Some DT O
degree NN O
of IN O
cyclical JJ B-C
breast NN I-C
tenderness NN I-C
is VBZ O
normal JJ O
in IN O
the DT O
menstrual NN O
cycle NN O
, , O
and CC O
is VBZ O
usually RB O
associated VBN O
with IN O
menstruation NN B-C
and CC O
/ , O
or CC O
premenstrual JJ B-C
syndrome NN I-C
( -LRB- O
PMS NN O
) -RRB- O
. . O

Larvae NNS B-C
tend VBP O
to TO O
migrate VB O
to IN O
the DT O
brain NN O
and CC O
other JJ O
vital JJ O
organs NNS O
, , O
cause VB O
damage NN B-C
, , O
and CC O
affect VB O
the DT O
behavior NN O
of IN O
the DT O
intermediate JJ O
host NN O
, , O
making VBG O
it PRP O
an DT O
easier JJR O
prey NN O
for IN O
pandas NNS O
if IN O
they PRP O
needed VBD O
to TO O
eat VB O
meat NN O
. . O

Rapid JJ B-C
changes NNS I-C
in IN I-C
uric NN I-C
acid NN I-C
may MD O
occur VB O
due IN O
to IN O
factors NNS O
including VBG O
trauma NN B-C
, , O
surgery NN B-C
, , O
chemotherapy NN B-C
, , O
diuretics NNS B-C
, , O
and CC O
stopping NN B-C
or CC I-C
starting VBG I-C
allopurinol NN I-C
. . O

The DT O
" `` O
Twinkie NN O
" '' O
defense NN O
was VBD O
presented VBN O
by IN O
a DT O
psychiatrist NN O
to IN O
the DT O
jury NN O
, , O
stating VBG O
that IN O
White NNP O
had VBD O
a DT O
diminished VBN B-C
capacity NN I-C
due IN O
to IN O
depression NN B-C
. . O

This DT O
significant JJ B-C
change NN I-C
in IN I-C
stress NN I-C
can MD O
lead VB O
to IN O
sudden JJ B-C
movement NN I-C
along IN I-C
the DT I-C
fault NN I-C
or CC I-C
fracture NN I-C
, , O
resulting VBG O
in IN O
an DT O
earthquake NN B-C
. . O

Vivanco NNP O
said VBD O
that IN O
" `` O
[ -LRB- O
the DT O
] -RRB- O
horrendous JJ B-C
tragedy NN I-C
[ -LRB- O
was VBD O
] -RRB- O
the DT O
result NN O
of IN O
prison JJ B-C
conditions NNS I-C
that WDT O
are VBP O
symptomatic JJ O
of IN O
the DT O
country NN O
’s POS O
larger JJR O
public JJ O
security NN O
crisis NN O
. . O
" '' O

Damping VBG B-C
off RP I-C
( -LRB- O
or CC O
damping NN O
- HYPH O
off NN O
) -RRB- O
is VBZ O
a DT O
horticultural JJ O
disease NN O
or CC O
condition NN O
, , O
caused VBN O
by IN O
several JJ B-C
different JJ I-C
pathogens NNS I-C
that WDT O
kill VBP O
or CC O
weaken VB O
seeds NNS O
or CC O
seedlings NNS O
before RB O
or CC O
after IN O
they PRP O
germinate VBP O
. . O

HSAN NN B-C
ID NN I-C
is VBZ O
caused VBN O
by IN O
heterozygous JJ B-C
missense NN I-C
mutations NNS I-C
in IN I-C
the DT I-C
ATL1 NN I-C
gene NN I-C
which WDT O
encodes VBZ O
atlastin NN O
- HYPH O
1 CD O
. . O

Induced VBN B-C
seismicity NN I-C
in IN I-C
Basel NNP I-C
led VBD O
to IN O
suspension NN B-C
of IN I-C
its PRP$ I-C
HDR NN I-C
project NN I-C
. . O

Large JJ B-C
- HYPH I-C
scale NN I-C
fossil NN I-C
fuel NN I-C
extraction NN I-C
can MD O
generate VB O
earthquakes NNS B-C
. . O

Type NN O
3 CD O
: : O
A DT O
mild JJ O
form NN O
of IN O
Dupuytren NNP B-C
's POS I-C
which WDT O
is VBZ O
common JJ O
among IN O
diabetics NNS O
or CC O
which WDT O
may MD O
also RB O
be VB O
caused VBN O
by IN O
certain JJ B-C
medications NNS I-C
such JJ O
as IN O
the DT O
anti-convulsants NNS B-C
taken VBN O
by IN O
people NNS O
with IN O
epilepsy NN O
. . O

Alcohol NN B-C
decreases VBZ B-C
the DT I-C
levels NNS I-C
of IN I-C
this DT I-C
vitamin NN I-C
and CC O
increases VBZ B-C
toxicity NN I-C
when WRB O
alcohol NN O
is VBZ O
consumed VBN O
in IN O
large JJ O
amounts NNS O
. . O

Signs NNS O
and CC O
symptoms NNS O
of IN O
PHF NN B-C
include VBP O
acute JJ B-C
- HYPH I-C
onset JJ I-C
fever NN I-C
, , O
depression NN B-C
( -LRB- O
sometimes RB O
profound JJ O
) -RRB- O
, , O
inappetence NN B-C
, , O
mild JJ B-C
colic NN I-C
- , I-C
like IN I-C
symptoms NNS I-C
, , O
decreased VBN B-C
manure NN I-C
production NN I-C
, , O
profuse JJ B-C
watery JJ I-C
non-fetid JJ I-C
diarrhea NN I-C
endotoxemia NN I-C
, , O
edema NN B-C
due IN O
to IN O
protein NN B-C
imbalances NNS I-C
, , O
abortion NN B-C
by IN I-C
pregnant JJ I-C
mares NNS I-C
, , O
and CC O
acute JJ B-C
laminitis NN I-C
( -LRB- O
20 CD O
to IN O
40 CD O
percent NN O
of IN O
cases NNS O
) -RRB- O
. . O

Very RB B-C
large JJ I-C
AVMs NNS I-C
cause VBP O
a DT O
marked VBN B-C
inability NN I-C
to TO I-C
absorb VB I-C
oxygen NN I-C
, , O
which WDT O
may MD O
be VB O
noted VBN O
by IN O
cyanosis NN B-C
( -LRB- O
bluish JJ O
discoloration NN O
of IN O
the DT O
lips NNS O
and CC O
skin NN O
) -RRB- O
, , O
clubbing NN B-C
of IN I-C
the DT I-C
fingernails NNS I-C
( -LRB- O
often RB O
encountered VBN O
in IN O
chronically RB O
low JJ O
oxygen NN O
levels NNS O
) -RRB- O
, , O
and CC O
a DT O
humming NN B-C
noise NN I-C
over IN I-C
the DT I-C
affected VBN I-C
part NN I-C
of IN I-C
the DT I-C
lung NN I-C
detectable JJ O
by IN O
stethoscope NN O
. . O

Since IN O
BFS NNP B-C
appears VBZ O
to TO O
cause VB O
no DT O
actual JJ O
nerve NN B-C
damage NN I-C
( -LRB- O
at RB O
least RBS O
as IN O
seen VBN O
on IN O
the DT O
EMG NN O
) -RRB- O
, , O
patients NNS O
will MD O
likely RB O
exhibit VB O
a DT O
completely RB O
normal JJ O
EMG NN O
( -LRB- O
or CC O
one CD O
where WRB O
the DT O
only JJ O
abnormality NN O
seen VBN O
is VBZ O
fasciculations NNS O
) -RRB- O
. . O

Though IN O
infants NNS B-C
, , I-C
children NNS I-C
, , I-C
and CC I-C
adolescents NNS I-C
suffer VBP I-C
more JJR I-C
from IN I-C
malnutrition NN I-C
because IN O
of IN O
their PRP$ O
need NN B-C
for IN I-C
critical JJ I-C
nutrients NNS I-C
for IN O
their PRP$ O
normal JJ O
development NN O
, , O
older JJR B-C
people NNS I-C
may MD I-C
have VB I-C
problems NNS I-C
because IN O
of IN O
aging NN B-C
or CC O
illness NN B-C
. . O

This DT O
enzymatic JJ B-C
conversion NN I-C
results NNS O
in IN O
coproporphyrinogen NN B-C
III CD I-C
as IN O
the DT O
primary JJ O
product NN O
. . O

The DT O
report NN O
suggested VBD O
that IN O
air NN B-C
pollution NN I-C
regulations NNS O
can MD O
have VB O
large JJ O
amplifying NN O
effects NNS O
on IN O
global JJ B-C
warming NN I-C
. . O

The DT O
near JJ B-C
death NN I-C
of IN I-C
a DT I-C
mother NN I-C
or CC I-C
baby NN I-C
, , O
heavy JJ B-C
bleeding NN I-C
, , O
and CC O
emergency NN B-C
operations NNS I-C
are VBP O
examples NNS O
of IN O
situations NNS O
that WDT O
can MD O
cause VB O
psychological JJ B-C
trauma NN I-C
. . O

The DT O
age NN B-C
- HYPH O
related VBN O
Type NN B-C
II CD I-C
fiber NN I-C
loss NN I-C
highlights VBZ O
the DT O
negative JJ O
effect NN O
of IN O
aging NN O
on IN O
muscle NN O
power NN O
. . O

Lung NN B-C
transplantation NN I-C
to TO O
replace VB O
the DT O
damaged VBN O
lung NN O
tissue NN O
is VBZ O
the DT O
most RBS O
effective JJ O
treatment NN O
, , O
but CC O
is VBZ O
associated VBN O
with IN O
severe JJ B-C
risks NNS I-C
of IN O
its PRP$ O
own JJ O
from IN O
the DT O
lung NN O
transplant JJ O
surgery NN O
as RB O
well RB O
as IN O
from IN O
consequences NNS O
of IN O
long JJ O
- HYPH O
term NN O
immunosuppression NN O
( -LRB- O
e.g. FW O
, , O
opportunistic JJ O
infections NNS O
) -RRB- O
. . O

Naked JJ B-C
crystals NNS I-C
may MD I-C
break VB I-C
out IN I-C
of IN I-C
walled VBN I-C
- HYPH I-C
off RP I-C
tophi NN I-C
due IN O
to IN O
minor JJ B-C
physical JJ I-C
damage NN I-C
to IN I-C
the DT I-C
joint JJ I-C
, , O
medical JJ B-C
or CC I-C
surgical JJ I-C
stress NN I-C
, , O
or CC O
rapid JJ B-C
changes NNS I-C
in IN I-C
uric NN I-C
acid NN I-C
levels NNS I-C
. . O

Generally RB O
, , O
the DT O
symptoms NNS O
and CC O
signs NNS O
that WDT O
lead VBP O
to IN O
the DT O
diagnosis NN O
of IN O
an DT O
immunodeficiency NN B-C
include VBP O
recurrent JJ B-C
or CC I-C
persistent JJ I-C
infections NNS I-C
or CC O
developmental JJ B-C
delay NN I-C
as IN O
a DT O
result NN O
of IN O
infection NN B-C
. . O

On IN O
26 CD O
May NNP O
2010 CD O
, , O
it PRP O
was VBD O
reported VBN O
that IN O
pilot NN B-C
error NN I-C
had VBD O
been VBN O
identified VBN O
as IN O
the DT O
reason NN O
for IN O
the DT O
crash NN B-C
. . O

Many JJ O
of IN O
these DT O
animals NNS O
act VBP O
as IN O
paratenic JJ O
hosts NNS O
and CC O
the DT O
infection NN B-C
results NNS O
in IN O
the DT O
penetration NN B-C
of IN I-C
the DT I-C
gut NN I-C
wall NN I-C
by IN I-C
the DT I-C
larvae NNS I-C
and CC O
subsequent JJ O
invasion NN B-C
of IN I-C
tissue NN I-C
, , O
resulting VBG O
in IN O
severe JJ B-C
disease NN I-C
. . O

While IN O
inherited VBN B-C
deficiencies NNS I-C
in IN I-C
uroporphyrinogen NN I-C
decarboxylase NN I-C
often RB O
lead VBP O
to IN O
the DT O
development NN O
of IN O
PCT NN B-C
, , O
there EX O
are VBP O
a DT O
number NN O
of IN O
risk NN O
factors NNS O
that WDT O
can MD O
both RB O
cause VB O
and CC O
exacerbate VB O
the DT O
symptoms NNS O
of IN O
this DT O
disease NN O
. . O

Bruising NN B-C
, , O
particularly RB O
purpura JJ O
in IN O
the DT O
forearms NNS O
and CC O
petechiae NN O
in IN O
the DT O
feet NNS O
, , O
legs NNS O
, , O
and CC O
mucous NN O
membranes NNS O
, , O
may MD O
be VB O
caused VBN O
by IN O
spontaneous JJ B-C
bleeding NN I-C
under IN I-C
the DT I-C
skin NN I-C
. . O

The DT O
impact NN B-C
of IN I-C
an DT I-C
SFF NNP I-C
normally RB O
causes VBZ O
a DT O
large JJ B-C
diameter NN I-C
, , I-C
but CC I-C
relatively RB I-C
shallow JJ I-C
hole NN I-C
( -LRB- O
relative JJ O
to IN O
a DT O
shaped VBN O
charge NN O
) -RRB- O
or CC O
, , O
at RB O
best RBS O
, , O
a DT O
few JJ O
CDs NNS O
. . O

After IN O
pregnancy NN O
, , O
breast NN B-C
pain NN I-C
can MD O
be VB O
caused VBN O
by IN O
breastfeeding NN B-C
. . O

There EX O
is VBZ O
a DT O
higher JJR B-C
prevalence NN I-C
of IN I-C
cholangiocarcinoma NN I-C
in IN I-C
Asia NNP I-C
, , O
which WDT O
has VBZ O
been VBN O
attributed VBN O
to IN O
endemic JJ B-C
chronic JJ I-C
parasitic JJ I-C
infestation NN I-C
. . O

In IN O
fruit NN O
fly NN O
( -LRB- O
Drosophila NN O
melanogaster NN O
) -RRB- O
, , O
atlastin NN B-C
- HYPH I-C
1 CD I-C
has VBZ O
been VBN O
shown VBN O
to TO O
induce VB O
tethering NN B-C
and CC I-C
fusion NN I-C
of IN I-C
membranes NNS I-C
adjacent JJ I-C
to IN I-C
the DT I-C
ER NN I-C
to TO O
help VB O
maintain VB O
the DT O
integrity NN O
of IN O
the DT O
ER NN O
. . O

In IN O
temporal JJ O
lobe NN O
epilepsy NN O
, , O
a DT O
focal JJ B-C
seizure NN I-C
usually RB O
causes VBZ O
abnormal JJ B-C
sensations NNS I-C
only RB O
. . O

About RB O
1500 CD B-C
genes NNS I-C
are VBP O
linked VBN O
to IN O
outcomes NNS B-C
in IN I-C
pancreatic JJ I-C
adenocarcinoma NN I-C
. . O

Another DT O
potential JJ O
contributor NN O
to IN O
dynapenia VB B-C
is VBZ O
a DT O
disruption NN B-C
of IN I-C
the DT I-C
process NN I-C
that WDT I-C
converts VBZ I-C
electrical JJ I-C
signal NN I-C
given VBN I-C
for IN I-C
muscular JJ I-C
activation NN I-C
into IN I-C
an DT I-C
actual JJ I-C
contraction NN I-C
. . O

Due IN O
to IN O
the DT O
presence NN B-C
of IN I-C
the DT I-C
chlorine NN I-C
atom NN I-C
, , O
the DT O
entire JJ B-C
complex NN I-C
is VBZ I-C
more JJR I-C
water NN I-C
soluble NN I-C
allowing VBG O
the DT O
kidneys NNS O
to TO O
preferentially RB O
remove VB O
it PRP O
from IN O
the DT O
blood NN O
stream NN O
and CC O
expel VB O
it PRP O
through IN O
urination NN O
. . O

Restated VBN O
, , O
radiation NN B-C
recall NN I-C
dermatitis NN I-C
is VBZ O
an DT O
inflammatory JJ O
skin NN O
reaction NN O
that WDT O
occurs VBZ O
in IN O
a DT O
previously RB O
irradiated VBN O
body NN O
part NN O
following VBG O
drug NN B-C
administration NN I-C
. . O

A DT O
katipo JJ B-C
bite NN I-C
produces VBZ O
the DT O
toxic JJ B-C
syndrome NN I-C
latrodectism NN I-C
; , O
symptoms NNS O
include VBP O
extreme JJ B-C
pain NN I-C
and CC O
, , O
potentially RB O
, , O
hypertension NN B-C
, , O
seizure NN B-C
, , O
or CC O
coma NN B-C
. . O

Finally RB O
, , O
due IN O
to IN O
the DT O
strong JJ O
association NN O
between IN O
PCT NN B-C
and CC O
Hepatitis NN B-C
C NN I-C
, , O
the DT O
treatment NN O
of IN O
Hepatitis NN O
C NN O
( -LRB- O
if IN O
present JJ O
) -RRB- O
is VBZ O
vital JJ O
to IN O
the DT O
effective JJ O
treatment NN O
of IN O
PCT NN O
. . O

The DT O
reason NN O
for IN O
the DT O
ineffectiveness NN B-C
of IN I-C
HEAT NN I-C
munitions NNS I-C
against IN O
modern JJ O
main JJ O
battle NN O
tanks NNS O
can MD O
be VB O
attributed VBN O
in IN O
part NN O
to IN O
the DT O
use NN O
of IN O
new JJ B-C
types NNS I-C
of IN I-C
armor NN I-C
. . O

Cyclical JJ B-C
breast NN I-C
pain NN I-C
( -LRB- O
cyclical JJ O
mastalgia NN O
) -RRB- O
is VBZ O
often RB O
associated VBN O
with IN O
fibrocystic JJ B-C
breast NN I-C
changes NNS I-C
or CC O
duct NN B-C
ectasia NN I-C
and CC O
thought VBN O
to TO O
be VB O
caused VBN O
by IN O
changes NNS B-C
of IN I-C
prolactin NN I-C
response NN I-C
to IN I-C
thyrotropin NN I-C
. . O

Alcohol NNP B-C
has VBZ O
been VBN O
shown VBN O
to TO O
decrease VB B-C
the DT I-C
levels NNS I-C
of IN I-C
B12 NN I-C
. . O

The DT O
Thor NNP B-C
decision NN I-C
caused VBD O
publishers NNS B-C
and CC I-C
booksellers NNS I-C
to TO I-C
be VB I-C
much RB I-C
quicker JJR I-C
to TO I-C
destroy VB I-C
stocks NNS I-C
of IN I-C
poorly RB I-C
- HYPH I-C
selling NN I-C
books NNS I-C
in IN O
order NN O
to TO O
realize VB O
a DT O
taxable NN O
loss NN O
. . O

Alcoholic JJ B-C
hepatitis NN I-C
is VBZ O
characterized VBN O
by IN O
myriad JJ B-C
symptoms NNS I-C
, , O
which WDT O
may MD O
include VB O
feeling VBG B-C
unwell JJ I-C
, , O
enlargement NN B-C
of IN I-C
the DT I-C
liver NN I-C
, , O
development NN B-C
of IN I-C
fluid NN I-C
in IN I-C
the DT I-C
abdomen NN I-C
( -LRB- O
ascites NNS O
) -RRB- O
, , O
and CC O
modest JJ B-C
elevation NN I-C
of IN I-C
liver NN I-C
enzyme NN I-C
levels NNS I-C
( -LRB- O
as IN O
determined VBN O
by IN O
liver NN O
function NN O
tests NNS O
) -RRB- O
. . O

Particularly RB O
, , O
impairments NNS B-C
in IN I-C
the DT I-C
release NN I-C
of IN I-C
calcium NN I-C
( -LRB- O
Ca2 NN O
+ SYM O
) -RRB- O
from IN O
the DT O
sarcoplasmic NN O
reticulum NN O
have VBP O
been VBN O
suggested VBN O
to TO O
explain VB O
why WRB O
decreased VBN B-C
muscle NN I-C
quality NN I-C
is VBZ O
so RB O
prevalent JJ O
in IN O
older JJR O
adults NNS O
. . O

The DT O
disease NN O
is VBZ O
characterized VBN O
by IN O
progressive JJ B-C
stiffness NN I-C
and CC O
contraction NN B-C
( -LRB- I-C
spasticity NN I-C
) -RRB- I-C
in IN I-C
the DT I-C
lower JJR I-C
limbs NNS I-C
due IN O
to IN O
damage NN B-C
to IN I-C
or CC I-C
dysfunction NN I-C
of IN I-C
the DT I-C
nerves NNS I-C
. . O

Care NN B-C
packages NNS I-C
filled VBN O
with IN O
unhealthy JJ O
treats NNS O
, , O
sent VBN O
usually RB O
by IN O
parents NNS O
, , O
was VBD O
found VBN O
to TO O
be VB O
the DT O
leading VBG O
cause NN O
of IN O
weight NN B-C
gain NN I-C
. . O

Rectocele NNP B-C
can MD O
be VB O
a DT O
cause NN O
of IN O
symptoms NNS B-C
of IN I-C
obstructed VBN I-C
defecation NN I-C
. . O

In IN O
people NNS O
in IN O
their PRP$ O
undergraduate JJ O
years NNS O
of IN O
study NN O
at IN O
a DT O
four CD O
- HYPH O
year NN O
university NN O
, , O
malnutrition NN B-C
can MD O
occur VB O
due IN O
to IN O
negligence NN B-C
of IN I-C
eating VBG I-C
and CC O
even RB O
their PRP$ O
diet NN B-C
. . O

Induced VBN B-C
seismicity NN I-C
can MD O
also RB O
be VB O
caused VBN O
by IN O
the DT O
injection NN B-C
of IN I-C
carbon NN I-C
dioxide NN I-C
as IN O
the DT O
storage NN O
step NN O
of IN O
carbon NN O
capture NN O
and CC O
storage NN O
, , O
which WDT O
aims VBZ O
to TO O
sequester VB O
carbon NN O
dioxide NN O
captured VBN O
from IN O
fossil JJ O
fuel NN O
production NN O
or CC O
other JJ O
sources NNS O
in IN O
earth NN O
’s POS O
crust NN O
as IN O
a DT O
means NN O
of IN O
climate NN O
change NN O
mitigation NN O
. . O

Industrial JJ B-C
radiography JJ I-C
sources NNS I-C
are VBP O
a DT O
common JJ O
source NN O
of IN O
beta NN B-C
burns NNS I-C
in IN I-C
workers NNS I-C
. . O

People NNS O
may MD O
report VB O
pain NN B-C
, , O
aching VBG B-C
and CC O
itching VBG B-C
with IN O
the DT O
contractions NNS B-C
. . O

These DT O
receptors NNS B-C
initiate VBP O
the DT O
nuclear JJ B-C
response NN I-C
leading VBG O
to IN O
De NNP B-C
novo NNP I-C
synthesis NN I-C
of IN I-C
proteins NNS I-C
. . O

Sleep NN B-C
deprivation NN I-C
is VBZ O
a DT O
major JJ O
factor NN O
in IN O
triggering VBG O
GTCs NNS B-C
. . O

James NNP O
Berardinelli NNP O
wrote VBD O
" `` O
The DT O
Phantom NNP O
Menace NNP O
was VBD O
probably RB O
the DT O
most RBS O
overhyped JJ O
motion NN O
picture NN O
of IN O
the DT O
last JJ O
decade NN O
( -LRB- O
if IN O
not RB O
longer RBR O
) -RRB- O
, , O
and CC O
its PRP$ O
reputation NN B-C
suffered VBD I-C
as IN O
a DT O
result NN O
of IN O
its PRP$ O
inability NN B-C
to TO I-C
satisfy VB I-C
unreasonable JJ I-C
expectations NNS I-C
. . O
" '' O

AGEP NNP B-C
is VBZ O
characterized VBN O
by IN O
sudden JJ B-C
skin NN I-C
eruptions NNS I-C
that WDT O
appear VBP O
on IN O
average JJ O
five CD O
days NNS O
after IN O
a DT O
medication NN O
is VBZ O
started VBN O
. . O

Binge NN B-C
- HYPH I-C
eating NN I-C
disorder NN I-C
, , O
as IN O
the DT O
name NN O
implies VBZ O
, , O
is VBZ O
characterized VBN O
by IN O
uncontrollable JJ B-C
, , I-C
excessive JJ I-C
eating NN I-C
, , O
followed VBN O
by IN O
feelings NNS B-C
of IN I-C
shame NN I-C
and CC I-C
guilt NN I-C
. . O

Clusters NNS B-C
of IN I-C
myoclonic JJ I-C
seizures NNS I-C
can MD O
lead VB O
to IN O
absence NN B-C
seizures NNS I-C
, , O
and CC O
clusters NNS B-C
of IN I-C
absence NN I-C
seizures NNS I-C
can MD O
lead VB O
to IN O
generalized VBN B-C
tonic NN I-C
- HYPH I-C
clonic JJ I-C
seizures NNS I-C
. . O

There EX O
are VBP O
some DT O
data NN O
revealing VBG O
an DT O
association NN O
between IN O
silicosis NN B-C
and CC O
certain JJ O
autoimmune JJ B-C
diseases NNS I-C
, , O
including VBG O
nephritis NN B-C
, , O
scleroderma NN B-C
, , O
and CC O
systemic JJ B-C
lupus JJ I-C
erythematosus NN I-C
, , O
especially RB O
in IN O
acute JJ O
or CC O
accelerated VBN O
silicosis NN O
. . O

When WRB O
the DT O
surface NN O
high JJ O
moves VBZ O
offshore NN O
, , O
the DT O
precipitation NN B-C
itself PRP O
can MD O
cause VB O
the DT O
CAD NN B-C
event NN I-C
. . O

Deletions NNS B-C
and CC I-C
mutations NNS I-C
in IN I-C
the DT I-C
NSD1 NN I-C
gene NN I-C
is VBZ O
a DT O
common JJ O
cause NN O
for IN O
patients NNS O
with IN O
Sotos NNP B-C
syndrome NN I-C
and CC O
in IN O
some DT O
cases NNS O
for IN O
Weaver NNP B-C
syndrome NN I-C
as RB O
well RB O
. . O

Consumption NN B-C
of IN I-C
alcohol NN I-C
can MD O
cause VB O
a DT O
loss NN B-C
of IN I-C
calcium NN I-C
through IN O
urinary JJ O
excretion NN O
. . O

In IN O
the DT O
brain NN O
, , O
AVMs NNS B-C
occasionally RB I-C
exert VBP I-C
pressure NN I-C
, , O
leading VBG O
to IN O
headaches NNS B-C
. . O

Results NNS O
of IN O
ongoing JJ O
multi CD O
- HYPH O
year NN O
research NN O
on IN O
induced VBN O
earthquakes NNS O
by IN O
the DT O
United NNP O
States NNP O
Geological NNP O
Survey NNP O
( -LRB- O
USGS NNP O
) -RRB- O
published VBD O
in IN O
2015 CD O
suggested VBN O
that IN O
most JJS O
of IN O
the DT O
significant JJ B-C
earthquakes NNS I-C
in IN I-C
Oklahoma NNP I-C
, , O
such JJ O
as IN O
the DT O
1952 CD B-C
magnitude NN I-C
5.7 CD I-C
El NNP I-C
Reno NNP I-C
earthquake NN I-C
may MD O
have VB O
been VBN O
induced VBN O
by IN O
deep JJ B-C
injection NN I-C
of IN I-C
waste NN I-C
water NN I-C
by IN O
the DT O
oil NN O
industry NN O
. . O

As RB O
many JJ O
as IN O
95 CD O
% NN O
of IN O
patients NNS O
treated VBN O
with IN O
radiation NN B-C
therapy NN I-C
for IN O
cancer NN O
will MD O
experience VB O
a DT O
skin NN B-C
reaction NN I-C
. . O

Reported VBN B-C
Weakness NN I-C
- HYPH O
Unintentional JJ B-C
weight NN I-C
loss NN I-C
, , O
muscle NN B-C
weakness NN I-C
measured VBN O
by IN O
reduced VBN O
grip NN O
strength NN O
, , O
physical JJ O
slowness NN O
, , O
and CC O
poor JJ O
endurance NN O
are VBP O
all DT O
symptoms NNS O
of IN O
muscle NN O
weakness NN O
that WDT O
put VBD O
individuals NNS O
at IN O
a DT O
high JJ O
risk NN O
of IN O
dynapenia NN B-C
. . O

Recently RB O
, , O
silicosis NN O
in IN O
Turkish JJ O
denim NN O
sandblasters NNS O
was VBD O
detected VBN O
as IN O
a DT O
new JJ O
cause NN O
of IN O
silicosis NN B-C
due IN O
to IN O
recurring VBG B-C
, , I-C
poor JJ I-C
working NN I-C
conditions NNS I-C
. . O

A DT O
systematic JJ O
review NN O
on IN O
the DT O
literature NN O
regarding VBG O
the DT O
relationship NN O
between IN O
type NN O
II CD O
muscle NN O
fiber NN O
loss NN O
and CC O
aging NN O
found VBD O
that IN O
the DT O
total JJ B-C
number NN I-C
of IN I-C
fibers NNS I-C
in IN I-C
the DT I-C
vastus JJ I-C
lateralis NN I-C
decreases VBZ I-C
tremendously RB O
with IN O
age NN B-C
. . O

The DT O
rare JJ O
inherited VBN O
disorders NNS O
Lynch NNP B-C
syndrome NN I-C
II CD I-C
and CC O
biliary JJ B-C
papillomatosis NN I-C
have VBP O
also RB O
been VBN O
found VBN O
to TO O
be VB O
associated VBN O
with IN O
cholangiocarcinoma NN B-C
. . O

Alcoholic JJ O
hepatitis NN O
is VBZ O
distinct JJ O
from IN O
cirrhosis NN B-C
caused VBN O
by IN O
long JJ B-C
- HYPH I-C
term NN I-C
alcohol NN I-C
consumption NN I-C
. . O

Inhalation NN B-C
of IN I-C
beta NN I-C
radioactive JJ I-C
isotopes NNS I-C
may MD O
cause VB O
beta NN B-C
burns NNS I-C
of IN O
lungs NNS O
and CC O
nasopharyngeal JJ O
region NN O
, , O
ingestion NN B-C
may MD O
lead VB O
to IN O
burns NNS B-C
of IN I-C
gastrointestinal JJ I-C
tract NN I-C
; , O
the DT O
latter NN O
being VBG O
a DT O
risk NN O
especially RB O
for IN O
grazing NN O
animals NNS O
. . O

The DT O
catastrophic JJ B-C
fire NN I-C
caused VBD O
the DT O
loss NN B-C
of IN I-C
human JJ I-C
life NN I-C
, , O
cattle NNS O
, , O
and CC O
land NN O
for IN O
miles NNS O
and CC O
affected VBN O
many JJ O
regions NNS O
including VBG O
Portland NNP O
, , O
Plenty NNP O
Ranges NNPS O
, , O
Western NNP O
Port NNP O
, , O
the DT O
Wimmera NNP O
and CC O
Dandenong NNP O
districts NNS O
, , O
Gippsland NNP O
, , O
and CC O
Mount NNP O
Macedon NNP O
. . O

Elevated VBN B-C
levels NNS I-C
of IN I-C
uric NN I-C
acid NN I-C
may MD O
also RB O
lead VB O
to IN O
crystals NNS B-C
precipitating VBG I-C
in IN I-C
the DT I-C
kidneys NNS I-C
, , O
resulting VBG O
in IN O
stone NN B-C
formation NN I-C
and CC O
subsequent JJ O
urate NN B-C
nephropathy NN I-C
. . O

Slight JJ O
weakness NN O
in IN O
a DT O
limb NN O
may MD O
be VB O
tolerated VBN O
, , O
but CC O
slight JJ B-C
weakness NN I-C
in IN I-C
the DT I-C
extraocular JJ I-C
muscles NNS I-C
would MD O
lead VB O
to IN O
misalignment NN B-C
of IN I-C
the DT I-C
two CD I-C
eyes NNS I-C
, , O
even RB O
a DT O
small JJ O
degree NN O
of IN O
which WDT O
could MD O
lead VB O
to IN O
diplopia NN O
. . O

Verbal JJ B-C
memory NN I-C
deficit NN I-C
correlates VBZ O
with IN O
pyramidal JJ B-C
cell NN I-C
loss NN I-C
in IN I-C
TLE NNP I-C
. . O

Radiation NN B-C
dermatitis NN I-C
generally RB O
manifests VBZ O
within IN O
a DT O
few JJ O
weeks NNS O
after IN O
the DT O
start NN O
of IN O
radiotherapy NN B-C
. . O

Due IN O
to IN O
the DT O
state NN B-C
's POS I-C
lack NN I-C
of IN I-C
institutional JJ I-C
strength NN I-C
and CC I-C
capacity NN I-C
to TO I-C
provide VB I-C
basic JJ I-C
education NN I-C
to IN O
the DT O
general JJ O
Haitian JJ O
population NN O
, , O
the DT O
education NN B-C
sector NN I-C
is VBZ I-C
now RB I-C
predominantly RB I-C
privatized VBN I-C
. . O

While IN O
the DT O
most RBS O
common JJ O
symptom NN O
of IN O
PCT NNP B-C
is VBZ O
the DT O
appearance NN O
of IN O
skin NN B-C
lesions NNS I-C
and CC O
blistering VBG B-C
, , O
their PRP$ O
appearance NN O
does VBZ O
not RB O
single VB O
- HYPH O
handedly RB O
lead VB O
to IN O
a DT O
conclusive JJ O
diagnosis NN O
. . O

Features NNS O
distinguishing JJ O
Weaver NNP B-C
syndrome NN I-C
from IN O
Sotos NNP O
syndrome NN O
include VBP O
broad JJ B-C
forehead NN I-C
and CC I-C
face NN I-C
, , O
ocular JJ B-C
hypertelorism NN I-C
, , O
prominent JJ B-C
wide JJ I-C
philtrum NN I-C
, , O
micrognathia NN B-C
, , O
deep JJ B-C
- HYPH I-C
set VBN I-C
nails NNS I-C
, , O
retrognathia NN B-C
with IN I-C
a DT I-C
prominent JJ I-C
chin NN I-C
crease NN I-C
, , O
increased VBN B-C
prenatal JJ I-C
growth NN I-C
, , O
and CC O
a DT O
carpal NN B-C
bone NN I-C
age NN I-C
that WDT O
is VBZ O
greatly RB O
advanced VBN O
compared VBN O
to IN O
metacarpal JJ O
and CC O
phalangeal JJ O
bone NN O
age NN O
. . O

Alcohol NN B-C
provides VBZ O
a DT O
large JJ O
amount NN O
of IN O
calories NNS O
in IN O
a DT O
small JJ O
quantity NN O
of IN O
liquid NN O
, , O
which WDT O
tends VBZ O
to TO O
lead VB O
to IN O
unwanted JJ B-C
extra JJ I-C
calories NNS I-C
. . O

Death NN B-C
may MD O
occur VB O
and CC O
is VBZ O
usually RB O
due IN O
to IN O
severe JJ B-C
laminitis NN I-C
leading VBG O
to IN O
founder NN B-C
. . O

In IN O
the DT O
United NNP O
States NNP O
, , O
90 CD O
% NN O
of IN O
allergic JJ B-C
responses NNS I-C
to IN O
foods NNS O
are VBP O
caused VBN O
by IN O
cow NN B-C
's POS I-C
milk NN I-C
, , O
eggs NNS B-C
, , O
wheat NN B-C
, , O
shellfish NNS B-C
, , O
peanuts NNS B-C
, , O
tree NN B-C
nuts NNS I-C
, , O
fish NNS B-C
, , O
and CC O
soy NN B-C
beans NNS I-C
. . O

The DT O
first JJ O
case NN O
of IN O
reservoir NN B-C
- HYPH O
induced VBN O
seismicity NN B-C
occurred VBD O
in IN O
1932 CD O
in IN O
Algeria NNP O
’s POS O
Oued NNP O
Fodda NNP O
Dam NNP O
. . O

Exposure NN B-C
to IN I-C
microwaves NNS I-C
of IN I-C
too RB I-C
high JJ I-C
intensity NN I-C
can MD O
cause VB O
microwave NN B-C
burns NNS I-C
. . O

In IN O
acute JJ O
silicosis NN B-C
, , O
microscopic JJ O
pathology NN O
shows VBZ O
a DT O
periodic JJ B-C
acid-Schiff JJ I-C
positive JJ I-C
alveolar JJ I-C
exudate NN I-C
( -LRB- O
alveolar JJ O
lipoproteinosis NN O
) -RRB- O
and CC O
a DT O
cellular JJ B-C
infiltrate NN I-C
of IN I-C
the DT I-C
alveolar JJ I-C
walls NNS I-C
. . O

Primarily RB O
, , O
it PRP O
is VBZ O
key JJ O
that IN O
patients NNS O
diagnosed VBN O
with IN O
PCT NN B-C
avoid VB O
alcohol NN B-C
consumption NN I-C
, , O
iron NN B-C
supplements NNS I-C
, , O
excess JJ B-C
exposure NN I-C
to IN I-C
sunlight NN I-C
( -LRB- O
especially RB O
in IN O
the DT O
summer NN O
) -RRB- O
, , O
as RB O
well RB O
as IN O
estrogen NN B-C
and CC O
chlorinated VBN B-C
cyclic JJ I-C
hydrocarbons NNS I-C
, , O
all DT O
of IN O
which WDT O
can MD O
potentially RB O
exacerbate VB O
the DT O
disorder NN O
. . O

The DT O
presence NN B-C
of IN I-C
ions NNS I-C
in IN I-C
an DT I-C
electrolyte NN I-C
, , O
in IN O
this DT O
case NN O
, , O
hard JJ O
water NN O
, , O
can MD O
also RB O
lead VB O
to IN O
galvanic JJ B-C
corrosion NN I-C
, , O
in IN O
which WDT O
one CD O
metal NN O
will MD O
preferentially RB O
corrode VB O
when WRB O
in IN O
contact NN O
with IN O
another DT O
type NN O
of IN O
metal NN O
, , O
when WRB O
both DT O
are VBP O
in IN O
contact NN O
with IN O
an DT O
electrolyte NN O
. . O

This DT O
is VBZ O
particularly RB O
done VBN O
in IN O
lesions NNS B-C
with IN I-C
a DT I-C
feeding NN I-C
blood NN I-C
vessel NN I-C
of IN I-C
3 CD I-C
mm NNS I-C
or CC I-C
larger JJR I-C
, , O
as IN O
these DT O
are VBP O
the DT O
most RBS O
likely JJ O
to TO O
cause VB O
long JJ B-C
- HYPH I-C
term NN I-C
complications NNS I-C
unless IN O
treated VBN O
. . O

Cortisol NNP B-C
has VBZ O
been VBN O
tested VBN O
to TO O
slow VB B-C
down RP I-C
the DT I-C
body NN I-C
's POS I-C
metabolism NN I-C
. . O

This DT O
may MD O
lead VB O
to IN O
more JJR O
of IN O
a DT O
focus NN B-C
on IN I-C
etiquette NN I-C
than IN O
on IN O
ethics NNS O
as IN O
understood VBN O
in IN O
Western JJ O
civilization NN O
, , O
leading VBG O
some DT O
in IN O
Western JJ O
civilizations NNS O
to TO O
question VB B-C
why WRB I-C
the DT I-C
word NN I-C
ethos NN I-C
was VBD I-C
adapted VBN I-C
from IN I-C
Ancient JJ I-C
Greek NNP I-C
with IN O
such JJ O
vast JJ O
differences NNS O
in IN O
cultural JJ O
norms NNS O
. . O

Tape NN B-C
baking NN I-C
is VBZ O
a DT O
somewhat RB O
risky JJ O
procedure NN O
, , O
since IN O
there EX O
is VBZ O
a DT O
risk NN O
of IN O
damaging VBG B-C
the DT I-C
tape NN I-C
from IN O
the DT O
heat NN B-C
. . O

The DT O
recognition NN O
of IN O
respiratory JJ B-C
problems NNS I-C
from IN O
breathing NN B-C
in IN I-C
dust NN I-C
dates NNS O
to IN O
ancient JJ O
Greeks NNPS O
and CC O
Romans NNPS O
. . O

Problems NNS O
related JJ O
to IN O
3 CD B-C
- HYPH I-C
hydroxyacyl NN I-C
- HYPH I-C
coenzyme NN I-C
A DT I-C
dehydrogenase NN I-C
deficiency NN I-C
can MD O
be VB O
triggered VBN O
by IN O
periods NNS B-C
of IN I-C
fasting NN I-C
or CC O
by IN O
illnesses NNS B-C
such JJ O
as IN O
viral JJ B-C
infections NNS I-C
. . O

The DT O
changes NNS B-C
in IN I-C
crustal JJ I-C
stress NN I-C
patterns NNS I-C
caused VBN O
by IN O
the DT O
large JJ B-C
scale NN I-C
extraction NN I-C
of IN I-C
groundwater NN I-C
has VBZ O
been VBN O
shown VBN O
to TO O
trigger VB O
earthquakes NNS B-C
, , O
as IN O
in IN O
the DT O
case NN O
of IN O
the DT O
2011 CD B-C
Lorca NNP I-C
earthquake NN I-C
. . O

From IN O
1991 CD O
to IN O
1995 CD O
, , O
China NNP O
reported VBD O
more JJR O
than IN O
24,000 CD O
deaths NNS B-C
due IN O
to IN O
silicosis NN B-C
each DT O
year NN O
. . O

Therefore RB O
, , O
a DT O
deficiency NN B-C
in IN O
this DT O
enzyme NN O
causes VBZ O
the DT O
aforementioned VBN O
buildup NN O
of IN O
uroporphyrinogen NN B-C
and CC O
hepta-carboxylic JJ B-C
porphyrinogen NN I-C
, , O
and CC O
to IN O
a DT O
lesser JJR O
extent NN O
hexa NN B-C
- HYPH I-C
carboxylic JJ I-C
porphyrinogen NN I-C
, , O
and CC O
penta-carboxylic JJ B-C
porphyrinogen NN I-C
in IN O
the DT O
urine NN O
, , O
which WDT O
can MD O
be VB O
helpful JJ O
in IN O
the DT O
diagnosis NN O
of IN O
this DT O
disorder NN O
. . O

Radiation NN B-C
dermatitis NN I-C
( -LRB- O
also RB O
known VBN O
as IN O
radiodermatitis NN O
) -RRB- O
is VBZ O
a DT O
skin NN O
disease NN O
associated VBN O
with IN O
prolonged VBN B-C
exposure NN I-C
to IN I-C
ionizing NN I-C
radiation NN I-C
. . O

Spaced VBN B-C
armor NN I-C
and CC O
slat NN B-C
armor NN I-C
are VBP O
also RB O
designed VBN O
to TO O
defend VB O
against IN O
HEAT NN O
rounds NNS O
, , O
protecting VBG O
vehicles NNS O
by IN O
causing VBG O
premature JJ B-C
detonation NN I-C
of IN I-C
the DT I-C
explosive NN I-C
at IN O
a DT O
relatively RB O
safe JJ O
distance NN O
away RB O
from IN O
the DT O
main JJ O
armor NN O
of IN O
the DT O
vehicle NN O
. . O

Drinking NN B-C
alcohol NN I-C
excessively RB I-C
is VBZ O
a DT O
major JJ O
cause NN O
of IN O
chronic NN B-C
pancreatitis NN I-C
, , O
which WDT O
in IN O
turn NN O
predisposes VBZ O
to IN O
pancreatic JJ B-C
cancer NN I-C
. . O

Rectoceles NNS B-C
result NN O
from IN O
the DT O
weakening NN B-C
of IN I-C
the DT I-C
pelvic JJ I-C
floor NN I-C
also RB O
called VBD O
pelvic JJ B-C
organ JJ I-C
prolapse NN I-C
. . O

In IN O
Thailand NNP O
and CC O
Laos NNP O
, , O
bangungot NN B-C
( -LRB- O
or CC O
in IN O
their PRP$ O
term NN O
, , O
sudden JJ O
adult JJ O
death NN O
syndrome NN O
) -RRB- O
is VBZ O
caused VBN O
by IN O
the DT O
Brugada NNP B-C
syndrome NN I-C
. . O

Signs NNS O
and CC O
symptoms NNS O
of IN O
the DT O
most RBS O
common JJ O
form NN O
of IN O
pancreatic JJ B-C
cancer NN I-C
may MD O
include VB O
yellow JJ B-C
skin NN I-C
, , O
abdominal JJ B-C
or CC I-C
back NN I-C
pain NN I-C
, , O
unexplained JJ O
weight NN B-C
loss NN I-C
, , O
light NN B-C
- HYPH I-C
colored VBN I-C
stools NNS I-C
, , O
dark JJ B-C
urine NN I-C
and CC O
loss NN B-C
of IN I-C
appetite NN I-C
. . O

Urushiol NNP B-C
- HYPH O
induced VBN O
contact NN B-C
dermatitis NN I-C
is VBZ O
caused VBN O
by IN O
contact NN B-C
with IN I-C
a DT I-C
plant NN I-C
or CC O
any DT O
other JJ O
object NN O
containing VBG O
urushiol JJ O
oil NN O
. . O

Animal NN O
models NNS O
and CC O
clinical JJ O
studies NNS O
show VBP O
that IN O
memory NN B-C
loss NN I-C
correlates VBZ I-C
with IN O
temporal JJ B-C
lobe NN I-C
neuronal JJ I-C
loss NN I-C
in IN O
temporal JJ B-C
lobe NN I-C
epilepsy NN I-C
. . O

These DT O
observations NNS O
establish VBP O
a DT O
formal JJ O
causal JJ O
link NN O
between IN O
DNA NN B-C
methylation NN I-C
defects NNS I-C
and CC O
a DT O
disease NN B-C
of IN I-C
the DT I-C
peripheral JJ I-C
nervous JJ I-C
system NN I-C
. . O

About RB O
1 CD O
in IN O
4 CD O
college NN O
students NNS O
report VBP O
academic JJ B-C
consequences NNS I-C
from IN O
drinking NN B-C
, , O
including VBG O
missing VBG B-C
classes NNS I-C
, , O
falling VBG B-C
behind RB I-C
, , O
doing VBG B-C
poorly RB I-C
on IN I-C
exams NNS I-C
, , O
and CC O
overall RB O
receiving VBG B-C
lower JJR I-C
grade NN I-C
. . O

As IN O
the DT O
name NN O
suggests VBZ O
, , O
dry JJ B-C
mouth NN I-C
is VBZ O
a DT O
medical JJ O
condition NN O
that WDT O
occurs VBZ O
when WRB O
saliva NN B-C
production NN I-C
goes VBZ I-C
down RP I-C
. . O

In IN O
this DT O
situation NN O
, , O
proteins NNS B-C
in IN I-C
egg NN I-C
white JJ O
directly RB O
trigger VBP O
the DT O
release NN B-C
of IN I-C
histamine NN I-C
from IN O
mast NN O
cells NNS O
. . O

This DT O
is VBZ O
because IN O
people NNS B-C
with IN I-C
the DT I-C
disease NN I-C
tend VBP I-C
to TO I-C
avoid VB I-C
the DT I-C
sun NN I-C
due IN O
to IN O
photosensitivity NN B-C
( -LRB- O
and CC O
therefore RB O
have VB O
a DT O
young JJ O
complexion NN O
and CC O
pallor NN O
) -RRB- O
and CC O
require VBP O
blood NN O
. . O

Black JJ B-C
carbon NN I-C
in IN I-C
snow NN I-C
causes VBZ O
about RB O
three CD O
times NNS O
the DT O
temperature NN B-C
change NN I-C
as IN O
carbon NN O
dioxide NN O
in IN O
the DT O
atmosphere NN O
. . O

The DT O
most RBS O
common JJ O
type NN O
of IN O
radiation NN B-C
burn NN I-C
is VBZ O
a DT O
sunburn NN B-C
caused VBN O
by IN O
UV NNP B-C
radiation NN I-C
. . O

Both CC O
ENG NNP B-C
and CC I-C
ACVRL1 JJ I-C
mutations NNS I-C
lead VBP O
predominantly RB O
to IN O
underproduction NN B-C
of IN I-C
the DT I-C
related JJ I-C
proteins NNS I-C
, , O
rather RB O
than IN O
misfunctioning NN B-C
of IN I-C
the DT I-C
proteins NNS I-C
. . O

The DT O
dermatological JJ B-C
symptoms NNS I-C
of IN I-C
PCT NN I-C
that WDT O
include VBP O
blistering NN B-C
and CC O
lesions NNS B-C
on IN O
sun-exposed JJ O
areas NNS O
of IN O
the DT O
skin NN O
are VBP O
caused VBN O
by IN O
a DT O
buildup NN B-C
of IN I-C
porphyrin NN I-C
compounds NNS I-C
( -LRB- O
specifically RB O
uroporphyrinogen NN O
) -RRB- O
close JJ O
to IN O
the DT O
surface NN O
of IN O
the DT O
skin NN O
that WDT O
have VBP O
been VBN O
oxidized VBN O
by IN O
free JJ O
radicals NNS O
or CC O
sunlight NN O
. . O

The DT O
results NNS O
showed VBD O
that IN O
students NNS O
that WDT O
had VBD O
higher JJR B-C
levels NNS I-C
of IN I-C
stress NN I-C
were VBD O
more RBR O
likely JJ O
to TO O
have VB O
NES NNP B-C
due IN O
to IN O
the DT O
inability NN O
to TO O
adapt VB O
. . O

The DT O
vast JJ O
majority NN O
of IN O
hemangiomas NNS B-C
are VBP O
not RB O
associated VBN O
with IN O
complications NNS B-C
. . O

After IN O
the DT O
chemical JJ O
mediators NNS O
of IN O
the DT O
acute JJ O
response NN O
subside VBP O
, , O
late JJ B-C
- HYPH I-C
phase NN I-C
responses NNS I-C
can MD O
often RB O
occur VB O
due IN O
to IN O
the DT O
migration NN B-C
of IN I-C
other JJ I-C
white JJ I-C
blood NN I-C
cells NNS I-C
such JJ O
as IN O
neutrophils NNS O
, , O
lymphocytes NNS O
, , O
eosinophils NNS O
, , O
and CC O
macrophages NNS O
to IN O
the DT O
initial JJ O
reaction NN O
sites NNS O
. . O

This DT O
is VBZ O
why WRB O
patients NNS O
experiencing VBG O
symptomatic JJ B-C
facet NN I-C
syndrome NN I-C
can MD O
feel VB O
pain NN B-C
in IN O
their PRP$ O
shoulders NNS O
, , O
legs NNS O
and CC O
even RB O
manifested VBN O
in IN O
the DT O
form NN O
of IN O
headaches NNS B-C
. . O

Radiation NN B-C
burns NNS I-C
should MD O
be VB O
covered VBN O
by IN O
a DT O
clean JJ O
, , O
dry JJ O
dressing NN O
as RB O
soon RB O
as IN O
possible JJ O
to TO O
prevent VB O
infection NN B-C
. . O

Births NNS B-C
that WDT I-C
involve VBP I-C
babies NNS I-C
over IN I-C
nine CD I-C
pounds NNS I-C
in IN I-C
weight NN I-C
, , O
or CC O
rapid JJ B-C
births NNS I-C
can MD O
contribute VB O
to IN O
the DT O
development NN O
of IN O
rectocele NN B-C
. . O

The DT O
infections NNS B-C
caused VBD O
more JJR O
than IN O
half NN O
of IN O
the DT O
acute JJ O
deaths NNS B-C
. . O

In IN O
the DT O
United NNP O
States NNP O
, , O
it PRP O
is VBZ O
estimated VBN O
that IN O
between IN O
one CD O
and CC O
two CD O
million CD O
workers NNS O
have VBP O
had VBN O
occupational JJ B-C
exposure NN I-C
to IN I-C
crystalline JJ I-C
silica NN I-C
dust NN I-C
and CC O
59,000 CD O
of IN O
these DT O
workers NNS O
will MD O
develop VB O
silicosis NN B-C
sometime RB O
in IN O
the DT O
course NN O
of IN O
their PRP$ O
lives NNS O
. . O

The DT O
primary JJ O
cause NN O
of IN O
this DT O
disorder NN B-C
is VBZ O
a DT O
deficiency NN B-C
of IN I-C
uroporphyrinogen NN I-C
decarboxylase NN I-C
( -LRB- O
UROD NN O
) -RRB- O
, , O
a DT O
cytosolic NN O
enzyme NN O
that WDT O
is VBZ O
a DT O
step NN O
in IN O
the DT O
enzymatic JJ O
pathway NN O
that WDT O
leads VBZ O
to IN O
the DT O
synthesis NN O
of IN O
heme NN O
. . O

Lower JJR B-C
education NN I-C
level NN I-C
, , O
abnormal JJ B-C
background NN I-C
EEG NN I-C
activity NN I-C
, , O
and CC O
hippocampal JJ B-C
sclerosis NN I-C
have VBP O
been VBN O
found VBN O
to TO O
be VB O
contributing VBG O
factors NNS O
for IN O
religiosity NN B-C
in IN I-C
Temporal JJ I-C
Lobe NN I-C
Epilepsy NN I-C
. . O

For IN O
example NN O
, , O
if IN O
a DT O
person NN B-C
with IN I-C
complex JJ I-C
partial JJ I-C
seizures NNS I-C
is VBZ I-C
driving VBG I-C
alone RB I-C
, , O
this DT O
can MD O
cause VB O
them PRP O
to TO O
run VB B-C
into IN I-C
the DT I-C
ditch NN I-C
, , O
or CC O
worse JJR O
, , O
cause VB O
an DT O
accident NN B-C
involving VBG I-C
multiple JJ I-C
people NNS I-C
. . O

Thrombocytopenia NN B-C
in IN I-C
hospitalized VBN I-C
alcoholics NNS I-C
may MD O
be VB O
caused VBN O
by IN O
spleen NN B-C
enlargement NN I-C
, , O
folate NN B-C
deficiency NN I-C
, , O
and CC O
, , O
most RBS O
frequently RB O
, , O
the DT O
direct JJ B-C
toxic JJ I-C
effect NN I-C
of IN I-C
alcohol NN I-C
on IN I-C
production NN I-C
, , I-C
survival NN I-C
time NN I-C
, , I-C
and CC I-C
function NN I-C
of IN I-C
platelets NNS I-C
. . O

After IN O
the DT O
recession NN B-C
triggered VBN O
by IN O
the DT O
economic JJ B-C
crisis NN I-C
and CC O
ending VBG O
in IN O
2002 CD O
, , O
Argentina NNP O
's POS O
energy NN O
demands NNS O
grew VBD O
quickly RB O
as IN O
industry NN O
recovered VBD O
, , O
but CC O
extraction NN O
and CC O
transportation NN O
of IN O
natural JJ O
gas NN O
, , O
a DT O
cheap JJ O
and CC O
relatively RB O
abundant JJ O
fossil NN O
fuel NN O
, , O
did VBD O
not RB O
match VB O
the DT O
surge NN O
. . O

Radiation NN B-C
acne NN I-C
is VBZ O
a DT O
cutaneous JJ O
condition NN O
characterized VBN O
by IN O
comedo JJ B-C
- , I-C
like IN I-C
papules NNS I-C
occurring VBG O
at IN O
sites NNS O
of IN O
previous JJ O
exposure NN O
to IN O
therapeutic JJ O
ionizing NN O
radiation NN O
, , O
skin NN B-C
lesions NNS I-C
that WDT O
begin VBP O
to TO O
appear VB O
as IN O
the DT O
acute JJ O
phase NN O
of IN O
radiation NN O
dermatitis NN O
begins VBZ O
to TO O
resolve VB O
. . O

al NNP O
recently RB O
conducted VBD O
an DT O
informal JJ O
meta-analysis NN O
that WDT O
showed VBD O
significant JJ O
correlation NN O
between IN O
low JJ B-C
levels NNS I-C
of IN I-C
muscle NN I-C
strength NN I-C
and CC O
poor JJ B-C
physical JJ I-C
performance NN I-C
and CC O
/ , O
or CC O
physical JJ B-C
disability NN I-C
in IN O
90 CD O
% NN O
of IN O
the DT O
studies NNS O
. . O