD O a DT O complete JJ B-C regime NN I-C shift NN I-C in IN I-C the DT I-C ecology NN I-C of IN I-C the DT I-C Australian JJ I-C bush NN I-C with IN O the DT O result NN O that IN O tremendous JJ B-C fires NNS I-C become VBN I-C possible JJ I-C . . O Both DT O surgery NN B-C and CC I-C advanced JJ I-C inoperable JJ I-C tumors NNS I-C often RB O lead VBP O to IN O digestive JJ B-C system NN I-C disorders NNS I-C from IN O a DT O lack NN B-C of IN I-C the DT I-C exocrine JJ I-C products NNS I-C of IN I-C the DT I-C pancreas NN I-C ( -LRB- O exocrine JJ O insufficiency NN O ) -RRB- O . . O These DT O lesions NNS B-C may MD O bleed VB O intermittently RB O , , O which WDT O is VBZ O rarely RB O significant JJ O enough RB O to TO O be VB O noticed VBN O ( -LRB- O in IN O the DT O form NN O of IN O bloody JJ O vomiting NN O or CC O black JJ O stool NN O ) -RRB- O , , O but CC O can MD O eventually RB O lead VB O to IN O depletion NN B-C of IN I-C iron NN I-C in IN I-C the DT I-C body NN I-C , , O resulting VBG O in IN O iron-deficiency JJ B-C anemia NN I-C . . O Continuous JJ O use NN O of IN O a DT O squealing NN B-C tape NN I-C risks NNS O permanently RB O damaging VBG B-C the DT I-C tape NN I-C , , O as IN O oxide NN O is VBZ O sometimes RB O torn VBN O off IN O the DT O tape NN O . . O Silicosis NN B-C ( -LRB- O also RB O known VBN O as IN O miner NN O 's POS O phthisis NNP O , , O grinder NN O 's POS O asthma NN O , , O potter NNP O 's POS O rot NN O and CC O other JJ O occupation NN O - HYPH O related JJ O names NNS O , , O or CC O by IN O the DT O invented VBN O name NN O pneumonoultramicroscopicsilicovolcanoconiosis ADD O ) -RRB- O is VBZ O a DT O form NN O of IN O occupational JJ O lung NN B-C disease NN I-C caused VBN O by IN O inhalation NN B-C of IN I-C crystalline JJ I-C silica NN I-C dust NN I-C , , O and CC O is VBZ O marked VBN O by IN O inflammation NN B-C and CC O scarring NN B-C in IN O the DT O form NN O of IN O nodular JJ B-C lesions NNS I-C in IN O the DT O upper JJ O lobes NNS O of IN O the DT O lungs NNS O . . O Hemangiomas NNS B-C , , O especially RB O those DT O that WDT O have VBP O gotten VBN O very RB O large JJ O during IN O the DT O growth NN O phase NN O , , O may MD O leave VB O behind IN O stretched VBN B-C skin NN I-C or CC O fibrofatty NN B-C tissue NN I-C that WDT O may MD O be VB O disfiguring VBG O or CC O require VB O future JJ O surgical JJ O correction NN O . . O A DT O number NN O of IN O different JJ O fungi NNS B-C and CC I-C fungi-like JJ I-C organisms NNS I-C cause VBP O the DT O symptoms NNS B-C of IN I-C damping VBG I-C off RP I-C , , O including VBG O : : O Alternaria JJ B-C species NNS I-C . . O There EX O are VBP O hundreds NNS O of IN O reasons NNS O for IN O why WRB O college NN O students NNS B-C get VBP I-C stressed VBN I-C , , O but CC O , , O whatever WDT O the DT O reason NN O , , O it PRP O also RB O can MD O lead VB O to IN O weight NN B-C gain NN I-C . . O It PRP O appears VBZ O as IN O a DT O range NN B-C of IN I-C visual JJ I-C changes NNS I-C in IN I-C enamel NN I-C causing VBG O degrees NNS B-C of IN I-C intrinsic JJ I-C tooth NN I-C discoloration NN I-C , , O and CC O , , O in IN O some DT O cases NNS O , , O physical JJ B-C damage NN I-C to IN I-C the DT I-C teeth NNS I-C . . O Silicosis NN B-C is VBZ O due IN O to IN O deposition NN B-C of IN I-C fine JJ I-C respirable JJ I-C dust NN I-C ( -LRB- O less JJR O than IN O 10 CD O micrometers NNS O in IN O diameter NN O ) -RRB- O containing VBG O crystalline JJ O silicon NN O dioxide NN O in IN O the DT O form NN O of IN O alpha-quartz JJ O , , O cristobalite NN O , , O or CC O tridymite NN O . . O The DT O pilot NN B-C lost VBD I-C control NN I-C and CC O the DT O aircraft NN B-C crashed VBD I-C as IN O a DT O result NN O of IN O this DT O unusual JJ B-C maneuver NN I-C plus CC O the DT O crew NN B-C 's POS I-C loss NN I-C of IN I-C situational JJ I-C awareness NN I-C in IN O the DT O low JJ O visibility NN O conditions NNS O . . O Aside RB O from IN O individual JJ O impact NN O , , O structural JJ B-C violence NN I-C also RB O impacts VBZ O the DT O society NN O as IN O a DT O whole JJ O as IN O it PRP O facilitates VBZ O inequity NN B-C in IN O terms NNS O of IN O the DT O distribution NN O of IN O a DT O society NN O 's POS O resources NNS O and CC O hence RB O the DT O overall JJ O quality NN O of IN O life NN O of IN O a DT O society NN O . . O History NN O and CC O examination NN O by IN O a DT O physician NN O for IN O characteristic JJ O signs NNS O and CC O symptoms NNS O are VBP O sufficient JJ O in IN O many JJ O cases NNS O in IN O ruling VBG O out RP O systemic JJ O causes NNS O of IN O venous JJ B-C hypertension NN I-C such JJ O as IN O hypervolemia NN B-C and CC O heart NN B-C failure NN I-C . . O Temporal JJ B-C lobe NN I-C epilepsy NN I-C ( -LRB- O TLE NN O ) -RRB- O is VBZ O a DT O chronic JJ O disorder NN O of IN O the DT O nervous JJ O system NN O characterized VBN O by IN O recurrent JJ O , , O unprovoked JJ O focal JJ B-C seizures NNS I-C that WDT O originate VBP O in IN O the DT O temporal JJ O lobe NN O of IN O the DT O brain NN O and CC O last VB O about RB O one CD O or CC O two CD O minutes NNS O . . O Columns NNS B-C at IN I-C the DT I-C perimeter NN I-C of IN I-C the DT I-C building NN I-C were VBD I-C not RB I-C strong JJ I-C enough RB I-C to TO I-C support VB I-C the DT I-C weight NN I-C of IN I-C cement NN I-C being VBG I-C poured VBN I-C on IN I-C the DT I-C roof NN I-C , , O leading VBG O to IN O a DT O structural JJ B-C failure NN I-C . . O Emotional JJ B-C difficulties NNS I-C in IN I-C coping VBG I-C with IN I-C the DT I-C pain NN I-C of IN O childbirth NN O can MD O also RB O cause VB O psychological JJ B-C trauma NN I-C . . O One CD O such JJ O example NN O is VBZ O massively RB B-C increased VBN I-C induced VBN I-C seismicity NN I-C in IN I-C Oklahoma NNP I-C , , I-C USA NNP I-C caused VBN O by IN O injection NN B-C of IN I-C huge JJ I-C volumes NNS I-C of IN I-C wastewater NN I-C into IN O the DT O Arbuckle NNP O Group NNP O sedimentary JJ O rock NN O . . O Chronic NN B-C simple JJ I-C silicosis NN I-C has VBZ O been VBN O reported VBN O to TO O occur VB O from IN O environmental JJ B-C exposures NNS I-C to IN I-C silica NN I-C in IN O regions NNS O with IN O high JJ O silica NN O soil NN O content NN O and CC O frequent JJ O dust NN O storms NNS O . . O However RB O , , O intrahepatic JJ B-C stones NNS I-C ( -LRB- O called VBN O hepatolithiasis NN O ) -RRB- O , , O which WDT O are VBP O rare JJ O in IN O the DT O West NNP O but CC O common JJ O in IN O parts NNS O of IN O Asia NNP O , , O have VBP O been VBN O strongly RB O associated VBN O with IN O cholangiocarcinoma NN B-C . . O Some DT O individuals NNS O with IN O thrombocytopenia NN B-C may MD O experience VB O external JJ B-C bleeding NN I-C such JJ O as IN O nosebleeds NNS B-C , , O and CC O / , O or CC O bleeding NN B-C gums NNS I-C . . O The DT O most RBS O common JJ O physical JJ O indications NNS O of IN O cholangiocarcinoma NN B-C are VBP O abnormal JJ B-C liver NN I-C function NN I-C tests NNS I-C , , O jaundice NN B-C ( -LRB- O yellowing NN O of IN O the DT O eyes NNS O and CC O skin NN O occurring VBG O when WRB O bile NN O ducts NNS O are VBP O blocked VBN O by IN O tumor NN O ) -RRB- O , , O abdominal JJ B-C pain NN I-C ( -LRB- O 30 CD O – SYM O 50 CD O % NN O ) -RRB- O , , O generalized VBN B-C itching NN I-C ( -LRB- O 66 CD O % NN O ) -RRB- O , , O weight NN B-C loss NN I-C ( -LRB- O 30 CD O – SYM O 50 CD O % NN O ) -RRB- O , , O fever NN B-C ( -LRB- O up IN O to IN O 20 CD O % NN O ) -RRB- O , , O and CC O changes NNS B-C in IN I-C the DT I-C color NN I-C of IN I-C stool NN I-C or CC I-C urine NN I-C . . O Allergenic JJ B-C extracts NNS I-C , , O hormones NNS B-C and CC O vaccines NNS B-C can MD O also RB O cause VB O serum NN B-C sickness NN I-C . . O The DT O Asian JJ B-C brown JJ I-C cloud NN I-C is VBZ O created VBN O by IN O a DT O range NN O of IN O airborne JJ B-C particles NNS I-C and CC O pollutants NNS B-C from IN I-C combustion NN I-C ( -LRB- O e.g. FW O , , O woodfires NNS O , , O cars NNS O , , O and CC O factories NNS O ) -RRB- O , , O biomass NN B-C burning NN I-C and CC O industrial JJ B-C processes NNS I-C with IN O incomplete JJ O burning NN O . . O Similarly RB O , , O computed VBN B-C tomography NN I-C and CC O traditional JJ B-C projectional JJ I-C radiography NN I-C have VBP O the DT O potential NN O to TO O cause VB O radiation NN B-C burns NNS I-C if IN O the DT O exposure NN O factors NNS O and CC O exposure NN O time NN O are VBP O not RB O appropriately RB O controlled VBN O by IN O the DT O operator NN O . . O Systemic NN B-C JIA NNP I-C is VBZ O characterized VBN O by IN O arthritis NN B-C , , O fever NN B-C , , O which WDT O typically RB O is VBZ O higher JJR O than IN O the DT O low JJ O - HYPH O grade NN O fever NN O associated VBN O with IN O polyarticular JJ B-C and CC O a DT O salmon NN B-C pink JJ I-C rash NN I-C . . O William NNP O Arnold NNP O of IN O the DT O Seattle NNP O Post NNP O - HYPH O Intelligencer NNP O agreed VBD O that IN O the DT O film NN B-C 's POS I-C massive JJ I-C hype NN I-C caused VBD O many JJ O of IN O the DT O negative JJ B-C reactions NNS I-C , , O saying VBG O " `` O it PRP O built VBD O expectations NNS O that WDT O ca MD O n't RB O possibly RB O be VB O matched VBN O and CC O scuttled VBN O [ -LRB- O the DT O ] -RRB- O element NN O of IN O storytelling NN O surprise NN O . . O " '' O The DT O biggest JJS O stress NN B-C factor NN O for IN O students NNS O is VBZ O exams NNS B-C and CC O doing VBG O well RB O on IN O them PRP O . . O The DT O causes NNS O of IN O TLE NN B-C include VBP O mesial JJ B-C temporal JJ I-C sclerosis NN I-C , , O traumatic JJ B-C brain NN I-C injury NN I-C , , O brain NN B-C infections NNS I-C , , O such JJ O as IN O encephalitis NN B-C and CC O meningitis NN B-C , , O hypoxic JJ B-C brain NN I-C injury NN I-C , , O stroke NN B-C , , O cerebral JJ B-C tumours NNS I-C , , O and CC O genetic JJ B-C syndromes NNS I-C . . O Some DT O firefighters NNS B-C suffered VBD I-C beta NN I-C burns NNS I-C of IN O lungs NNS O and CC O nasopharyngeal JJ O region NN O after IN O inhalation NN B-C of IN I-C massive JJ I-C amounts NNS I-C of IN I-C radioactive JJ I-C smoke NN I-C . . O Mutations NNS B-C in IN I-C the DT I-C ATL1 NN I-C gene NN I-C are VBP O also RB O a DT O common JJ O cause NN O of IN O early JJ B-C - HYPH I-C onset JJ I-C hereditary JJ I-C spastic JJ I-C paraplegia NN I-C ( -LRB- I-C HSP NN I-C ) -RRB- I-C in IN I-C humans NNS I-C . . O As IN O the DT O penetration NN O continues VBZ O through IN O the DT O armor NN O , , O the DT O width NN B-C of IN I-C the DT I-C hole NN I-C decreases VBZ I-C leading VBG O to IN O a DT O characteristic JJ B-C fist NN I-C to IN I-C finger NN I-C penetration NN I-C , , O where WRB O the DT O size NN O of IN O the DT O eventual JJ O finger NN O is VBZ O based VBN O on IN O the DT O size NN O of IN O the DT O original JJ O fist NN O . . O Possibly RB B-C damage NN I-C to IN I-C the DT I-C lateral JJ I-C sulcus NN I-C also RB O known VBN O as IN O Sylvian NNP O fissure NN O may MD O contribute VB O to IN O an DT O individual NN B-C ’s POS I-C deterioration NN I-C of IN I-C object NN I-C recognition NN I-C . . O However RB O , , O some DT O cassette NN O tape NN O formulations NNS O do VBP O suffer VB O from IN O a DT O similar JJ O problem NN B-C caused VBN O by IN O fatty NN B-C acids NNS I-C working VBG I-C to IN I-C the DT I-C surface NN I-C of IN I-C the DT I-C tape NN I-C that WDT O can MD O cause VB O sticking VBG B-C to IN I-C heads NNS I-C and CC I-C guides NNS I-C and CC O severe JJ B-C modulation NN I-C of IN I-C signals NNS I-C through IN O the DT O playback NN O head NN O until IN O it PRP O is VBZ O cleaned VBN O . . O Patients NNS O with IN O Weaver NNP B-C syndrome NN I-C typically RB O have VBP O mild JJ B-C intellectual JJ I-C disability NN I-C with IN O poor JJ O coordination NN O and CC O balance NN O . . O December NNP O was VBD O unusually RB O cold JJ O so RB O frostbite NN O was VBD O common JJ O among IN O the DT O captives NNS O ; , O death NN B-C was VBD O caused VBN O by IN O exposure NN B-C or CC O starvation NN B-C . . O A DT O non-occupational JJ O form NN O of IN O silicosis NN B-C has VBZ O been VBN O described VBN O that WDT O is VBZ O caused VBN O by IN O long JJ B-C - HYPH I-C term NN I-C exposure NN I-C to IN I-C sand VB I-C dust NN I-C in IN O desert NN O areas NNS O , , O with IN O cases NNS O reported VBN O from IN O the DT O Sahara NNP O , , O Libyan JJ O desert NN O and CC O the DT O Negev NNP O . . O Most JJS O bites NNS B-C are VBP O caused VBN O by IN O female JJ B-C spiders NNS I-C ; , O the DT O male JJ B-C katipo NN I-C was VBD O considered VBN O too RB O small JJ O to TO O cause VB O systemic JJ B-C envenoming NN I-C in IN I-C humans NNS I-C . . O The DT O symptoms NNS O of IN O this DT O breakdown NN B-C can MD O be VB O immediately RB O obvious JJ O even RB O when WRB O rewinding VBG O the DT O tape NN O : : O tearing VBG B-C sounds NNS I-C and CC O sluggish JJ B-C behavior NN I-C . . O Cavity NN B-C magnetrons NNS I-C can MD O be VB O improperly RB O used VBN O to TO O create VB O surface NN O and CC O internal JJ O burning NN B-C . . O Another DT O cause NN O of IN O structural JJ B-C violence NN I-C is VBZ O the DT O impact NN B-C of IN I-C post-colonialism JJ I-C . . O Inflammation NN O – HYPH O neutrophilic JJ B-C invasion NN I-C is VBZ O triggered VBN O by IN O the DT O necrotic JJ B-C changes NNS I-C and CC O presence NN B-C of IN I-C cellular JJ I-C debris NN I-C within IN I-C the DT I-C lobules NNS I-C . . O After IN O the DT O insulin NN O injection NN O patients NNS O would MD O experience VB O various JJ O symptoms NNS O of IN O decreased VBN B-C blood NN I-C glucose NN I-C : : O flushing NN B-C , , O pallor NN B-C , , O perspiration NN B-C , , O salivation NN B-C , , O drowsiness NN B-C or CC O restlessness NN B-C . . O Avoidance NN O of IN O antitoxins NNS B-C that WDT O may MD O cause VB O serum NN B-C sickness NN I-C is VBZ O the DT O best JJS O way NN O to TO O prevent VB O serum NN O sickness NN O . . O High JJ B-C doses NNS I-C of IN I-C radiation NN I-C can MD O cause VB O rapid JJ B-C browning NN I-C of IN I-C skin NN I-C , , O known VBN O as IN O " `` O nuclear JJ O tan NN O " '' O . . O Burning VBG B-C plant NN I-C material NN I-C is VBZ O commonly RB O said VBN O to TO O create VB O urushiol JJ B-C - HYPH I-C laden NN I-C smoke NN I-C that WDT O causes VBZ O a DT O systemic JJ B-C reaction NN I-C , , O as RB O well RB O as IN O a DT O rash NN B-C in IN I-C the DT I-C throat NN I-C and CC I-C eyes NNS I-C . . O Drinking NN B-C alcohol NN I-C also RB O increases VBZ O the DT O body NN B-C fat JJ I-C percentage NN O . . O As IN O a DT O result NN O , , O stereotypes NNS B-C can MD O lead VB O to IN O discrimination NN B-C in IN I-C labor NN I-C markets NNS I-C and CC O other JJ O domains NNS O . . O Since IN O then RB O , , O the DT O penetration NN B-C of IN I-C HEAT NN I-C rounds NNS I-C relative JJ I-C to IN I-C projectile NN I-C diameters NNS I-C has VBZ I-C steadily RB I-C increased VBN I-C as IN O a DT O result NN O of IN O improved VBN B-C liner NN I-C material NN I-C and CC O metal NN B-C jet NN I-C performance NN I-C . . O Signs NNS O and CC O symptoms NNS O of IN O alcoholic JJ B-C hepatitis NN I-C include VBP O jaundice NN B-C , , O ascites NNS B-C ( -LRB- O fluid NN O accumulation NN O in IN O the DT O abdominal JJ O cavity NN O ) -RRB- O , , O fatigue NN B-C and CC O hepatic JJ B-C encephalopathy NN I-C ( -LRB- O brain NN B-C dysfunction NN I-C due IN O to IN O liver NN B-C failure NN I-C ) -RRB- O . . O BFS NN B-C can MD O also RB O be VB O attributed VBN O to IN O long JJ B-C term NN I-C use NN I-C of IN I-C anticholinergics NNS I-C such JJ O as IN O diphenhydramine NN B-C and CC O opiates NNS B-C such JJ O as IN O morphine NN B-C , , O but CC O the DT O latter JJ O case NN O is VBZ O usually RB O when WRB O withdrawal NN O symptoms NNS O are VBP O present JJ O . . O The DT O disorder NN B-C results NNS O from IN O low JJ B-C levels NNS I-C of IN I-C the DT I-C enzyme NN I-C responsible JJ O for IN O the DT O fifth JJ O step NN O in IN O heme NN O production NN O . . O The DT O beta NN O activity NN O was VBD O extremely RB O high JJ O , , O with IN O beta NN O / , O gamma JJ O ratio NN O reaching VBG O 10–30 CD O and CC O beta NN B-C energy NN I-C high JJ O enough JJ O to TO O damage VB B-C basal JJ I-C layer NN I-C of IN I-C the DT I-C skin NN I-C , , O resulting VBG O in IN O large JJ B-C area NN I-C portals NNS I-C for IN I-C infections NNS I-C , , O exacerbated VBN O by IN O damage NN B-C to IN I-C bone NN I-C marrow JJ I-C and CC O weakened VBN B-C immune JJ I-C system NN I-C . . O HSAN NNP B-C IA NNP I-C is VBZ O associated VBN O with IN O heterozygous JJ B-C missense NN I-C mutations NNS I-C in IN I-C the DT I-C SPTLC1 NN I-C gene NN I-C . . O The DT O term NN O " `` O brain NN O - HYPH O eating NN O amoeba NN O " '' O has VBZ O also RB O been VBN O applied VBN O to IN O Balamuthia NNP O mandrillaris NN O , , O causing VBG O some DT O confusion NN O between IN O the DT O two CD O ; , O Balamuthia NNP B-C mandrillaris NN I-C is VBZ O unrelated JJ O to IN O Naegleria NNP O fowleri NN O , , O however RB O , , O and CC O causes VBZ O a DT O different JJ O disease NN O called VBN O granulomatous JJ B-C amoebic JJ I-C encephalitis NN I-C . . O Ideational JJ O apraxia NN O is VBZ O a DT O condition NN O in IN O which WDT O an DT O individual NN O is VBZ O unable JJ B-C to TO I-C plan VB I-C movements NNS I-C related JJ O to IN O interaction NN O with IN O objects NNS O , , O because IN O he PRP O has VBZ O lost VBN B-C the DT I-C perception NN I-C of IN I-C the DT I-C object NN I-C 's POS I-C purpose NN I-C . . O In IN O the DT O 20 CD O % NN O of IN O cases NNS O where WRB O porphyria JJ O cutanea NN O tarda NN O is VBZ O inherited VBN O , , O it PRP O is VBZ O inherited VBN O in IN O an DT O autosomal JJ O dominant JJ O pattern NN O , , O which WDT O means VBZ O one CD O copy NN O of IN O the DT O altered VBN B-C gene NN I-C is VBZ O sufficient JJ O to TO O decrease VB B-C enzyme NN I-C activity NN I-C and CC O cause VBP O the DT O signs NNS B-C and CC I-C symptoms NNS I-C of IN I-C the DT I-C disorder NN I-C . . O A DT O lack NN B-C of IN I-C ketones NNS I-C leads VBZ O to IN O hypoglycemia NN B-C , , O and CC O compounds NNS O called VBN O organic NN B-C acids NNS I-C ( -LRB- O which WDT O are VBP O formed VBN O as IN O products NNS O of IN O amino NN O acid NN O and CC O fat JJ O breakdown NN O ) -RRB- O can MD O cause VB O the DT O blood NN B-C to TO I-C become VB I-C too RB I-C acidic JJ I-C . . O Delayed VBN O pressure NN O urticaria NN O is VBZ O known VBN O as IN O one CD O of IN O the DT O more RBR O painful JJ O subsets NNS O of IN O physical JJ B-C urticaria NN I-C due IN O to IN O formed VBN B-C hives NNS I-C being VBG I-C deep JJ I-C - HYPH I-C seated VBN I-C and CC O appearing VBG O after IN O 4 CD O – SYM O 6 CD O hours NNS O . . O Rarely RB O , , O the DT O bile NN B-C ducts NNS I-C are VBP I-C deprived VBN I-C of IN I-C blood NN I-C , , O leading VBG O to IN O severe JJ B-C cholangitis NN I-C ( -LRB- O inflammation NN O of IN O the DT O bile NN O ducts NNS O ) -RRB- O . . O Radiographically RB O , , O chronic JJ B-C simple JJ I-C silicosis NN I-C reveals VBZ O a DT O profusion NN O of IN O small JJ O ( -LRB- O < -LRB- O 10 CD O mm NN O in IN O diameter NN O ) -RRB- O opacities NNS B-C , , O typically RB O rounded VBN O , , O and CC O predominating VBG O in IN O the DT O upper JJ O lung NN O zones NNS O . . O Accelerated VBN O silicosis NN O : : O Silicosis NN O that WDT O develops VBZ O 5 CD O – SYM O 10 CD O years NNS O after IN O first JJ O exposure NN O to IN O higher JJR O concentrations NNS O of IN O silica NN O dust NN O . . O It PRP O has VBZ O been VBN O shown VBN O that IN O it PRP O is VBZ O theoretically RB O possible JJ O to TO O generate VB O seizures NNS B-C in IN O the DT O neural JJ O networks NNS O due IN O to IN O down-regulation NN B-C of IN I-C KCC2 NNP I-C , , O consistent JJ O with IN O the DT O chloride NN O measurements NNS O during IN O the DT O transition NN O to IN O seizure NN O and CC O KCC2 NNP O blockade NN O experiments NNS O . . O This DT O is VBZ O a DT O rare JJ O condition NN O in IN O which WDT O blood NN B-C clots NNS I-C occur VBP I-C in IN I-C the DT I-C iliofemoral JJ I-C vein NN I-C due IN O to IN O compression NN B-C of IN I-C the DT I-C blood NN I-C vessels NNS I-C in IN I-C the DT I-C leg NN I-C . . O For IN O instance NN O , , O if IN O the DT O dendrites NNS B-C of IN I-C granule NN I-C cells NNS I-C reconnect VBP I-C , , O it PRP O may MD O be VB O in IN O a DT O way NN O ( -LRB- O through IN O the DT O laminar JJ O planes NNS O ) -RRB- O that WDT O allows VBZ O hyperexcitability NN B-C . . O Poison NN O ivy NN O and CC O poison NN O oak NN O are VBP O still RB O harmful JJ O when WRB O the DT O leaves NNS O have VBP O fallen VBN O off RP O , , O as IN O the DT O toxic JJ O residue NN O is VBZ O persistent JJ O , , O and CC O exposure NN B-C to IN I-C any DT I-C parts NNS I-C of IN I-C plants NNS I-C containing VBG I-C urushiol NN I-C can MD O cause VB O a DT O rash NN B-C at IN O any DT O time NN O of IN O the DT O year NN O . . O Induced VBN O seismicity NN O refers VBZ O to IN O typically RB O minor JJ O earthquakes NNS B-C and CC O tremors NNS B-C that WDT O are VBP O caused VBN O by IN O human JJ B-C activity NN I-C that WDT O alters VBZ O the DT O stresses NNS O and CC O strains NNS O on IN O the DT O Earth NNP O 's POS O crust NN O . . O A DT O " `` O contributing VBG O factor NN O " '' O to IN O the DT O accident NN B-C was VBD O a DT O long JJ B-C discussion NN I-C with IN I-C the DT I-C Protocol NNP I-C director NN I-C and CC I-C the DT I-C crew NN I-C of IN I-C the DT I-C Yak NNP I-C - HYPH O 40 CD O regarding VBG O the DT O actual JJ O weather NN O and CC O the DT O impossibility NN O of IN O landing VBG O at IN O Smolensk NNP O in IN O such JJ O weather NN O conditions NNS O . . O One CD O review NN O of IN O a DT O collection NN O of IN O PCT NNP O studies NNS O noted VBD O Hepatitis NN B-C C NN I-C infection NN I-C in IN O 50 CD O % NN O of IN O documented VBN O cases NNS O of IN O PCT NNP B-C . . O Masaki NNP O Ichinose NNP O , , O head NN O of IN O the DT O University NNP O of IN O Tokyo NNP O 's POS O Institute NNP O of IN O Death NNP O and CC O Life NNP O Studies NNPS O , , O hypothesizes VBZ O that IN O the DT O increase NN B-C in IN I-C kodokushi NN I-C is VBZ O linked VBN O to IN O Japan NNP B-C 's POS I-C contemporary JJ I-C culture NN I-C which WDT O ignores VBZ O death NN O . . O HSAN RB B-C I PRP I-C is VBZ O characterized VBN O by IN O marked VBN B-C sensory JJ I-C disturbances NNS I-C mainly RB O as IN O the DT O loss NN B-C of IN I-C pain NN I-C and CC I-C temperature NN I-C sensation NN I-C in IN I-C the DT I-C distal JJ I-C parts NNS I-C of IN I-C the DT I-C lower JJR I-C limbs NNS I-C . . O Potomac NNP B-C Horse NNP I-C Fever NNP I-C ( -LRB- O PHF NNP O ) -RRB- O is VBZ O a DT O potentially RB O - HYPH O fatal JJ O febrile JJ O illness NN O affecting VBG O horses NNS O caused VBN O by IN O the DT O intracellular JJ B-C bacterium NN I-C Neorickettsia NNP I-C risticii NN I-C . . O There EX O are VBP O three CD O mutations NNS B-C in IN O this DT O gene NN O associated VBN O with IN O JME NNP B-C , , O E177A NN B-C , , O R220C NN B-C , , O and CC O R220H NN B-C , , O all RB O located VBN O in IN O the DT O N-terminus NN O of IN O the DT O protein NN O . . O